Matthew D. Howell

1.2k total citations
29 papers, 934 citations indexed

About

Matthew D. Howell is a scholar working on Molecular Biology, Genetics and Surgery. According to data from OpenAlex, Matthew D. Howell has authored 29 papers receiving a total of 934 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 10 papers in Genetics and 5 papers in Surgery. Recurrent topics in Matthew D. Howell's work include Neurogenetic and Muscular Disorders Research (10 papers), RNA modifications and cancer (9 papers) and RNA Research and Splicing (5 papers). Matthew D. Howell is often cited by papers focused on Neurogenetic and Muscular Disorders Research (10 papers), RNA modifications and cancer (9 papers) and RNA Research and Splicing (5 papers). Matthew D. Howell collaborates with scholars based in United States, Canada and United Kingdom. Matthew D. Howell's co-authors include Ravindra Singh, Natalia N. Singh, Paul E. Gottschall, Eric W. Ottesen, Joonbae Seo, Senthilkumar Sivanesan, Elizabeth M. Whitley, Marc Freichel, Kevin D. Phelan and Sung W. Rhee and has published in prestigious journals such as Nucleic Acids Research, The Journal of Immunology and PLoS ONE.

In The Last Decade

Matthew D. Howell

27 papers receiving 929 citations

Peers

Matthew D. Howell

GENET

CMN

SURGE

Tara Martinez United States

Hidemasa Kato Japan

Johann Böhm France

Catia Andreassi United Kingdom

Teuta Domi Italy

Laura Viltono Italy

Martina Nardini Italy

David R. Piper United States

Shinji Kusakawa Japan

Jong-Sun Kang South Korea

Tara Martinez United States

Matthew D. Howell

581 ×0.9

422 ×1.0

GENET

209 ×1.3

CMN

143 ×1.0

SURGE

56 ×0.6

Citations per year, relative to Matthew D. Howell Matthew D. Howell (= 1×) peers Tara Martinez

Countries citing papers authored by Matthew D. Howell

Since Specialization

Citations

This map shows the geographic impact of Matthew D. Howell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew D. Howell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew D. Howell more than expected).

Fields of papers citing papers by Matthew D. Howell

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew D. Howell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew D. Howell. The network helps show where Matthew D. Howell may publish in the future.

Co-authorship network of co-authors of Matthew D. Howell

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew D. Howell. A scholar is included among the top collaborators of Matthew D. Howell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew D. Howell. Matthew D. Howell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Johnson, Peter N., et al.. (2025). Conservation aquaculture of wild-origin offspring preserves genetic diversity in an endangered population of white sturgeon. Conservation Genetics. 26(2). 335–346. 1 indexed citations

Crossman, James A., et al.. (2025). High densities of hatchery‐origin white sturgeon suppress somatic growth rates of an endangered wild population. Ecological Applications. 35(4). e70042–e70042. 2 indexed citations

Crossman, James A., et al.. (2023). Competition overwhelms environment and genetic effects on growth rates of endangered white sturgeon from a conservation aquaculture program. Canadian Journal of Fisheries and Aquatic Sciences. 80(6). 958–977. 9 indexed citations

Nguyen, Khiem, Yiming Jin, Matthew D. Howell, et al.. (2023). Mutations to the BTN2A1 Linker Region Impact Its Homodimerization and Its Cytoplasmic Interaction with Phospho-Antigen–Bound BTN3A1. The Journal of Immunology. 211(1). 23–33. 7 indexed citations

Howell, Matthew D., et al.. (2022). Exploring synergy and its role in antimicrobial peptide biology. Methods in enzymology on CD-ROM/Methods in enzymology. 663. 99–130. 11 indexed citations

Howell, Matthew D., Eric W. Ottesen, Natalia N. Singh, et al.. (2017). TIA1 is a gender-specific disease modifier of a mild mouse model of spinal muscular atrophy. Scientific Reports. 7(1). 7183–7183. 25 indexed citations

Singh, Natalia N., et al.. (2017). Activation of a cryptic 5′ splice site reverses the impact of pathogenic splice site mutations in the spinal muscular atrophy gene. Nucleic Acids Research. 45(21). 12214–12240. 27 indexed citations

Singh, Natalia N., Matthew D. Howell, Elliot J. Androphy, & Ravindra Singh. (2017). How the discovery of ISS-N1 led to the first medical therapy for spinal muscular atrophy. Iowa State University Digital Repository (Iowa State University). 1 indexed citations

Howell, Matthew D., Eric W. Ottesen, Natalia N. Singh, Rachel Anderson, & Ravindra Singh. (2017). Gender-Specific Amelioration of SMA Phenotype upon Disruption of a Deep Intronic Structure by an Oligonucleotide. Molecular Therapy. 25(6). 1328–1341. 25 indexed citations

10.

Singh, Ravindra, Matthew D. Howell, Eric W. Ottesen, & Natalia N. Singh. (2017). Diverse role of survival motor neuron protein. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1860(3). 299–315. 214 indexed citations

11.

Ottesen, Eric W., Matthew D. Howell, Natalia N. Singh, et al.. (2016). Severe impairment of male reproductive organ development in a low SMN expressing mouse model of spinal muscular atrophy. Scientific Reports. 6(1). 20193–20193. 53 indexed citations

12.

Gottschall, Paul E. & Matthew D. Howell. (2015). ADAMTS expression and function in central nervous system injury and disorders. Matrix Biology. 44-46. 70–76. 68 indexed citations

13.

Howell, Matthew D., et al.. (2015). Hippocampal administration of chondroitinase ABC increases plaque-adjacent synaptic marker and diminishes amyloid burden in aged APPswe/PS1dE9 mice. Acta Neuropathologica Communications. 3(1). 54–54. 44 indexed citations

14.

Seo, Joonbae, et al.. (2014). A Short Antisense Oligonucleotide Ameliorates Symptoms of Severe Mouse Models of Spinal Muscular Atrophy. Molecular Therapy — Nucleic Acids. 3. e174–e174. 53 indexed citations

15.

Seo, Joonbae, Matthew D. Howell, Natalia N. Singh, & Ravindra Singh. (2013). Spinal muscular atrophy: An update on therapeutic progress. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(12). 2180–2190. 58 indexed citations

16.

Sivanesan, Senthilkumar, Matthew D. Howell, Christine J. DiDonato, & Ravindra Singh. (2013). Antisense oligonucleotide mediated therapy of spinal muscular atrophy. Translational Neuroscience. 4(1). 1–7. 36 indexed citations

17.

Phelan, Kevin D., Joel Abramowitz, Hong Wu, et al.. (2012). Canonical Transient Receptor Channel 5 (TRPC5) and TRPC1/4 Contribute to Seizure and Excitotoxicity by Distinct Cellular Mechanisms. Molecular Pharmacology. 83(2). 429–438. 105 indexed citations

18.

Howell, Matthew D. & Paul E. Gottschall. (2012). Lectican proteoglycans, their cleaving metalloproteinases, and plasticity in the central nervous system extracellular microenvironment. Neuroscience. 217. 6–18. 54 indexed citations

19.

Gottschall, Paul E., et al.. (2010). Panel of synaptic protein ELISAs for evaluating neurological phenotype. Experimental Brain Research. 201(4). 885–893. 22 indexed citations

20.

Howell, Matthew D. & Robert D. Powers. (2006). Utility of thrombocytopenia as a marker for heparin allergy in adult ED patients. The American Journal of Emergency Medicine. 24(3). 268–270. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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