Maw-Shung Liu

993 total citations
50 papers, 800 citations indexed

About

Maw-Shung Liu is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Maw-Shung Liu has authored 50 papers receiving a total of 800 indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 15 papers in Pathology and Forensic Medicine and 15 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Maw-Shung Liu's work include Cardiac electrophysiology and arrhythmias (13 papers), Ion channel regulation and function (11 papers) and Receptor Mechanisms and Signaling (11 papers). Maw-Shung Liu is often cited by papers focused on Cardiac electrophysiology and arrhythmias (13 papers), Ion channel regulation and function (11 papers) and Receptor Mechanisms and Signaling (11 papers). Maw-Shung Liu collaborates with scholars based in United States, Taiwan and Russia. Maw-Shung Liu's co-authors include Li‐Ling Wu, Chaoshu Tang, Sanjay Ghosh, Rei‐Cheng Yang, Hseng‐Kuang Hsu, K. J. Kako, Chin Hsu, Yong Ji, Haruo Takeda and Chaoshu Tang and has published in prestigious journals such as Hepatology, Biochemical Journal and Biochemical and Biophysical Research Communications.

In The Last Decade

Maw-Shung Liu

50 papers receiving 768 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Maw-Shung Liu United States 17 331 215 208 119 99 50 800
Xiuxiu Lv China 14 297 0.9× 177 0.8× 126 0.6× 52 0.4× 98 1.0× 26 701
P.‐O. Sjöquist Sweden 19 263 0.8× 74 0.3× 232 1.1× 288 2.4× 47 0.5× 36 876
Jorge G. Peralta Argentina 13 354 1.1× 115 0.5× 82 0.4× 321 2.7× 47 0.5× 20 749
Jia Shi China 17 627 1.9× 259 1.2× 54 0.3× 127 1.1× 132 1.3× 69 1.2k
Olena Oliyarnyk Czechia 18 367 1.1× 177 0.8× 88 0.4× 253 2.1× 46 0.5× 48 1.0k
Yuxiao Sun China 12 444 1.3× 301 1.4× 56 0.3× 63 0.5× 87 0.9× 34 826
Murat Giriş Türkiye 18 190 0.6× 92 0.4× 65 0.3× 123 1.0× 70 0.7× 35 836
Jiankai Zhong China 13 321 1.0× 159 0.7× 83 0.4× 100 0.8× 39 0.4× 19 584
Hiroshi Tsuchihashi Japan 17 330 1.0× 149 0.7× 67 0.3× 157 1.3× 73 0.7× 77 779

Countries citing papers authored by Maw-Shung Liu

Since Specialization
Citations

This map shows the geographic impact of Maw-Shung Liu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maw-Shung Liu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maw-Shung Liu more than expected).

Fields of papers citing papers by Maw-Shung Liu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maw-Shung Liu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maw-Shung Liu. The network helps show where Maw-Shung Liu may publish in the future.

