Melissa Rusch

498 total citations
8 papers, 365 citations indexed

About

Melissa Rusch is a scholar working on Cell Biology, Molecular Biology and Genetics. According to data from OpenAlex, Melissa Rusch has authored 8 papers receiving a total of 365 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cell Biology, 3 papers in Molecular Biology and 2 papers in Genetics. Recurrent topics in Melissa Rusch's work include Proteoglycans and glycosaminoglycans research (7 papers), Hippo pathway signaling and YAP/TAZ (3 papers) and Glycosylation and Glycoproteins Research (2 papers). Melissa Rusch is often cited by papers focused on Proteoglycans and glycosaminoglycans research (7 papers), Hippo pathway signaling and YAP/TAZ (3 papers) and Glycosylation and Glycoproteins Research (2 papers). Melissa Rusch collaborates with scholars based in United States, United Kingdom and Netherlands. Melissa Rusch's co-authors include Scott B. Selleck, Henry Roehl, Sally E. Stringer, Chi‐Bin Chien, Aurélie Clément, Małgorzata Wiweger, Eleanor Y. Chen, Stephen C. Ekker, Jeong-Soo Lee and Robert Geisler and has published in prestigious journals such as Neuron, Endocrinology and Developmental Biology.

In The Last Decade

Melissa Rusch

8 papers receiving 362 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Melissa Rusch United States 6 223 218 77 55 54 8 365
Deanna Grant United States 5 154 0.7× 153 0.7× 59 0.8× 15 0.3× 29 0.5× 5 279
Ikuko Sugiyama Japan 8 213 1.0× 172 0.8× 21 0.3× 16 0.3× 29 0.5× 8 331
Yanzhen Cui United States 9 532 2.4× 76 0.3× 45 0.6× 34 0.6× 96 1.8× 10 609
Gene Elliott United States 6 363 1.6× 111 0.5× 21 0.3× 33 0.6× 108 2.0× 8 456
Gene Elliot United States 5 454 2.0× 154 0.7× 30 0.4× 30 0.5× 96 1.8× 6 543
R Landolt Switzerland 5 169 0.8× 155 0.7× 67 0.9× 21 0.4× 62 1.1× 10 345
Lucy J. Patterson Germany 5 386 1.7× 264 1.2× 26 0.3× 19 0.3× 35 0.6× 5 489
Megana Prasad United States 14 379 1.7× 71 0.3× 39 0.5× 49 0.9× 124 2.3× 19 550
Olga Lozynska United States 9 474 2.1× 275 1.3× 55 0.7× 34 0.6× 104 1.9× 11 571
Jodi L. Klessner United States 6 203 0.9× 120 0.6× 33 0.4× 24 0.4× 19 0.4× 6 390

Countries citing papers authored by Melissa Rusch

Since Specialization
Citations

This map shows the geographic impact of Melissa Rusch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Melissa Rusch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Melissa Rusch more than expected).

Fields of papers citing papers by Melissa Rusch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Melissa Rusch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Melissa Rusch. The network helps show where Melissa Rusch may publish in the future.

Co-authorship network of co-authors of Melissa Rusch

This figure shows the co-authorship network connecting the top 25 collaborators of Melissa Rusch. A scholar is included among the top collaborators of Melissa Rusch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Melissa Rusch. Melissa Rusch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Dang, Julie, Jing Ma, Jinjun Cheng, et al.. (2017). AMKL chimeric transcription factors are potent inducers of leukemia. Leukemia. 31(10). 2228–2234. 23 indexed citations
2.
Adhikari, Neeta, David L. Basi, DeWayne Townsend, et al.. (2010). Heparan sulfate Ndst1 regulates vascular smooth muscle cell proliferation, vessel size and vascular remodeling. Journal of Molecular and Cellular Cardiology. 49(2). 287–293. 23 indexed citations
3.
Rusch, Melissa, et al.. (2010). Sulfatase 1 Is an Inhibitor of Ductal Morphogenesis with Sexually Dimorphic Expression in the Urogenital Sinus. Endocrinology. 151(7). 3420–3431. 13 indexed citations
4.
Clément, Aurélie, Małgorzata Wiweger, Melissa Rusch, et al.. (2008). Regulation of Zebrafish Skeletogenesis by ext2/dackel and papst1/pinscher. PLoS Genetics. 4(7). e1000136–e1000136. 99 indexed citations
5.
Adhikari, Neeta, Melissa Rusch, Ami Mariash, et al.. (2008). Alterations in Heparan Sulfate in the Vessel in Response to Vascular Injury in the Mouse. Journal of Cardiovascular Translational Research. 1(3). 236–240. 5 indexed citations
6.
Dasgupta, Ujjaini, Bharat L. Dixit, Melissa Rusch, Scott B. Selleck, & Inge The. (2007). Functional conservation of the human EXT1 tumor suppressor gene and its Drosophila homolog tout velu. Development Genes and Evolution. 217(8). 555–561. 3 indexed citations
7.
Chen, Eleanor Y., Sally E. Stringer, Melissa Rusch, Scott B. Selleck, & Stephen C. Ekker. (2005). A unique role for 6-O sulfation modification in zebrafish vascular development. Developmental Biology. 284(2). 364–376. 70 indexed citations
8.
Lee, Jeong-Soo, Melissa Rusch, Sally E. Stringer, et al.. (2004). Axon Sorting in the Optic Tract Requires HSPG Synthesis by ext2 (dackel) and extl3 (boxer). Neuron. 44(6). 947–960. 129 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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