Max G. Bachem

7.6k total citations · 2 hit papers
96 papers, 6.0k citations indexed

About

Max G. Bachem is a scholar working on Molecular Biology, Surgery and Oncology. According to data from OpenAlex, Max G. Bachem has authored 96 papers receiving a total of 6.0k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Molecular Biology, 32 papers in Surgery and 31 papers in Oncology. Recurrent topics in Max G. Bachem's work include Pancreatitis Pathology and Treatment (20 papers), Liver physiology and pathology (19 papers) and Pancreatic and Hepatic Oncology Research (17 papers). Max G. Bachem is often cited by papers focused on Pancreatitis Pathology and Treatment (20 papers), Liver physiology and pathology (19 papers) and Pancreatic and Hepatic Oncology Research (17 papers). Max G. Bachem collaborates with scholars based in Germany, United States and China. Max G. Bachem's co-authors include Axel M. Gressner, Marco Siech, Guido Adler, Shaoxia Zhou, Alexandra Schmid‐Kotsas, A. Grünert, André Menke, H Weidenbach, Hans G. Beger and Hans Groß and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Gastroenterology.

In The Last Decade

Max G. Bachem

95 papers receiving 5.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identification, culture, and characterization of pancreatic stellate cells in rats and humans

1998 808 citations Max G. Bachem, André Menke et al. Gastroenterology profile →
Pancreatic carcinoma cells induce fibrosis by stimulating proliferation and matrix synthesis of stellate cells

2005 515 citations Max G. Bachem, Marco Ramadani et al. Gastroenterology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Max G. Bachem Germany	45	2.2k	2.0k	1.9k	1.1k	1.1k	96	6.0k
Rishu Takimoto Japan	40	1.6k 0.7×	777 0.4×	2.6k 1.4×	593 0.5×	760 0.7×	155	5.8k
Yutaka Nakashima Japan	38	793 0.4×	1.6k 0.8×	2.0k 1.1×	1.4k 1.2×	1.2k 1.2×	124	6.7k
Yoshikazu Kuroda Japan	42	1.7k 0.8×	3.8k 1.9×	1.2k 0.7×	896 0.8×	474 0.4×	277	6.3k
Eiso Hiyama Japan	41	1.8k 0.8×	1.3k 0.6×	4.0k 2.1×	584 0.5×	628 0.6×	230	8.8k
Hirohisa Okabe Japan	37	1.7k 0.7×	1.8k 0.9×	1.2k 0.7×	430 0.4×	1.2k 1.1×	253	4.8k
Susumu Satomi Japan	39	670 0.3×	2.8k 1.4×	1.5k 0.8×	754 0.7×	624 0.6×	215	5.7k
Yasuo Takano Japan	38	1.6k 0.7×	900 0.5×	1.5k 0.8×	667 0.6×	764 0.7×	149	4.5k
Diana Julie Leeming Denmark	48	1.2k 0.5×	1.1k 0.6×	1.8k 1.0×	640 0.6×	2.2k 2.1×	213	7.7k
Joji Kitayama Japan	49	2.7k 1.2×	1.5k 0.8×	2.9k 1.6×	857 0.7×	1.1k 1.0×	195	7.4k
Jesús M. Bañales Spain	41	1.3k 0.6×	2.4k 1.2×	2.4k 1.3×	364 0.3×	1.3k 1.3×	169	6.0k

Countries citing papers authored by Max G. Bachem

Since Specialization

Citations

This map shows the geographic impact of Max G. Bachem's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Max G. Bachem with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Max G. Bachem more than expected).

Fields of papers citing papers by Max G. Bachem

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Max G. Bachem. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Max G. Bachem. The network helps show where Max G. Bachem may publish in the future.

Co-authorship network of co-authors of Max G. Bachem

This figure shows the co-authorship network connecting the top 25 collaborators of Max G. Bachem. A scholar is included among the top collaborators of Max G. Bachem based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Max G. Bachem. Max G. Bachem is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Benihoud, Karim, Frédéric Vigant, Christoph Q. Schmidt, et al.. (2015). Hexon Modification to Improve the Activity of Oncolytic Adenovirus Vectors against Neoplastic and Stromal Cells in Pancreatic Cancer. PLoS ONE. 10(2). e0117254–e0117254. 17 indexed citations

Karpel‐Massler, Georg, Mike‐Andrew Westhoff, Shaoxia Zhou, et al.. (2013). Combined Inhibition of HER1/EGFR and RAC1 Results in a Synergistic Antiproliferative Effect on Established and Primary Cultured Human Glioblastoma Cells. Molecular Cancer Therapeutics. 12(9). 1783–1795. 48 indexed citations

Westhoff, Mike‐Andrew, Shaoxia Zhou, Lisa Nonnenmacher, et al.. (2013). Inhibition of NF-κB Signaling Ablates the Invasive Phenotype of Glioblastoma. Molecular Cancer Research. 11(12). 1611–1623. 58 indexed citations

