Matthew P. Keasey

835 total citations
22 papers, 627 citations indexed

About

Matthew P. Keasey is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Matthew P. Keasey has authored 22 papers receiving a total of 627 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 6 papers in Neurology. Recurrent topics in Matthew P. Keasey's work include Neuroinflammation and Neurodegeneration Mechanisms (6 papers), Neurogenesis and neuroplasticity mechanisms (6 papers) and MicroRNA in disease regulation (4 papers). Matthew P. Keasey is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (6 papers), Neurogenesis and neuroplasticity mechanisms (6 papers) and MicroRNA in disease regulation (4 papers). Matthew P. Keasey collaborates with scholars based in United States, Brazil and United Kingdom. Matthew P. Keasey's co-authors include Theo Hagg, Cuihong Jia, Seong Su Kang, Jianping Cai, Sang Soo Kang, Stephen Kelly, João Ricardo Mendes de Oliveira, James B. Uney, Rollie Reid and Antonio Souto and has published in prestigious journals such as Journal of Neuroscience, Stroke and Scientific Reports.

In The Last Decade

Matthew P. Keasey

22 papers receiving 613 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matthew P. Keasey United States 16 278 147 132 117 69 22 627
Tomoko Hisaoka Japan 13 291 1.0× 181 1.2× 50 0.4× 124 1.1× 101 1.5× 21 632
Elizabeth A. Novotny United States 16 669 2.4× 330 2.2× 58 0.4× 155 1.3× 147 2.1× 22 1.4k
Cassandre Labelle‐Dumais United States 17 533 1.9× 223 1.5× 100 0.8× 81 0.7× 253 3.7× 29 1.2k
K. M�llg�rd Denmark 21 481 1.7× 372 2.5× 267 2.0× 223 1.9× 67 1.0× 29 1.1k
Kaya J.E. Matson United States 8 560 2.0× 190 1.3× 170 1.3× 126 1.1× 36 0.5× 9 994
Taeko Ichise Japan 11 504 1.8× 360 2.4× 108 0.8× 66 0.6× 59 0.9× 14 849
Yoojin Choi United States 8 413 1.5× 255 1.7× 27 0.2× 110 0.9× 107 1.6× 11 877
Mark E. Basham United States 11 138 0.5× 177 1.2× 55 0.4× 44 0.4× 52 0.8× 11 658
Noel L. Wys United States 8 167 0.6× 125 0.9× 88 0.7× 56 0.5× 23 0.3× 8 564
Cristina Garcı́a-Frigola Spain 12 590 2.1× 517 3.5× 106 0.8× 279 2.4× 91 1.3× 16 1.0k

Countries citing papers authored by Matthew P. Keasey

Since Specialization
Citations

This map shows the geographic impact of Matthew P. Keasey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew P. Keasey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew P. Keasey more than expected).

Fields of papers citing papers by Matthew P. Keasey

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew P. Keasey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew P. Keasey. The network helps show where Matthew P. Keasey may publish in the future.

