Mazhar N. Malik

2.9k total citations · 1 hit paper
52 papers, 2.1k citations indexed

About

Mazhar N. Malik is a scholar working on Molecular Biology, Cell Biology and Genetics. According to data from OpenAlex, Mazhar N. Malik has authored 52 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 13 papers in Cell Biology and 10 papers in Genetics. Recurrent topics in Mazhar N. Malik's work include Autism Spectrum Disorder Research (8 papers), Protein Hydrolysis and Bioactive Peptides (7 papers) and Genetics and Neurodevelopmental Disorders (7 papers). Mazhar N. Malik is often cited by papers focused on Autism Spectrum Disorder Research (8 papers), Protein Hydrolysis and Bioactive Peptides (7 papers) and Genetics and Neurodevelopmental Disorders (7 papers). Mazhar N. Malik collaborates with scholars based in United States, China and United Kingdom. Mazhar N. Malik's co-authors include Ashfaq M. Sheikh, W. Ted Brown, Xiumin Li, Xiaohong Li, Hongen Wei, Ved Chauhan, Abha Chauhan, Ted Brown, Sangita Patil and Lina Ji and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Mazhar N. Malik

52 papers receiving 2.0k citations

Hit Papers

Elevated immune response in the brain of autistic patients 2009 2026 2014 2020 2009 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mazhar N. Malik United States 21 823 640 625 251 245 52 2.1k
Ashfaq M. Sheikh United States 16 800 1.0× 418 0.7× 540 0.9× 202 0.8× 229 0.9× 30 1.7k
Helen Wong United States 14 514 0.6× 933 1.5× 504 0.8× 441 1.8× 291 1.2× 19 2.3k
Miklós Sántha Hungary 24 278 0.3× 941 1.5× 291 0.5× 527 2.1× 144 0.6× 57 2.4k
Zhe Jin Sweden 28 269 0.3× 926 1.4× 430 0.7× 678 2.7× 364 1.5× 104 2.8k
B Antoine France 27 633 0.8× 859 1.3× 203 0.3× 812 3.2× 193 0.8× 73 2.6k
Wenqiang Li China 27 242 0.3× 994 1.6× 447 0.7× 223 0.9× 197 0.8× 163 2.4k
Limin Shi China 23 324 0.4× 689 1.1× 366 0.6× 229 0.9× 224 0.9× 43 2.6k
Amaicha Mara Depino Argentina 23 503 0.6× 451 0.7× 317 0.5× 536 2.1× 684 2.8× 33 2.2k
Cristian Bonvicini Italy 30 345 0.4× 597 0.9× 321 0.5× 320 1.3× 351 1.4× 77 2.7k
Kodavali V. Chowdari United States 30 245 0.3× 996 1.6× 859 1.4× 496 2.0× 105 0.4× 58 2.4k

Countries citing papers authored by Mazhar N. Malik

Since Specialization
Citations

This map shows the geographic impact of Mazhar N. Malik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mazhar N. Malik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mazhar N. Malik more than expected).

Fields of papers citing papers by Mazhar N. Malik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mazhar N. Malik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mazhar N. Malik. The network helps show where Mazhar N. Malik may publish in the future.

