Matthias Muhar
Impact in
- Molecular Biology top 5%
- Genomics and Chromatin Dynamics
- RNA Research and Splicing
- RNA modifications and cancer
- RNA and protein synthesis mechanisms
- Protein Degradation and Inhibitors
- Ubiquitin and proteasome pathways
- Epigenetics and DNA Methylation
- CRISPR and Genetic Engineering
- Cancer Research top 10%
Papers in
-
- Protein Degradation and Inhibitors 6
- Ubiquitin and proteasome pathways 5
- Genomics and Chromatin Dynamics 4
- CRISPR and Genetic Engineering 3
- RNA Interference and Gene Delivery 2
- Oncology 2
- Peptidase Inhibition and Analysis 2
- Co-authors
- Johannes Zuber (10 shared papers)Thomas Hoffmann (2 shared papers)David A. Cisneros (2 shared papers)Stefan Schoenfelder (2 shared papers)Gordana Wutz (2 shared papers)Wen Tang (2 shared papers)Peter Fraser (2 shared papers)Jan‐Michael Peters (2 shared papers)
- Journals
- Cell Reports (2 papers)Nature Communications (2 papers)PLoS ONE (1 paper)The EMBO Journal (1 paper)BMC Bioinformatics (1 paper)
- Partner nations
- AustriaUnited StatesGermany
In The Last Decade
Matthias Muhar
14 papers receiving 2.0k citations
Matthias Muhar's Hit Papers
Peers
Comparison fields: 5 of 89
- Molecular Biology 1.6k
- Cancer Research 170
- Cell Biology 183
- Hematology 109
- Aging 17
Countries citing papers authored by Matthias Muhar
This map shows the geographic impact of Matthias Muhar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthias Muhar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthias Muhar more than expected).
Fields of papers citing papers by Matthias Muhar
This network shows the impact of papers produced by Matthias Muhar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthias Muhar. The network helps show where Matthias Muhar may publish in the future.
Co-authors
The 25 scholars most cited alongside Matthias Muhar, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Topologically associating domains and chromatin loops depend on cohesin and are regulated by CTCF, WAPL, and PDS5 proteins Hit paper breakdown → | 2017 | 567 |
| 2 | 2013 | 455 | |
| 3 | 2018 | 247 | |
| 4 | 2020 | 218 | |
| 5 | 2016 | 141 | |
| 6 | 2020 | 115 | |
| 7 | 2019 | 92 | |
| 8 | 2013 | 55 | |
| 9 | 2018 | 44 | |
| 10 | 2021 | 25 | |
| 11 | 2021 | 16 | |
| 12 | 2025 | 11 | |
| 13 | 2023 | 10 | |
| 14 | 2013 | 7 |
About Matthias Muhar
Matthias Muhar is a scholar working on Molecular Biology, Oncology, Epidemiology, Hematology and Cell Biology, having authored 14 papers that have together received 2.0k indexed citations. Recurring topics across this work include Protein Degradation and Inhibitors (6 papers), Ubiquitin and proteasome pathways (5 papers), Genomics and Chromatin Dynamics (4 papers), CRISPR and Genetic Engineering (3 papers), Acute Myeloid Leukemia Research (2 papers), Peptidase Inhibition and Analysis (2 papers), RNA Interference and Gene Delivery (2 papers) and Autophagy in Disease and Therapy (2 papers). The work is most often cited by research in Molecular Biology (1.6k citations), Cancer Research (170 citations), Cell Biology (183 citations), Hematology (109 citations) and Aging (17 citations). Matthias Muhar has collaborated with scholars based in Austria, United States and Germany. Frequent co-authors include Johannes Zuber, Thomas Hoffmann, David A. Cisneros, Stefan Schoenfelder, Gordana Wutz, Wen Tang, Peter Fraser, Jan‐Michael Peters, Roman R. Stocsits and Mareike Roth. Their work appears in journals such as Cell Reports, Nature Communications, PLoS ONE, The EMBO Journal and BMC Bioinformatics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.