Maxime Lamontagne

3.0k total citations
18 papers, 522 citations indexed

About

Maxime Lamontagne is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Maxime Lamontagne has authored 18 papers receiving a total of 522 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Pulmonary and Respiratory Medicine and 5 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Maxime Lamontagne's work include Chronic Obstructive Pulmonary Disease (COPD) Research (6 papers), Cardiac Valve Diseases and Treatments (5 papers) and RNA modifications and cancer (4 papers). Maxime Lamontagne is often cited by papers focused on Chronic Obstructive Pulmonary Disease (COPD) Research (6 papers), Cardiac Valve Diseases and Treatments (5 papers) and RNA modifications and cancer (4 papers). Maxime Lamontagne collaborates with scholars based in Canada, United States and Netherlands. Maxime Lamontagne's co-authors include Yohan Bossé, Nathalie Gaudreault, Patrick Mathieu, Philippe Pîbarot, Wim Timens, Michel Laviolette, Peter D. Paré, Don D. Sin, Dirkje S. Postma and James C. Hogg and has published in prestigious journals such as Nature Communications, PLoS ONE and Cancer Research.

In The Last Decade

Maxime Lamontagne

18 papers receiving 518 citations

Peers

Maxime Lamontagne
B.L.Y. Cheuk Hong Kong
Xing Lyu China
P Even Italy
Maria Haslip United States
Justin Chin-Bong Choi United States
Kimberly A. Queisser United States
John T. Berg United States
B.L.Y. Cheuk Hong Kong
Maxime Lamontagne
Citations per year, relative to Maxime Lamontagne Maxime Lamontagne (= 1×) peers B.L.Y. Cheuk

Countries citing papers authored by Maxime Lamontagne

Since Specialization
Citations

This map shows the geographic impact of Maxime Lamontagne's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maxime Lamontagne with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maxime Lamontagne more than expected).

Fields of papers citing papers by Maxime Lamontagne

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maxime Lamontagne. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maxime Lamontagne. The network helps show where Maxime Lamontagne may publish in the future.

Co-authorship network of co-authors of Maxime Lamontagne

This figure shows the co-authorship network connecting the top 25 collaborators of Maxime Lamontagne. A scholar is included among the top collaborators of Maxime Lamontagne based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maxime Lamontagne. Maxime Lamontagne is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Bossé, Yohan, Maxime Lamontagne, Nathalie Gaudreault, et al.. (2019). Early-onset emphysema in a large French-Canadian family: a genetic investigation. The Lancet Respiratory Medicine. 7(5). 427–436. 15 indexed citations
2.
Sakornsakolpat, Phuwanat, Dmitry Prokopenko, Maxime Lamontagne, Nicola Reeve, & Milo A. Puhan. (2019). Genetic landscape of chronic obstructive pulmonary disease identifies heterogeneous cell-type and phenotype associations. Zurich Open Repository and Archive (University of Zurich). 1 indexed citations
3.
Thériault, Sébastien, Nathalie Gaudreault, Maxime Lamontagne, et al.. (2018). A transcriptome-wide association study identifies PALMD as a susceptibility gene for calcific aortic valve stenosis. Nature Communications. 9(1). 988–988. 77 indexed citations
4.
Lamontagne, Maxime, Jean‐Christophe Bérubé, Ma’en Obeidat, et al.. (2018). Leveraging lung tissue transcriptome to uncover candidate causal genes in COPD genetic associations. Human Molecular Genetics. 27(10). 1819–1829. 27 indexed citations
5.
Boucherat, Olivier, Maxime Lamontagne, Sabrina Biardel, et al.. (2018). Lung cancer susceptibility genetic variants modulate HOXB2 expression in the lung. The International Journal of Developmental Biology. 62(11-12). 857–864. 9 indexed citations
6.
Sauler, Maor, Maxime Lamontagne, Jose D. Herazo‐Maya, et al.. (2018). The DNA repair transcriptome in severe COPD. European Respiratory Journal. 52(4). 1701994–1701994. 21 indexed citations
7.
Bérubé, Jean‐Christophe, Nathalie Gaudreault, Maxime Lamontagne, et al.. (2017). Deleterious variants in DCHS1 are prevalent in sporadic cases of mitral valve prolapse. Molecular Genetics & Genomic Medicine. 6(1). 114–120. 7 indexed citations
8.
Matsson, Hans, Cilla Söderhäll, Elísabet Einarsdóttir, et al.. (2016). Targeted high-throughput sequencing of candidate genes for chronic obstructive pulmonary disease. BMC Pulmonary Medicine. 16(1). 146–146. 10 indexed citations
9.
Lamontagne, Maxime, Nathalie Gaudreault, Cyndi Henry, et al.. (2015). Identification of Gender-Specific Genetic Variants in Patients With Bicuspid Aortic Valve. The American Journal of Cardiology. 117(3). 420–426. 51 indexed citations
10.
Saferali, Aabida, Ma’en Obeidat, Jean‐Christophe Bérubé, et al.. (2015). Polymorphisms Associated with Expression of BPIFA1/BPIFB1 and Lung Disease Severity in Cystic Fibrosis. American Journal of Respiratory Cell and Molecular Biology. 53(5). 607–614. 26 indexed citations
11.
Guénard, Frédéric, Maxime Lamontagne, Yohan Bossé, et al.. (2015). Influences of Gestational Obesity on Associations between Genotypes and Gene Expression Levels in Offspring following Maternal Gastrointestinal Bypass Surgery for Obesity. PLoS ONE. 10(1). e0117011–e0117011. 3 indexed citations
12.
Guauque-Olarte, Sandra, David Messika‐Zeitoun, Arnaud Droit, et al.. (2015). Calcium Signaling Pathway Genes RUNX2 and CACNA1C Are Associated With Calcific Aortic Valve Disease. Circulation Cardiovascular Genetics. 8(6). 812–822. 49 indexed citations
13.
Lamontagne, Maxime, Christian Couture, Massimo Conti, et al.. (2014). Susceptibility loci for lung cancer are associated with mRNA levels of nearby genes in the lung. Carcinogenesis. 35(12). 2653–2659. 14 indexed citations
14.
Morissette, Mathieu C., Maxime Lamontagne, Jean‐Christophe Bérubé, et al.. (2014). Impact of Cigarette Smoke on the Human and Mouse Lungs: A Gene-Expression Comparison Study. PLoS ONE. 9(3). e92498–e92498. 44 indexed citations
15.
Obeidat, Ma’en, S. Miller, Charlotte K. Billington, et al.. (2013). GSTCD and INTS12 Regulation and Expression in the Human Lung. PLoS ONE. 8(9). e74630–e74630. 21 indexed citations
16.
Lamontagne, Maxime, Christian Couture, Dirkje S. Postma, et al.. (2013). Refining Susceptibility Loci of Chronic Obstructive Pulmonary Disease with Lung eqtls. PLoS ONE. 8(7). e70220–e70220. 32 indexed citations
17.
Bossé, Yohan, Dirkje S. Postma, Don D. Sin, et al.. (2012). Molecular Signature of Smoking in Human Lung Tissues. Cancer Research. 72(15). 3753–3763. 93 indexed citations
18.
Gaudreault, Nathalie, Maxime Lamontagne, Sandra Guauque-Olarte, et al.. (2011). Replication of Genetic Association Studies in Aortic Stenosis in Adults. The American Journal of Cardiology. 108(9). 1305–1310. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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