Max van Lessen

775 citations
9 papers · 535 · h-index 7

Impact in

Papers in

    • Developmental Biology and Gene Regulation 2
    • Signaling Pathways in Disease 1
    • Hippo pathway signaling and YAP/TAZ 1

Max van Lessen

8 papers receiving 529 citations

Peers

Max van Lessen
Comparison fields: 5 of 74
  • Immunology and Allergy 102
  • Neurology 79
  • Cell Biology 155
  • Cellular and Molecular Neuroscience 97
  • Molecular Biology 306
Replace Ulrich Tigges with:
Ulrich Tigges United States
Kim Paes United States
Anu Muona Finland
Barbara M. Braunger Germany
Tanya L. Henshall Australia
Alessandra Granata United Kingdom
Julia Meller United States
Harwin Sidik Singapore
Xu Hou China
Sonja Mertsch Germany
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Citations per field
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Citations per year

Countries citing papers authored by Max van Lessen

Since Specialization
Citations

This map shows the geographic impact of Max van Lessen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Max van Lessen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Max van Lessen more than expected).

Fields of papers citing papers by Max van Lessen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Max van Lessen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Max van Lessen. The network helps show where Max van Lessen may publish in the future.

Co-authors

The 25 scholars most cited alongside Max van Lessen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Max van Lessen Line = papers co-authored together Max van Lessen links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 2013194
2 2015168
3 201796
4 202238
5 201516
6 201912
7 20236
8 20205
9 20220

About Max van Lessen

Max van Lessen is a scholar working on Molecular Biology, Cell Biology, Cellular and Molecular Neuroscience, Dermatology and Immunology and Allergy, having authored 9 papers that have together received 535 indexed citations. Recurring topics across this work include Developmental Biology and Gene Regulation (2 papers), Cell Adhesion Molecules Research (2 papers), Skin Protection and Aging (2 papers), Axon Guidance and Neuronal Signaling (2 papers), Signaling Pathways in Disease (1 paper), Hippo pathway signaling and YAP/TAZ (1 paper), Galectins and Cancer Biology (1 paper) and Neuroinflammation and Neurodegeneration Mechanisms (1 paper). The work is most often cited by research in Immunology and Allergy (102 citations), Neurology (79 citations), Cell Biology (155 citations), Cellular and Molecular Neuroscience (97 citations) and Molecular Biology (306 citations). Max van Lessen has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Ralf H. Adams, Masanori Nakayama, Dietmar Vestweber, Hiroyuki Yamamoto, Stefan Schulte‐Merker, Katsuhiro Kato, Dagmar Zeuschner, Kenichi Kanai, Kozo Kaibuchi and Maike Frye. Their work appears in journals such as Nature Communications, Nature Cell Biology, Arteriosclerosis Thrombosis and Vascular Biology, Journal of Investigative Dermatology and eLife.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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