Co-authorship network of co-authors of Matthew P. Keasey

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew P. Keasey. A scholar is included among the top collaborators of Matthew P. Keasey based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew P. Keasey. Matthew P. Keasey is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Keasey, Matthew P., et al.. (2022). Liver vitronectin release into the bloodstream increases due to reduced vagal muscarinic signaling after cerebral stroke in female mice. Physiological Reports. 10(9). e15301–e15301. 2 indexed citations
2.
Jia, Cuihong, et al.. (2020). Blood Vitronectin Induces Detrimental Brain Interleukin-6 and Correlates With Outcomes After Stroke Only in Female Mice. Stroke. 51(5). 1587–1595. 13 indexed citations
3.
Jia, Cuihong, et al.. (2019). Vitronectin mitigates stroke-increased neurogenesis only in female mice and through FAK-regulated IL-6. Experimental Neurology. 323. 113088–113088. 10 indexed citations
4.
Jia, Cuihong, et al.. (2018). Ciliary neurotrophic factor is a key sex-specific regulator of depressive-like behavior in mice. Psychoneuroendocrinology. 100. 96–105. 23 indexed citations
5.
Jia, Cuihong, et al.. (2018). Vitronectin from brain pericytes promotes adult forebrain neurogenesis by stimulating CNTF. Experimental Neurology. 312. 20–32. 23 indexed citations
6.
Jia, Cuihong, et al.. (2018). Inhibition of astrocyte FAK–JNK signaling promotes subventricular zone neurogenesis through CNTF. Glia. 66(11). 2456–2469. 27 indexed citations
7.
Keasey, Matthew P., et al.. (2017). MiR-9-5p Down-Regulates PiT2, but not PiT1 in Human Embryonic Kidney 293 Cells. Journal of Molecular Neuroscience. 62(1). 28–33. 2 indexed citations
8.
Keasey, Matthew P., et al.. (2017). Blood vitronectin is a major activator of LIF and IL-6 in the brain through integrin–FAK and uPAR signaling. Journal of Cell Science. 131(3). 25 indexed citations
9.
Visavadiya, Nishant P., Matthew P. Keasey, Kalpita Banerjee, et al.. (2016). Integrin-FAK signaling rapidly and potently promotes mitochondrial function through STAT3. Cell Communication and Signaling. 14(1). 32–32. 65 indexed citations
10.
Keasey, Matthew P., et al.. (2016). Vitamin-D receptor agonist calcitriol reduces calcification in vitro through selective upregulation of SLC20A2 but not SLC20A1 or XPR1. Scientific Reports. 6(1). 25802–25802. 22 indexed citations
11.
Keasey, Matthew P., Helen L. Scott, Ioannis Bantounas, James B. Uney, & Stephen Kelly. (2016). MiR-132 Is Upregulated by Ischemic Preconditioning of Cultured Hippocampal Neurons and Protects them from Subsequent OGD Toxicity. Journal of Molecular Neuroscience. 59(3). 404–410. 19 indexed citations
12.
Bezerra, Bruna M., Matthew P. Keasey, Nicola Schiel, & Antonio Souto. (2014). Responses towards a dying adult group member in a wild New World monkey. Primates. 55(2). 185–188. 24 indexed citations
13.
Keasey, Matthew P., Matheus Fernandes de Oliveira, Nina Jensen, et al.. (2014). First Report of a De Novo Mutation at SLC20A2 in a Patient with Brain Calcification. Journal of Molecular Neuroscience. 54(4). 748–751. 27 indexed citations
14.
Bezerra, Bruna M., Antonio Souto, Matthew P. Keasey, et al.. (2014). Camera Trap Observations of Nonhabituated Critically Endangered Wild Blonde Capuchins, Sapajus flavius (Formerly Cebus flavius). International Journal of Primatology. 35(5). 895–907. 38 indexed citations
15.
Lemos, Roberta R., et al.. (2013). An Update on Primary Familial Brain Calcification. International review of neurobiology. 110. 349–371. 19 indexed citations
16.
Keasey, Matthew P., et al.. (2013). Inhibition of a novel specific neuroglial integrin signaling pathway increases STAT3-mediated CNTF expression. Cell Communication and Signaling. 11(1). 35–35. 31 indexed citations
17.
Kang, Sang Soo, Matthew P. Keasey, Jianping Cai, & Theo Hagg. (2012). Loss of Neuron-Astroglial Interaction Rapidly Induces Protective CNTF Expression after Stroke in Mice. Journal of Neuroscience. 32(27). 9277–9287. 56 indexed citations
18.
Kang, Seong Su, et al.. (2012). Endogenous CNTF mediates stroke-induced adult CNS neurogenesis in mice. Neurobiology of Disease. 49. 68–78. 69 indexed citations
19.
Kelly, Stephen, et al.. (2009). Identifying Neural Progenitor Cells in the Adult Brain. Methods in molecular biology. 549. 217–230. 2 indexed citations
20.
Howarth, Joanna, Stephen Kelly, Matthew P. Keasey, et al.. (2007). Hsp40 Molecules That Target to the Ubiquitin-proteasome System Decrease Inclusion Formation in Models of Polyglutamine Disease. Molecular Therapy. 15(6). 1100–1105. 88 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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