Matthew A. Bill

1.2k total citations
17 papers, 837 citations indexed

About

Matthew A. Bill is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Matthew A. Bill has authored 17 papers receiving a total of 837 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Oncology and 5 papers in Immunology. Recurrent topics in Matthew A. Bill's work include Pulmonary Hypertension Research and Treatments (4 papers), Cytokine Signaling Pathways and Interactions (3 papers) and Protein Tyrosine Phosphatases (2 papers). Matthew A. Bill is often cited by papers focused on Pulmonary Hypertension Research and Treatments (4 papers), Cytokine Signaling Pathways and Interactions (3 papers) and Protein Tyrosine Phosphatases (2 papers). Matthew A. Bill collaborates with scholars based in United States, India and Germany. Matthew A. Bill's co-authors include Gregory B. Lesinski, Gregory Young, William E. Carson, James R. Fuchs, Courtney E. Bakan, Don M. Benson, Roham T. Zamanian, Janel Long‐Boyle, Valerie Scott and François Haddad and has published in prestigious journals such as Journal of Clinical Oncology, PLoS ONE and Circulation Research.

In The Last Decade

Matthew A. Bill

17 papers receiving 830 citations

Peers

Matthew A. Bill

ONCOL

PRM

IMMUN

CCM

Xinzheng Dai China

Haitao Chen China

Xiao Zhou China

Xianghong Yang China

Daliu Min China

Jiaming Huang China

Zhan‐Qi Cao China

Diptendra Kumar Sarkar India

Saswati Hazra United States

Xinzheng Dai China

Matthew A. Bill

407 ×1.2

196 ×0.9

ONCOL

144 ×0.6

PRM

126 ×0.6

IMMUN

17 ×0.2

CCM

Citations per year, relative to Matthew A. Bill Matthew A. Bill (= 1×) peers Xinzheng Dai

Countries citing papers authored by Matthew A. Bill

Since Specialization

Citations

This map shows the geographic impact of Matthew A. Bill's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew A. Bill with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew A. Bill more than expected).

Fields of papers citing papers by Matthew A. Bill

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew A. Bill. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew A. Bill. The network helps show where Matthew A. Bill may publish in the future.

Co-authorship network of co-authors of Matthew A. Bill

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew A. Bill. A scholar is included among the top collaborators of Matthew A. Bill based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew A. Bill. Matthew A. Bill is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Hennigs, Jan K., Aiqin Cao, Caiyun G. Li, et al.. (2020). PPARγ-p53-Mediated Vasculoregenerative Program to Reverse Pulmonary Hypertension. Circulation Research. 128(3). 401–418. 58 indexed citations

Blum, Lisa K., Andrew J. Sweatt, Matthew A. Bill, et al.. (2018). Circulating plasmablasts are elevated and produce pathogenic anti‐endothelial cell autoantibodies in idiopathic pulmonary arterial hypertension. European Journal of Immunology. 48(5). 874–884. 28 indexed citations

Spiekerkoetter, Edda, Yon K. Sung, Valerie Scott, et al.. (2017). Randomised placebo-controlled safety and tolerability trial of FK506 (tacrolimus) for pulmonary arterial hypertension. European Respiratory Journal. 50(3). 1602449–1602449. 125 indexed citations

Tojais, Nancy F., Aiqin Cao, Ying‐Ju Lai, et al.. (2017). Codependence of Bone Morphogenetic Protein Receptor 2 and Transforming Growth Factor-β in Elastic Fiber Assembly and Its Perturbation in Pulmonary Arterial Hypertension. Arteriosclerosis Thrombosis and Vascular Biology. 37(8). 1559–1569. 37 indexed citations

Yang, Jennifer, Matthew A. Bill, Gregory Young, et al.. (2014). Novel Small Molecule XPO1/CRM1 Inhibitors Induce Nuclear Accumulation of TP53, Phosphorylated MAPK and Apoptosis in Human Melanoma Cells. PLoS ONE. 9(7). e102983–e102983. 44 indexed citations

Nicholas, Courtney, Jennifer Yang, Sara Peters, et al.. (2013). PRMT5 Is Upregulated in Malignant and Metastatic Melanoma and Regulates Expression of MITF and p27Kip1. PLoS ONE. 8(9). e74710–e74710. 71 indexed citations

Bill, Matthew A., Courtney Nicholas, Thomas A. Mace, et al.. (2012). Structurally Modified Curcumin Analogs Inhibit STAT3 Phosphorylation and Promote Apoptosis of Human Renal Cell Carcinoma and Melanoma Cell Lines. PLoS ONE. 7(8). e40724–e40724. 70 indexed citations

Lesinski, Gregory B., Jennifer Yang, Matthew A. Bill, et al.. (2012). Effect of small inhibitors of nuclear export (SINE) on growth inhibition and apoptosis of human melanoma cells.. Journal of Clinical Oncology. 30(15_suppl). e13549–e13549. 3 indexed citations

Mundy-Bosse, Bethany L., Gregory Young, Todd M. Bauer, et al.. (2011). Distinct myeloid suppressor cell subsets correlate with plasma IL-6 and IL-10 and reduced interferon-alpha signaling in CD4+ T cells from patients with GI malignancy. Cancer Immunology Immunotherapy. 60(9). 1269–1279. 108 indexed citations

10.

Sullivan, Nicholas J., Kathleen L. Tober, Erin Burns, et al.. (2011). UV Light B–Mediated Inhibition of Skin Catalase Activity Promotes Gr-1+CD11b+ Myeloid Cell Expansion. Journal of Investigative Dermatology. 132(3). 695–702. 40 indexed citations

11.

Nicholas, Courtney, Fengting Yan, Sara Peters, et al.. (2011). Abstract 933: The expression of PRMT5 methyltransferase mediates cell survival and metastatic phenotype in malignant melanoma. Cancer Research. 71(8_Supplement). 933–933. 2 indexed citations

12.

Lesinski, Gregory B., Jason M. Zimmerer, John C. Trefry, et al.. (2010). Modulation of SOCS protein expression influences the interferon responsiveness of human melanoma cells. BMC Cancer. 10(1). 142–142. 50 indexed citations

13.

Bill, Matthew A., James R. Fuchs, Chenglong Li, et al.. (2010). The small molecule curcumin analog FLLL32 induces apoptosis in melanoma cells via STAT3 inhibition and retains the cellular response to cytokines with anti-tumor activity. Molecular Cancer. 9(1). 165–165. 100 indexed citations

14.

Bill, Matthew A., Courtney E. Bakan, Don M. Benson, et al.. (2009). Curcumin induces proapoptotic effects against human melanoma cells and modulates the cellular response to immunotherapeutic cytokines. Molecular Cancer Therapeutics. 8(9). 2726–2735. 80 indexed citations

15.

Currier, John H., Matthew A. Bill, & Michael B. Mayor. (2004). Analysis of wear asymmetry in a series of 94 retrieved polyethylene tibial bearings. Journal of Biomechanics. 38(2). 367–375. 17 indexed citations

16.

Pączek, Leszek, Matthew A. Bill, Janusz Wyzgał, et al.. (1997). [The effect of hypolipidemia treatment on the function of kidney transplanted from cadavers].. PubMed. 97(2). 144–56. 3 indexed citations

17.

Pączek, Leszek, Matthew A. Bill, Janusz Wyzgał, et al.. (1997). Non-immune factors in chronic rejection. Is there a role for hypolipemic drugs?. PubMed. 2(2). 65–9. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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