Co-authorship network of co-authors of Maw-Shung Liu

This figure shows the co-authorship network connecting the top 25 collaborators of Maw-Shung Liu. A scholar is included among the top collaborators of Maw-Shung Liu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maw-Shung Liu. Maw-Shung Liu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Maw-Shung, et al.. (2013). Bioenergetic failure correlates with autophagy and apoptosis in rat liver following silver nanoparticle intraperitoneal administration. Particle and Fibre Toxicology. 10(1). 40–40. 73 indexed citations
2.
Liu, Maw-Shung, et al.. (2012). Attenuation of Mitochondrial Unfolded Protein Response is Associated With Hepatic Dysfunction in Septic Rats. Shock. 38(6). 642–648. 9 indexed citations
3.
Liu, Maw-Shung, et al.. (2012). Changes in group II phospholipase A2 gene expression in rat heart during sepsis. Journal of Surgical Research. 181(2). 272–278. 3 indexed citations
4.
Liu, Maw-Shung, et al.. (2012). Antisense inhibition of secretory and cytosolic phospholipase A2 reduces the mortality in rats with sepsis*. Critical Care Medicine. 40(7). 2132–2140. 12 indexed citations
5.
Hsu, Chin, Guang Wu, Hseng‐Kuang Hsu, et al.. (2007). Intracellular Redistribution of Dihydropyridine Receptor in the Rat Heart During the Progression of Sepsis. Journal of Surgical Research. 141(2). 146–152. 8 indexed citations
6.
Wu, Guang, Chin Hsu, Rei‐Cheng Yang, et al.. (2004). TRANSCRIPTIONAL REGULATION OF CARDIAC SARCOPLASMIC RETICULUM CALCIUM-ATPase GENE DURING THE PROGRESSION OF SEPSIS. Shock. 22(1). 46–50. 11 indexed citations
7.
Wu, Li‐Ling, et al.. (2002). Altered Phospholamban-Calcium ATPase Interaction in Cardiac Sarcoplasmic Reticulum During the Progression of Sepsis. Shock. 17(5). 389–393. 30 indexed citations
8.
Wu, Li‐Ling, et al.. (2001). CALCIUM UPTAKE BY SARCOPLASMIC RETICULUM IS IMPAIRED DURING THE HYPODYNAMIC PHASE OF SEPSIS IN THE RAT HEART. Shock. 15(1). 49–55. 45 indexed citations
10.
Yang, Jun, et al.. (1999). Transcriptional regulation of α1-adrenoceptor gene in the rat liver during different phases of sepsis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1453(2). 207–215. 16 indexed citations
11.
Tang, Chaoshu, Jun Yang, & Maw-Shung Liu. (1998). Progressive internalization of β-adrenoceptors in the rat liver during different phases of sepsis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1407(3). 225–233. 5 indexed citations
12.
Hsu, Chin, et al.. (1997). Differential Activation of Protein Kinase A in Various Regions of Myocardium during Sepsis. Journal of Surgical Research. 71(2). 161–165. 3 indexed citations
13.
Hsu, Chin, et al.. (1997). PROTEIN KINASE A ACTIVITY IS INCREASED IN RAT HEART DURING LATE HYPODYNAMIC PHASE OF SEPSIS. Shock. 8(1). 68–72. 12 indexed citations
14.
Hwang, Tsann‐Long, Ying‐Tung Lau, Ming-Ming Tsai, & Maw-Shung Liu. (1997). Changes of adenosine triphosphate-dependent calcium uptake in microsomal fractions of rat liver during sepsis. Surgery. 121(6). 662–667. 2 indexed citations
15.
Lau, Ying‐Tung, et al.. (1992). Calcium transport by rat liver plasma membranes during sepsis.. PubMed. 38(4). 238–44. 4 indexed citations
16.
Clark, B. Ruth & Maw-Shung Liu. (1991). Kinetic studies on the biological activity of calmodulin in canine heart and liver during endotoxin shock. Canadian Journal of Physiology and Pharmacology. 69(11). 1670–1676. 1 indexed citations
17.
Wu, Li‐Ling & Maw-Shung Liu. (1991). Impaired calcium uptake by cardiac sarcoplasmic reticulum and its underlying mechanism in endotoxin shock. Molecular and Cellular Biochemistry. 108(1). 9–17. 18 indexed citations
18.
Liu, Maw-Shung, et al.. (1986). Myocardial sodium pump activity in endotoxin shock.. PubMed. 19(2). 177–84. 5 indexed citations
19.
Liu, Maw-Shung & Haruo Takeda. (1982). Endotoxin-induced stimulation on phospholipase A activities in dog hearts. Biochemical Medicine. 28(1). 62–69. 15 indexed citations
20.
Liu, Maw-Shung & K. J. Kako. (1974). Characteristics of mitochondrial and microsomal monoacyl- and diacyl-glycerol 3-phosphate biosynthesis in rabbit heart. Biochemical Journal. 138(1). 11–21. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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