Melanie D. Mark

3.4k total citations · 1 hit paper
67 papers, 2.6k citations indexed

About

Melanie D. Mark is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Melanie D. Mark has authored 67 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 54 papers in Cellular and Molecular Neuroscience, 40 papers in Molecular Biology and 12 papers in Cognitive Neuroscience. Recurrent topics in Melanie D. Mark's work include Neuroscience and Neuropharmacology Research (34 papers), Photoreceptor and optogenetics research (16 papers) and Ion channel regulation and function (16 papers). Melanie D. Mark is often cited by papers focused on Neuroscience and Neuropharmacology Research (34 papers), Photoreceptor and optogenetics research (16 papers) and Ion channel regulation and function (16 papers). Melanie D. Mark collaborates with scholars based in Germany, United States and France. Melanie D. Mark's co-authors include Stefan Herlitze, Daniel R. Storm, Charles Chavkin, Steven Poser, Enrique C. Villacres, Soren Impey, Davina V. Gutierrez, Hillel J. Chiel, M. Gartz Hanson and Xiang Li and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Melanie D. Mark

63 papers receiving 2.5k citations

Hit Papers

Induction of CRE-Mediated Gene Expression by Stimuli That... 1996 2026 2006 2016 1996 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Melanie D. Mark Germany 26 1.8k 1.4k 420 178 170 67 2.6k
Tommaso Patriarchi United States 27 1.7k 1.0× 1.4k 1.0× 736 1.8× 151 0.8× 125 0.7× 48 2.8k
Peter Vanhoutte France 27 2.1k 1.2× 1.9k 1.3× 492 1.2× 220 1.2× 180 1.1× 46 3.3k
Damiana Leo Italy 27 1.5k 0.8× 1.6k 1.1× 364 0.9× 132 0.7× 95 0.6× 54 2.6k
Christine E. Gee Germany 28 1.2k 0.7× 837 0.6× 573 1.4× 106 0.6× 132 0.8× 61 2.0k
Mark H. Pausch United States 22 1.5k 0.9× 1.7k 1.2× 547 1.3× 103 0.6× 149 0.9× 28 2.9k
Duk-Su Koh United States 23 1.8k 1.0× 1.8k 1.3× 501 1.2× 162 0.9× 225 1.3× 44 3.1k
Rebecca A. Piskorowski France 23 1.2k 0.7× 1.1k 0.8× 475 1.1× 156 0.9× 106 0.6× 35 2.0k
Yajun Zhang United States 17 1.4k 0.8× 1.0k 0.7× 406 1.0× 166 0.9× 107 0.6× 34 2.4k
Asim J. Rashid Canada 28 2.1k 1.2× 1.4k 1.0× 1.1k 2.5× 105 0.6× 108 0.6× 43 2.9k
Guy C.‐K. Chan United States 26 1.8k 1.0× 1.4k 1.0× 545 1.3× 288 1.6× 166 1.0× 35 3.2k

Countries citing papers authored by Melanie D. Mark

Since Specialization
Citations

This map shows the geographic impact of Melanie D. Mark's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Melanie D. Mark with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Melanie D. Mark more than expected).

Fields of papers citing papers by Melanie D. Mark

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Melanie D. Mark. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Melanie D. Mark. The network helps show where Melanie D. Mark may publish in the future.