Stadel, Dominic, Andrea Mohr, Marion MacFarlane, et al.. (2010). TRAIL-Induced Apoptosis Is Preferentially Mediated via TRAIL Receptor 1 in Pancreatic Carcinoma Cells and Profoundly Enhanced by XIAP Inhibitors. Clinical Cancer Research. 16(23). 5734–5749. 61 indexed citations

Geismann, Claudia, Dorothee Koch, Frank Bergmann, et al.. (2009). Up-regulation of L1CAM in Pancreatic Duct Cells Is Transforming Growth Factor β1– and Slug-Dependent: Role in Malignant Transformation of Pancreatic Cancer. Cancer Research. 69(10). 4517–4526. 85 indexed citations

Westhoff, Mike‐Andrew, Sijie Zhou, Max G. Bachem, Klaus‐Michael Debatin, & Simone Fulda. (2008). Identification of a novel switch in the dominant forms of cell adhesion-mediated drug resistance in glioblastoma cells. Oncogene. 27(39). 5169–5181. 54 indexed citations

Li, Ling, Max G. Bachem, Shaoxia Zhou, et al.. (2008). Pancreatitis-Associated Protein Inhibits Human Pancreatic Stellate Cell MMP-1 and -2, TIMP-1 and -2 Secretion and RECK Expression. Pancreatology. 9(1-2). 99–110. 19 indexed citations

Haug, Cornelia, et al.. (2005). Oxidized LDL inhibit hepatocyte growth factor synthesis in coronary smooth muscle cells. International Journal of Cardiology. 103(3). 298–306. 4 indexed citations

Bachem, Max G., Marco Ramadani, Marco Siech, et al.. (2005). Pancreatic carcinoma cells induce fibrosis by stimulating proliferation and matrix synthesis of stellate cells. Gastroenterology. 128(4). 907–921. 515 indexed citations breakdown →

10.

Buchholz, Malte, Hans A. Kestler, Karlheinz Holzmann, et al.. (2005). Transcriptome analysis of human hepatic and pancreatic stellate cells: organ-specific variations of a common transcriptional phenotype. Journal of Molecular Medicine. 83(10). 795–805. 93 indexed citations

11.

Zhou, Shaoxia, Andreas Schmelz, Thomas Seufferlein, et al.. (2004). Molecular Mechanisms of Low Intensity Pulsed Ultrasound in Human Skin Fibroblasts. Journal of Biological Chemistry. 279(52). 54463–54469. 205 indexed citations

12.

Köninger, Jörg, Thomas Giese, Fabio F. di Mola, et al.. (2004). Pancreatic tumor cells influence the composition of the extracellular matrix. Biochemical and Biophysical Research Communications. 322(3). 943–949. 76 indexed citations

13.

Zhao, Jinshun, et al.. (2003). Sensitivity and specificity of different staining methods to monitor apoptosis induced by oxidative stress in adherent cells.. PubMed. 116(12). 1923–9. 20 indexed citations

14.

Shen, Changxian, Holger Schirrmeister, Alexandra Schmid‐Kotsas, et al.. (2002). Liposomal Delivery of Antisense Oligonucleotides for Efficient Downregulation of Bcl-2 and Induction of Apoptosis. Cancer Biotherapy and Radiopharmaceuticals. 17(3). 281–289. 8 indexed citations

15.

Haug, Cornelia, et al.. (2001). Influence of hepatocyte growth factor, epidermal growth factor, and mycophenolic acid on endothelin‐1 synthesis in human endothelial cells. Nephrology Dialysis Transplantation. 16(12). 2310–2316. 9 indexed citations

16.

Schmid‐Kotsas, Alexandra, André Menke, Marco Siech, et al.. (2000). Platelet-Derived Growth Factors Stimulate Proliferation and Extracellular Matrix Synthesis of Pancreatic Stellate Cells: Implications in Pathogenesis of Pancreas Fibrosis. Laboratory Investigation. 80(1). 47–55. 165 indexed citations

17.

Müller, A., et al.. (1995). Immediate metabolic effects of different nutritional regimens in critically ill medical patients. Intensive Care Medicine. 21(7). 561–566. 45 indexed citations

18.

Gressner, Axel M., Nora Krull, & Max G. Bachem. (1994). Regulation of Proteoglycan Expression in Fibrotic Liver and Cultured Fat-Storing Cells. Pathology - Research and Practice. 190(9-10). 864–882. 43 indexed citations

19.

Bachem, Max G., et al.. (1992). Activation of rat liver perisinusoidal lipocytes by transforming growth factors derived from myofibroblastlike cells. A potential mechanism of self perpetuation in liver fibrogenesis.. Journal of Clinical Investigation. 89(1). 19–27. 205 indexed citations

20.

Bachem, Max G., et al.. (1989). Transforming growth factors (TGFα and TGFβH) stimulate chondroitin sulfate and hyaluronate synthesis in cultured rat liver fat storing cells. FEBS Letters. 257(1). 134–137. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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