Co-authorship network of co-authors of Mazhar N. Malik

This figure shows the co-authorship network connecting the top 25 collaborators of Mazhar N. Malik. A scholar is included among the top collaborators of Mazhar N. Malik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mazhar N. Malik. Mazhar N. Malik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Wei, Hongen, Kathryn K. Chadman, Daniel P. McCloskey, et al.. (2012). Brain IL-6 elevation causes neuronal circuitry imbalances and mediates autism-like behaviors. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1822(6). 831–842. 205 indexed citations
3.
Wei, Hongen, Carl Dobkin, Ashfaq M. Sheikh, et al.. (2012). The Therapeutic effect of Memantine through the Stimulation of Synapse Formation and Dendritic Spine Maturation in Autism and Fragile X Syndrome. PLoS ONE. 7(5). e36981–e36981. 47 indexed citations
4.
Yang, Kun, Fujiang Cao, Ashfaq M. Sheikh, et al.. (2012). Up-regulation of Ras/Raf/ERK1/2 signaling impairs cultured neuronal cell migration, neurogenesis, synapse formation, and dendritic spine development. Brain Structure and Function. 218(3). 669–682. 34 indexed citations
5.
Wei, Hongen, Mazhar N. Malik, Ashfaq M. Sheikh, et al.. (2011). Abnormal Cell Properties and Down-Regulated FAK-Src Complex Signaling in B Lymphoblasts of Autistic Subjects. American Journal Of Pathology. 179(1). 66–74. 29 indexed citations
6.
Seaver, Laurie H., Keith Abe, Tina M. Cowan, et al.. (2011). A Novel Mutation in the HSD17B10 Gene of a 10-Year-Old Boy with Refractory Epilepsy, Choreoathetosis and Learning Disability. PLoS ONE. 6(11). e27348–e27348. 22 indexed citations
7.
Malik, Mazhar N., et al.. (2011). A Novel Approach for Characterization of Cathepsin D Protease and Its Effect on Tau and β-Amyloid Proteins. Neurochemical Research. 36(5). 754–760. 10 indexed citations
8.
Sheikh, Ashfaq M., Mazhar N. Malik, G. Y. Wen, et al.. (2010). BDNF‐Akt‐Bcl2 antiapoptotic signaling pathway is compromised in the brain of autistic subjects. Journal of Neuroscience Research. 88(12). 2641–2647. 90 indexed citations
9.
Li, Xiaohong, Abha Chauhan, Ashfaq M. Sheikh, et al.. (2009). Elevated immune response in the brain of autistic patients. Journal of Neuroimmunology. 207(1-2). 111–116. 590 indexed citations breakdown →
10.
Sheikh, Ashfaq M., et al.. (2009). Cathepsin D and apoptosis related proteins are elevated in the brain of autistic subjects. Neuroscience. 165(2). 363–370. 62 indexed citations
11.
Chauhan, Ved, et al.. (2005). Fibrillar amyloid beta-protein inhibits the activity of high molecular weight brain protease and trypsin. Journal of Alzheimer s Disease. 7(1). 37–44. 10 indexed citations
12.
Wen, G. Y., Henryk M. Wı́sniewski, Mazhar N. Malik, et al.. (1995). Light and electron microscopic immunocytochemical localization of two major proteins in garlic bulb. Journal of Cellular Biochemistry. 58(4). 481–489. 13 indexed citations
13.
Malik, Mazhar N., et al.. (1991). High molecular weight glycosylated protease in calf brain. Purification and partial characterization.. Journal of Biological Chemistry. 266(10). 6594–6599. 4 indexed citations
14.
Malik, Mazhar N., et al.. (1987). Micromolar Ca2+ requiring protease from human platelets: Purification, partial characterization and effect on the cytoskeletal proteins. Life Sciences. 40(6). 593–604. 13 indexed citations
15.
Malik, Mazhar N., et al.. (1983). Purification and Characterization of Myosin from Calf Brain. Journal of Neurochemistry. 40(6). 1620–1629. 14 indexed citations
16.
Malik, Mazhar N., et al.. (1981). Calcium activated proteolysis of fibrous proteins in central nervous system. Life Sciences. 29(8). 795–802. 31 indexed citations
17.
Malik, Mazhar N.. (1978). Steady-state kinetics of smooth muscle myosin. Biochemistry. 17(1). 27–32. 15 indexed citations
18.
Malik, Mazhar N., Joel Abramowitz, Thomas C. Detwiler, & Alfred Stracher. (1974). Enzymatic properties of platelet actomyosin. Archives of Biochemistry and Biophysics. 161(1). 268–274. 8 indexed citations
19.
Martonosi, A. & Mazhar N. Malik. (1973). Kinetics of Formation and Dissociation of H-Meromyosin-ADP Complex. Cold Spring Harbor Symposia on Quantitative Biology. 37(0). 184–185. 14 indexed citations
20.
Malik, Mazhar N. & Anthony Martonosi. (1972). The regulation of the rate of ATP hydrolysis by H-meromyosin. Archives of Biochemistry and Biophysics. 152(1). 243–257. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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