Co-authorship network of co-authors of Melanie D. Mark

This figure shows the co-authorship network connecting the top 25 collaborators of Melanie D. Mark. A scholar is included among the top collaborators of Melanie D. Mark based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Melanie D. Mark. Melanie D. Mark is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yang, Yi, Ádám Fekete, Jason Arsenault, et al.. (2024). Intersectin‐1 enhances calcium‐dependent replenishment of the readily releasable pool of synaptic vesicles during development. The Journal of Physiology. 603(20). 6185–6206. 5 indexed citations
2.
Batsikadze, Giorgi, Michael Klein, Thomas Ernst, et al.. (2024). Mild Deficits in Fear Learning: Evidence from Humans and Mice with Cerebellar Cortical Degeneration. eNeuro. 11(2). ENEURO.0365–23.2023.
3.
Spoida, Katharina, Melanie D. Mark, Ida Siveke, et al.. (2024). Gain control of sensory input across polysynaptic circuitries in mouse visual cortex by a single G protein-coupled receptor type (5-HT2A). Nature Communications. 15(1). 8078–8078. 3 indexed citations
4.
Bader, Verian, et al.. (2023). Loss of parkin causes endoplasmic reticulum calcium dyshomeostasis by upregulation of reticulocalbin 1. European Journal of Neuroscience. 57(5). 739–761. 4 indexed citations
6.
Mark, Melanie D., et al.. (2022). Cerebellum and Emotion Memory. Advances in experimental medicine and biology. 1378. 53–73. 7 indexed citations
7.
Jancke, Dirk, et al.. (2022). Optogenetic activation of mGluR1 signaling in the cerebellum induces synaptic plasticity. iScience. 26(1). 105828–105828. 4 indexed citations
8.
Lutz, David, Elisabeth Petrasch‐Parwez, Jan Claudius Schwitalla, et al.. (2020). Reelin signaling modulates GABAB receptor function in the neocortex. Journal of Neurochemistry. 156(5). 589–603. 16 indexed citations
9.
Müller, Julia, Peter Bedner, Xavier Helluy, et al.. (2020). Lipoprotein receptor loss in forebrain radial glia results in neurological deficits and severe seizures. Glia. 68(12). 2517–2549. 9 indexed citations
10.
Spoida, Katharina, et al.. (2020). Separable gain control of ongoing and evoked activity in the visual cortex by serotonergic input. eLife. 9. 32 indexed citations
11.
Rudack, Till, Melanie D. Mark, Katharina Spoida, et al.. (2019). Design of an Ultrafast G Protein Switch Based on a Mouse Melanopsin Variant. ChemBioChem. 20(14). 1766–1771. 7 indexed citations
12.
Fekete, Ádám, Yukihiro Nakamura, Yi Yang, et al.. (2019). Underpinning heterogeneity in synaptic transmission by presynaptic ensembles of distinct morphological modules. Nature Communications. 10(1). 826–826. 45 indexed citations
13.
Mark, Melanie D., Martin Krause, Henk‐Jan Boele, et al.. (2015). Spinocerebellar Ataxia Type 6 Protein Aggregates Cause Deficits in Motor Learning and Cerebellar Plasticity. Journal of Neuroscience. 35(23). 8882–8895. 53 indexed citations
15.
Mark, Melanie D., Takashi Maejima, Jong W. Yoo, et al.. (2011). Delayed Postnatal Loss of P/Q-Type Calcium Channels Recapitulates the Absence Epilepsy, Dyskinesia, and Ataxia Phenotypes of GenomicCacna1AMutations. Journal of Neuroscience. 31(11). 4311–4326. 85 indexed citations
16.
Herlitze, Stefan, et al.. (2003). Targeting Mechanisms of High Voltage-Activated Ca2+ Channels. Journal of Bioenergetics and Biomembranes. 35(6). 621–637. 26 indexed citations
17.
Mark, Melanie D., et al.. (2000). G protein modulation of recombinant P/Q‐type calcium channels by regulators of G protein signalling proteins. The Journal of Physiology. 528(1). 65–77. 31 indexed citations
18.
Mark, Melanie D. & Daniel R. Storm. (1997). Coupling of Epidermal Growth Factor (EGF) with the Antiproliferative Activity of cAMP Induces Neuronal Differentiation. Journal of Biological Chemistry. 272(27). 17238–17244. 43 indexed citations
19.
Impey, Soren, Melanie D. Mark, Enrique C. Villacres, et al.. (1996). Induction of CRE-Mediated Gene Expression by Stimuli That Generate Long-Lasting LTP in Area CA1 of the Hippocampus. Neuron. 16(5). 973–982. 501 indexed citations breakdown →
20.
Jerushalmy, Z., et al.. (1988). Kinetic Values of Active Serotonin Transport by Platelets of Bipolar, Unipolar and Schizophrenic Patients at 2 and at 8 a.m.. Neuropsychobiology. 20(2). 57–61. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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