Melissa Gilbert‐Ross

801 total citations
19 papers, 492 citations indexed

About

Melissa Gilbert‐Ross is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Melissa Gilbert‐Ross has authored 19 papers receiving a total of 492 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 10 papers in Oncology and 9 papers in Cell Biology. Recurrent topics in Melissa Gilbert‐Ross's work include Hippo pathway signaling and YAP/TAZ (7 papers), Ubiquitin and proteasome pathways (4 papers) and Cancer-related Molecular Pathways (4 papers). Melissa Gilbert‐Ross is often cited by papers focused on Hippo pathway signaling and YAP/TAZ (7 papers), Ubiquitin and proteasome pathways (4 papers) and Cancer-related Molecular Pathways (4 papers). Melissa Gilbert‐Ross collaborates with scholars based in United States and Japan. Melissa Gilbert‐Ross's co-authors include Adam I. Marcus, Gabriel Sica, John A. Shupe, Kenneth H. Moberg, Jessica Konen, Lauren S. Havel, William Martin, Allyson E. Koyen, Hans E. Grossniklaus and Nancy C. Reich and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Journal of Clinical Oncology.

In The Last Decade

Melissa Gilbert‐Ross

19 papers receiving 487 citations

Peers

Melissa Gilbert‐Ross
Rory Flinn United States
Nayantara Kothari United States
Celine Ngouenet United States
Ekaterina Gresko Switzerland
Karan Bedi United States
Christophe Royer United Kingdom
Yoshiya Yonekubo United States
Ion Cristian Cirstea Germany
Jeffrey Wojton United States
Émilie Louvet France
Rory Flinn United States
Melissa Gilbert‐Ross
Citations per year, relative to Melissa Gilbert‐Ross Melissa Gilbert‐Ross (= 1×) peers Rory Flinn

Countries citing papers authored by Melissa Gilbert‐Ross

Since Specialization
Citations

This map shows the geographic impact of Melissa Gilbert‐Ross's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Melissa Gilbert‐Ross with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Melissa Gilbert‐Ross more than expected).

Fields of papers citing papers by Melissa Gilbert‐Ross

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Melissa Gilbert‐Ross. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Melissa Gilbert‐Ross. The network helps show where Melissa Gilbert‐Ross may publish in the future.

