Mélissa Chénard

1.6k citations
13 papers · 605 · h-index 10

Impact in

Papers in

    • Histone Deacetylase Inhibitors Research 5
    • Protein Degradation and Inhibitors 4
    • PI3K/AKT/mTOR signaling in cancer 3
    • Glycosylation and Glycoproteins Research 2
    • Peptidase Inhibition and Analysis 5

Mélissa Chénard

13 papers receiving 597 citations

Peers

Mélissa Chénard
Comparison fields: 5 of 82
  • Computational Theory and Mathematics 194
  • Molecular Biology 453
  • Oncology 150
  • Biophysics 23
  • Cancer Research 56
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Jocelyn Chan United States
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Tea Pemovska Finland
Neil Kumar United States
Aliyu Musa Finland
Theonie Anastassiadis United States
Göran Dahl Sweden
Silvia Senese United States
Qiaodan Zhou China
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Citations per field
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Citations per year

Countries citing papers authored by Mélissa Chénard

Since Specialization
Citations

This map shows the geographic impact of Mélissa Chénard's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mélissa Chénard with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mélissa Chénard more than expected).

Fields of papers citing papers by Mélissa Chénard

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mélissa Chénard. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mélissa Chénard. The network helps show where Mélissa Chénard may publish in the future.

Co-authors

The 25 scholars most cited alongside Mélissa Chénard, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mélissa Chénard Line = papers co-authored together Mélissa Chénard links everyone, so they are left out of the graph.

All Works

13 of 13 papers shown
#Work
1 2016227
2 2010107
3 200780
4 200346
5 200836
6 200936
7 200820
8 200917
9 201416
10 200012
11 20156
12 20011
13 20101

About Mélissa Chénard

Mélissa Chénard is a scholar working on Molecular Biology, Oncology, Organic Chemistry, Cancer Research and Surgery, having authored 13 papers that have together received 605 indexed citations. Recurring topics across this work include Histone Deacetylase Inhibitors Research (5 papers), Peptidase Inhibition and Analysis (5 papers), Protein Degradation and Inhibitors (4 papers), PI3K/AKT/mTOR signaling in cancer (3 papers), Click Chemistry and Applications (2 papers), Glycosylation and Glycoproteins Research (2 papers), Circadian rhythm and melatonin (1 paper) and Cholinesterase and Neurodegenerative Diseases (1 paper). The work is most often cited by research in Computational Theory and Mathematics (194 citations), Molecular Biology (453 citations), Oncology (150 citations), Biophysics (23 citations) and Cancer Research (56 citations). Mélissa Chénard has collaborated with scholars based in United States and Canada. Frequent co-authors include Brian B. Haines, Astrid M. Kral, William T. Arthur, Theresa Zhang, Igor Feldman, Brian S. Roberts, Andrey Loboda, Judith C. Fleming, Răzvan Cristescu and Andrew Bloecher. Their work appears in journals such as Bioorganic & Medicinal Chemistry Letters, Molecular Cancer Therapeutics, Neoplasia, Journal of Cellular Biochemistry and Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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