Meghan C. Kopp

864 total citations · 1 hit paper
15 papers, 514 citations indexed

About

Meghan C. Kopp is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Oncology. According to data from OpenAlex, Meghan C. Kopp has authored 15 papers receiving a total of 514 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Pulmonary and Respiratory Medicine and 5 papers in Oncology. Recurrent topics in Meghan C. Kopp's work include Neonatal Respiratory Health Research (4 papers), Ubiquitin and proteasome pathways (4 papers) and Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (3 papers). Meghan C. Kopp is often cited by papers focused on Neonatal Respiratory Health Research (4 papers), Ubiquitin and proteasome pathways (4 papers) and Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (3 papers). Meghan C. Kopp collaborates with scholars based in United States, Russia and Japan. Meghan C. Kopp's co-authors include Yaniv Tomer, Surafel Mulugeta, Jeremy Katzen, Alessandro Venosa, Michael F. Beers, Scott J. Russo, Erica A. Golemis, Shin‐ichi Nureki, Alexander Y. Deneka and Anna S. Nikonova and has published in prestigious journals such as Journal of Clinical Investigation, Nature Communications and The Journal of Immunology.

In The Last Decade

Meghan C. Kopp

15 papers receiving 512 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Desmoplastic stroma restricts T cell extravasation and mediates immune exclusion and immunosuppression in solid tumors

2023 129 citations Zebin Xiao, Leslie Todd et al. Nature Communications profile →

Peers

Meghan C. Kopp

PRM

ONCOL

IMMUN

SURGE

Min Peng China

Sisi Liu China

Wei Zhuang China

Arnaud Bonnomet France

Miriam Hetzel Germany

Lucille N. Meliton United States

Elise Bonvin France

Fujun Han China

Binita Chakraborty United States

Wenping Li China

Min Peng China

Meghan C. Kopp

137 ×0.5

PRM

213 ×1.2

206 ×1.8

ONCOL

72 ×0.9

IMMUN

36 ×0.6

SURGE

Citations per year, relative to Meghan C. Kopp Meghan C. Kopp (= 1×) peers Min Peng

Countries citing papers authored by Meghan C. Kopp

Since Specialization

Citations

This map shows the geographic impact of Meghan C. Kopp's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Meghan C. Kopp with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Meghan C. Kopp more than expected).

Fields of papers citing papers by Meghan C. Kopp

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Meghan C. Kopp. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Meghan C. Kopp. The network helps show where Meghan C. Kopp may publish in the future.

Co-authorship network of co-authors of Meghan C. Kopp

This figure shows the co-authorship network connecting the top 25 collaborators of Meghan C. Kopp. A scholar is included among the top collaborators of Meghan C. Kopp based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Meghan C. Kopp. Meghan C. Kopp is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

15 of 15 papers shown

Xiao, Zebin, Leslie Todd, Li Huang, et al.. (2023). Desmoplastic stroma restricts T cell extravasation and mediates immune exclusion and immunosuppression in solid tumors. Nature Communications. 14(1). 5110–5110. 129 indexed citations breakdown →

Kopp, Meghan C., et al.. (2022). Abstract 2475: Visualization of cell to cell and cell to matrix interactions in the disruption of tumor-promoting desmoplasia by adoptive transfer of fibroblast activation protein targeted chimeric antigen receptor (CAR) T cells. Cancer Research. 82(12_Supplement). 2475–2475. 1 indexed citations

Deneka, Alexander Y., Meghan C. Kopp, Anna S. Nikonova, et al.. (2021). Nedd9 Restrains Autophagy to Limit Growth of Early Stage Non–Small Cell Lung Cancer. Cancer Research. 81(13). 3717–3726. 13 indexed citations

Katzen, Jeremy, Brandie D. Wagner, Alessandro Venosa, et al.. (2019). A SFTPC BRICHOS mutant links epithelial ER stress and spontaneous lung fibrosis. JCI Insight. 4(6). 80 indexed citations

Deneka, Alexander Y., Anna S. Nikonova, Meghan C. Kopp, et al.. (2019). Abstract 1840: Nedd9 controls glycolysis and autophagy in non-small cell lung cancer (NSCLC). Cancer Research. 79(13_Supplement). 1840–1840. 1 indexed citations

Deneka, Alexander Y., Anna S. Nikonova, Meghan C. Kopp, et al.. (2019). Abstract 1840:Nedd9controls glycolysis and autophagy in non-small cell lung cancer (NSCLC). 1840–1840. 1 indexed citations

Katzen, Jeremy, Alessandro Venosa, Yaniv Tomer, et al.. (2019). Interstitial lung disease related surfactant protein-C mutations alter the transcriptome and progenitor cell function of alveolar epithelial cells in mice. PA1291–PA1291. 1 indexed citations

Venosa, Alessandro, Jeremy Katzen, Yaniv Tomer, et al.. (2019). Epithelial Expression of an Interstitial Lung Disease–Associated Mutation in Surfactant Protein-C Modulates Recruitment and Activation of Key Myeloid Cell Populations in Mice. The Journal of Immunology. 202(9). 2760–2771. 34 indexed citations

Nureki, Shin‐ichi, Yaniv Tomer, Alessandro Venosa, et al.. (2018). Expression of mutant Sftpc in murine alveolar epithelia drives spontaneous lung fibrosis. Journal of Clinical Investigation. 128(9). 4008–4024. 143 indexed citations

10.

Nikonova, Anna S., Alexander Y. Deneka, Anna A. Kiseleva, et al.. (2018). Ganetespib limits ciliation and cystogenesis in autosomal‐dominant polycystic kidney disease (ADPKD). The FASEB Journal. 32(5). 2735–2746. 30 indexed citations

11.

Deneka, Alexander Y., et al.. (2017). Tumor-targeted SN38 inhibits growth of early stage non-small cell lung cancer (NSCLC) in a KRas/p53 transgenic mouse model. PLoS ONE. 12(4). e0176747–e0176747. 8 indexed citations

12.

Deneka, Alexander Y., et al.. (2017). Abstract 1834: Constitutive Nedd9 null genotype promotes lung cancer aggressiveness. Cancer Research. 77(13_Supplement). 1834–1834. 1 indexed citations

13.

Nikonova, Anna S., Alexander Y. Deneka, Meghan C. Kopp, et al.. (2016). A Novel HSP90 Inhibitor–Drug Conjugate to SN38 Is Highly Effective in Small Cell Lung Cancer. Clinical Cancer Research. 22(20). 5120–5129. 25 indexed citations

14.

Nikonova, Anna S., Alexander Y. Deneka, Meghan C. Kopp, et al.. (2015). Opposing Effects of Inhibitors of Aurora-A and EGFR in Autosomal-Dominant Polycystic Kidney Disease. Frontiers in Oncology. 5. 228–228. 15 indexed citations

15.

Beck, Tim N., Émmanuelle Nicolas, Meghan C. Kopp, & Erica A. Golemis. (2014). Adaptors for disorders of the brain? The cancer signaling proteins NEDD9, CASS4, and PTK2B in Alzheimer’s disease. Oncoscience. 1(7). 486–503. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact