Matthew J. Toth

1.7k total citations
20 papers, 1.3k citations indexed

About

Matthew J. Toth is a scholar working on Molecular Biology, Surgery and Genetics. According to data from OpenAlex, Matthew J. Toth has authored 20 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 3 papers in Surgery and 3 papers in Genetics. Recurrent topics in Matthew J. Toth's work include Peroxisome Proliferator-Activated Receptors (4 papers), RNA and protein synthesis mechanisms (4 papers) and Mitochondrial Function and Pathology (3 papers). Matthew J. Toth is often cited by papers focused on Peroxisome Proliferator-Activated Receptors (4 papers), RNA and protein synthesis mechanisms (4 papers) and Mitochondrial Function and Pathology (3 papers). Matthew J. Toth collaborates with scholars based in United States, Switzerland and Canada. Matthew J. Toth's co-authors include Paul Schimmel, Luis B. Agellon, Galya Vassileva, A. B. R. Thomson, Ken L. Chambliss, Debra D. Hinson, Ryouichi Tanaka, Michael Schlame, K. Michael Gibson and Frédéric M. Vaz and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Journal of Molecular Biology.

In The Last Decade

Matthew J. Toth

20 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matthew J. Toth United States 17 1.0k 186 153 134 122 20 1.3k
Ken Karasawa Japan 18 413 0.4× 92 0.5× 121 0.8× 213 1.6× 81 0.7× 49 947
Akira Imaizumi Japan 22 924 0.9× 80 0.4× 289 1.9× 94 0.7× 123 1.0× 74 1.5k
J.C. Komen Netherlands 14 642 0.6× 232 1.2× 175 1.1× 73 0.5× 35 0.3× 16 879
C L Kiang United States 11 784 0.8× 193 1.0× 351 2.3× 92 0.7× 27 0.2× 16 1.2k
T D Chrisman United States 18 735 0.7× 50 0.3× 201 1.3× 121 0.9× 56 0.5× 22 1.2k
Sven W. Sauer Germany 14 454 0.4× 182 1.0× 88 0.6× 45 0.3× 54 0.4× 17 858
Katrina J. Binger Australia 21 551 0.5× 95 0.5× 305 2.0× 105 0.8× 33 0.3× 35 1.2k
Ronald J. Holewinski United States 22 833 0.8× 66 0.4× 164 1.1× 73 0.5× 41 0.3× 41 1.3k
Charles M. Mansbach United States 25 736 0.7× 69 0.4× 334 2.2× 609 4.5× 113 0.9× 59 1.8k
Marina Mojena Spain 19 630 0.6× 29 0.2× 92 0.6× 163 1.2× 134 1.1× 47 1.3k

Countries citing papers authored by Matthew J. Toth

Since Specialization
Citations

This map shows the geographic impact of Matthew J. Toth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Toth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Toth more than expected).

Fields of papers citing papers by Matthew J. Toth

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Toth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Toth. The network helps show where Matthew J. Toth may publish in the future.

Co-authorship network of co-authors of Matthew J. Toth

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Toth. A scholar is included among the top collaborators of Matthew J. Toth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Toth. Matthew J. Toth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ren, Mindong, et al.. (2019). A Bayesian Analysis to Determine the Prevalence of Barth Syndrome in the Pediatric Population. The Journal of Pediatrics. 217. 139–144. 35 indexed citations
2.
Toth, Matthew J., Richard S. Yoon, Frank A. Liporace, & Kenneth J. Koval. (2017). What’s new in ankle fractures. Injury. 48(10). 2035–2041. 30 indexed citations
3.
4.
Clarke, Sarah L. N., Ann Bowron, Iris L. Gonzalez, et al.. (2013). Barth syndrome. Orphanet Journal of Rare Diseases. 8(1). 23–23. 269 indexed citations
5.
Acehan, Devrim, Frédéric M. Vaz, Riekelt H. Houtkooper, et al.. (2010). Cardiac and Skeletal Muscle Defects in a Mouse Model of Human Barth Syndrome. Journal of Biological Chemistry. 286(2). 899–908. 207 indexed citations
6.
Soustek, Meghan S., Darin J. Falk, Cathryn Mah, et al.. (2010). Characterization of a Transgenic Short Hairpin RNA-Induced Murine Model of Tafazzin Deficiency. Human Gene Therapy. 22(7). 865–871. 93 indexed citations
7.
Agellon, Luis B., Laurie Drozdowski, Lena Li, et al.. (2007). Loss of intestinal fatty acid binding protein increases the susceptibility of male mice to high fat diet-induced fatty liver. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1771(10). 1283–1288. 23 indexed citations
8.
Agellon, Luis B., Matthew J. Toth, & A. B. R. Thomson. (2002). Intracellular lipid binding proteins of the small intestine. Molecular and Cellular Biochemistry. 239(1-2). 79–82. 68 indexed citations
9.
Honer, Christian, Ping Chen, Matthew J. Toth, & Christoph Schumacher. (2001). Identification of SCAN dimerization domains in four gene families. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1517(3). 441–448. 16 indexed citations
10.
Vassileva, Galya, et al.. (2000). The intestinal fatty acid binding protein is not essential for dietary fat absorption in mice. The FASEB Journal. 14(13). 2040–2046. 146 indexed citations
11.
Zhang, Xiaolu, William C. Boyar, Matthew J. Toth, Lawrence P. Wennogle, & Nina C. Gonnella. (1997). Structural definition of the C5a C terminus by two-dimensional nuclear magnetic resonance spectroscopy. Proteins Structure Function and Bioinformatics. 28(2). 261–267. 66 indexed citations
12.
Hinson, Debra D., Ken L. Chambliss, Matthew J. Toth, Ryouichi Tanaka, & K. Michael Gibson. (1997). Post-translational regulation of mevalonate kinase by intermediates of the cholesterol and nonsterol isoprene biosynthetic pathways. Journal of Lipid Research. 38(11). 2216–2223. 98 indexed citations
13.
Toth, Matthew J., et al.. (1996). Purification of Rat Liver Mevalonate Pyrophosphate Decarboxylase. Preparative Biochemistry & Biotechnology. 26(1). 47–51. 15 indexed citations
14.
Toth, Matthew J., et al.. (1996). Molecular Cloning and Expression of the cDNAs Encoding Human and Yeast Mevalonate Pyrophosphate Decarboxylase. Journal of Biological Chemistry. 271(14). 7895–7898. 49 indexed citations
15.
Toth, Matthew J., William C. Boyar, Albert Braunwalder, et al.. (1994). The pharmacophore of the human C5a anaphylatoxin. Protein Science. 3(8). 1159–1168. 33 indexed citations
16.
Toth, Matthew J. & Paul Schimmel. (1990). Deletions in the large (beta) subunit of a hetero-oligomeric aminoacyl-tRNA synthetase.. Journal of Biological Chemistry. 265(2). 1000–1004. 22 indexed citations
17.
Toth, Matthew J. & Paul Schimmel. (1990). A mutation in the small (alpha) subunit of glycyl-tRNA synthetase affects amino acid activation and subunit association parameters.. Journal of Biological Chemistry. 265(2). 1005–1009. 22 indexed citations
18.
Toth, Matthew J., Emanuel J. Murgola, & Paul Schimmel. (1988). Evidence for a unique first position codon-anticodon mismatch in vivo. Journal of Molecular Biology. 201(2). 451–454. 46 indexed citations
19.
Toth, Matthew J. & Paul Schimmel. (1986). Internal structural features of E. coli glycyl-tRNA synthetase examined by subunit polypeptide chain fusions.. Journal of Biological Chemistry. 261(15). 6643–6646. 42 indexed citations
20.
Taylor, Dean P., et al.. (1983). Physical characterization of plasmids isolated fromStreptosporangium. Plasmid. 9(3). 240–246. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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