Meleah A. Hickman

932 total citations
21 papers, 669 citations indexed

About

Meleah A. Hickman is a scholar working on Molecular Biology, Infectious Diseases and Plant Science. According to data from OpenAlex, Meleah A. Hickman has authored 21 papers receiving a total of 669 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 11 papers in Infectious Diseases and 11 papers in Plant Science. Recurrent topics in Meleah A. Hickman's work include Antifungal resistance and susceptibility (11 papers), Fungal and yeast genetics research (9 papers) and Fungal Infections and Studies (6 papers). Meleah A. Hickman is often cited by papers focused on Antifungal resistance and susceptibility (11 papers), Fungal and yeast genetics research (9 papers) and Fungal Infections and Studies (6 papers). Meleah A. Hickman collaborates with scholars based in United States, Israel and France. Meleah A. Hickman's co-authors include Judith Berman, Laura N. Rusché, Darren Abbey, Aimée M. Dudley, Ching‐Hua Su, Anja Forche, Yue Wang, Benjamin D. Harrison, Guisheng Zeng and Matthew P. Hirakawa and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and SHILAP Revista de lepidopterología.

In The Last Decade

Meleah A. Hickman

21 papers receiving 657 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Meleah A. Hickman United States 13 367 336 262 217 90 21 669
Mélanie Legrand France 16 422 1.1× 576 1.7× 439 1.7× 158 0.7× 57 0.6× 29 846
Darren Abbey United States 10 386 1.1× 647 1.9× 497 1.9× 224 1.0× 136 1.5× 11 936
Mary E. Logue Ireland 8 458 1.2× 280 0.8× 193 0.7× 231 1.1× 139 1.5× 9 706
Csilla Csank Canada 12 529 1.4× 496 1.5× 345 1.3× 119 0.5× 49 0.5× 15 829
Benjamin D. Harrison United States 5 316 0.9× 246 0.7× 192 0.7× 247 1.1× 81 0.9× 7 546
Jennifer L. Reedy United States 14 364 1.0× 490 1.5× 453 1.7× 255 1.2× 116 1.3× 22 930
Diego Martinez United States 4 200 0.5× 369 1.1× 324 1.2× 119 0.5× 118 1.3× 5 580
Ci Fu United States 16 458 1.2× 303 0.9× 355 1.4× 339 1.6× 149 1.7× 27 861
Annemiek Andel Netherlands 9 301 0.8× 219 0.7× 130 0.5× 365 1.7× 90 1.0× 9 683
Yu Sang China 14 314 0.9× 64 0.2× 73 0.3× 38 0.2× 17 0.2× 20 565

Countries citing papers authored by Meleah A. Hickman

Since Specialization
Citations

This map shows the geographic impact of Meleah A. Hickman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Meleah A. Hickman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Meleah A. Hickman more than expected).

Fields of papers citing papers by Meleah A. Hickman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Meleah A. Hickman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Meleah A. Hickman. The network helps show where Meleah A. Hickman may publish in the future.

Co-authorship network of co-authors of Meleah A. Hickman

This figure shows the co-authorship network connecting the top 25 collaborators of Meleah A. Hickman. A scholar is included among the top collaborators of Meleah A. Hickman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Meleah A. Hickman. Meleah A. Hickman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Smith, Amanda, et al.. (2022). Tetraploidy accelerates adaptation under drug selection in a fungal pathogen. SHILAP Revista de lepidopterología. 3. 984377–984377. 9 indexed citations
2.
Smith, Amanda, et al.. (2022). Increased Virulence and Large-Scale Reduction in Genome Size of Tetraploid Candida albicans Evolved in Nematode Hosts. SHILAP Revista de lepidopterología. 3. 903135–903135. 3 indexed citations
3.
Smith, Amanda, Levi T. Morran, & Meleah A. Hickman. (2021). Host Defense Mechanisms Induce Genome Instability Leading to Rapid Evolution in an Opportunistic Fungal Pathogen. Infection and Immunity. 90(2). e0032821–e0032821. 8 indexed citations
4.
Smith, Amanda, et al.. (2021). Two Infection Assays to Study Non-Lethal Virulence Phenotypes in <em>C. Albicans</em> using <em>C. Elegans</em>. Journal of Visualized Experiments. 2 indexed citations
5.
Ene, Iuliana V., Meleah A. Hickman, & Aleeza C. Gerstein. (2021). The Interplay Between Neutral and Adaptive Processes Shapes Genetic Variation During Candida Species Evolution. Current Clinical Microbiology Reports. 8(3). 129–138. 3 indexed citations
6.
Zhu, Lisha, et al.. (2020). Evolution of Distinct Responses to Low NAD+ Stress by Rewiring the Sir2 Deacetylase Network in Yeasts. Genetics. 214(4). 855–868. 7 indexed citations
7.
Smith, Amanda & Meleah A. Hickman. (2020). Host-Induced Genome Instability Rapidly Generates Phenotypic Variation across Candida albicans Strains and Ploidy States. mSphere. 5(3). 15 indexed citations
8.
Hickman, Meleah A., et al.. (2020). Virulence phenotypes result from interactions between pathogen ploidy and genetic background. Ecology and Evolution. 10(17). 9326–9338. 3 indexed citations
9.
10.
Hickman, Meleah A., et al.. (2019). The Magnitude of Candida albicans Stress-Induced Genome Instability Results from an Interaction Between Ploidy and Antifungal Drugs. G3 Genes Genomes Genetics. 9(12). 4019–4027. 19 indexed citations
11.
Gerstein, Aleeza C., et al.. (2017). Ploidy tug-of-war: Evolutionary and genetic environments influence the rate of ploidy drive in a human fungal pathogen. Evolution. 71(4). 1025–1038. 33 indexed citations
12.
Hickman, Meleah A., et al.. (2016). Phenotypic Consequences of a Spontaneous Loss of Heterozygosity in a Common Laboratory Strain of Candida albicans. Genetics. 203(3). 1161–1176. 24 indexed citations
13.
Hickman, Meleah A., et al.. (2015). Parasexual Ploidy Reduction Drives Population Heterogeneity Through Random and Transient Aneuploidy in Candida albicans. Genetics. 200(3). 781–794. 81 indexed citations
14.
15.
Hickman, Meleah A., Guisheng Zeng, Anja Forche, et al.. (2013). The ‘obligate diploid’ Candida albicans forms mating-competent haploids. Nature. 494(7435). 55–59. 216 indexed citations
16.
Abbey, Darren, Meleah A. Hickman, David Gresham, & Judith Berman. (2011). High-Resolution SNP/CGH Microarrays Reveal the Accumulation of Loss of Heterozygosity in Commonly UsedCandida albicansStrains. G3 Genes Genomes Genetics. 1(7). 523–530. 60 indexed citations
17.
Hickman, Meleah A., et al.. (2011). Reinventing Heterochromatin in Budding Yeasts: Sir2 and the Origin Recognition Complex Take Center Stage. Eukaryotic Cell. 10(9). 1183–1192. 41 indexed citations
18.
Hickman, Meleah A. & Laura N. Rusché. (2010). Transcriptional silencing functions of the yeast protein Orc1/Sir3 subfunctionalized after gene duplication. Proceedings of the National Academy of Sciences. 107(45). 19384–19389. 41 indexed citations
19.
20.
Hickman, Meleah A. & Laura N. Rusché. (2007). Substitution as a Mechanism for Genetic Robustness: The Duplicated Deacetylases Hst1p and Sir2p in Saccharomyces cerevisiae. PLoS Genetics. 3(8). e126–e126. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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