Co-authorship network of co-authors of Melissa Gilbert‐Ross

This figure shows the co-authorship network connecting the top 25 collaborators of Melissa Gilbert‐Ross. A scholar is included among the top collaborators of Melissa Gilbert‐Ross based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Melissa Gilbert‐Ross. Melissa Gilbert‐Ross is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Huayamares, Sebastian G., Yuning Hou, Hyejin Kim, et al.. (2025). Nanoparticle delivery of a prodrug-activating bacterial enzyme leads to anti-tumor responses. Nature Communications. 16(1). 3490–3490. 5 indexed citations
2.
Koo, Junghui, Chang-Soo Seong, Rebecca E. Parker, et al.. (2024). Live-Cell Invasive Phenotyping Uncovers ALK2 as a Therapeutic Target in LKB1 -Mutant Lung Cancer. Cancer Research. 84(22). 3761–3771. 3 indexed citations
3.
Sharma, Richa, Janna K. Mouw, Junghui Koo, et al.. (2024). Intra-tumoral YAP and TAZ heterogeneity drives collective NSCLC invasion that is targeted by SUMOylation inhibitor TAK-981. iScience. 27(11). 111133–111133. 2 indexed citations
4.
Ehrhardt, Annette, Bhagelu R. Achyut, Disha Joshi, et al.. (2022). Evaluating antitumor activity of Escherichia coli purine nucleoside phosphorylase against head and neck patient‐derived xenografts. Cancer Reports. 6(2). e1708–e1708. 2 indexed citations
5.
Seong, Chang-Soo, Rebecca E. Parker, Manali Rupji, et al.. (2021). The level of oncogenic Ras determines the malignant transformation of Lkb1 mutant tissue in vivo. Communications Biology. 4(1). 142–142. 3 indexed citations
6.
Jin, Rui, Boxuan Liu, Xiuju Liu, et al.. (2020). Leflunomide Suppresses the Growth of LKB1-Inactivated Tumors in the Immune-Competent Host and Attenuates Distant Cancer Metastasis. Molecular Cancer Therapeutics. 20(2). 274–283. 15 indexed citations
7.
Parker, William B., Paula W. Allan, William R. Waud, et al.. (2020). The use of Trichomonas vaginalis purine nucleoside phosphorylase to activate fludarabine in the treatment of solid tumors. Cancer Chemotherapy and Pharmacology. 85(3). 573–583. 8 indexed citations
8.
Owonikoko, Taofeek K., Bhakti Dwivedi, Zhengjia Chen, et al.. (2020). YAP1 Expression in SCLC Defines a Distinct Subtype With T-cell–Inflamed Phenotype. Journal of Thoracic Oncology. 16(3). 464–476. 100 indexed citations
9.
Owonikoko, Taofeek K., Bhakti Dwivedi, Zhengjia Chen, et al.. (2020). YAP1 positive small-cell lung cancer subtype is associated with the T-cell inflamed gene expression profile and confers good prognosis and long term survival.. Journal of Clinical Oncology. 38(15_suppl). 9019–9019. 7 indexed citations
10.
Havel, Lauren S., Allyson E. Koyen, Jessica Konen, et al.. (2017). Vimentin Is Required for Lung Adenocarcinoma Metastasis via Heterotypic Tumor Cell–Cancer-Associated Fibroblast Interactions during Collective Invasion. Clinical Cancer Research. 24(2). 420–432. 181 indexed citations
11.
Wei, Changyong, Abhinav Achreja, Jessica Konen, et al.. (2017). Abstract 4904: GLUT4 exhibits a non-canonical role of regulating lung cancer metastasis. Cancer Research. 77(13_Supplement). 4904–4904. 2 indexed citations
12.
Gilbert‐Ross, Melissa, Adam I. Marcus, & Wei Zhou. (2014). RhoA, a novel tumor suppressor or oncogene as a therapeutic target?. Genes & Diseases. 2(1). 2–3. 6 indexed citations
13.
Kline, Erik R., John A. Shupe, Melissa Gilbert‐Ross, Wei Zhou, & Adam I. Marcus. (2013). LKB1 Represses Focal Adhesion Kinase (FAK) Signaling via a FAK-LKB1 Complex to Regulate FAK Site Maturation and Directional Persistence. Journal of Biological Chemistry. 288(24). 17663–17674. 29 indexed citations
14.
Gilbert‐Ross, Melissa, Marla Tipping, Alexey Veraksa, & Kenneth H. Moberg. (2011). A Screen for Conditional Growth Suppressor Genes Identifies the Drosophila Homolog of HD-PTP as a Regulator of the Oncoprotein Yorkie. Developmental Cell. 20(5). 700–712. 40 indexed citations
15.
Gilbert‐Ross, Melissa, et al.. (2009). The archipelago Tumor Suppressor Gene Limits Rb/E2F-Regulated Apoptosis in Developing Drosophila Tissues. Current Biology. 19(18). 1503–1510. 15 indexed citations
16.
Gilbert‐Ross, Melissa, Brian Robinson, & Kenneth H. Moberg. (2009). Functional Interactions between the erupted/tsg101 Growth Suppressor Gene and the DaPKC and rbf1 Genes in Drosophila Imaginal Disc Tumors. PLoS ONE. 4(9). e7039–e7039. 7 indexed citations
17.
Gilbert‐Ross, Melissa, et al.. (2009). Genetic Interactions between the Drosophila Tumor Suppressor Gene ept and the stat92E Transcription Factor. PLoS ONE. 4(9). e7083–e7083. 10 indexed citations
18.
Gilbert‐Ross, Melissa & Kenneth H. Moberg. (2006). ESCRTing Cell Proliferation Off the Beaten Path: Unexpected Lessons from the Drosophila Eye. Cell Cycle. 5(3). 283–287. 4 indexed citations
19.
Gilbert‐Ross, Melissa, Brian K. Weaver, J. Peter Gergen, & Nancy C. Reich. (2005). A novel functional activator of the Drosophila JAK/STAT pathway, unpaired2, is revealed by an in vivo reporter of pathway activation. Mechanisms of Development. 122(7-8). 939–948. 53 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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