Massimo Lopes

12.6k total citations · 4 hit papers
73 papers, 8.9k citations indexed

About

Massimo Lopes is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Massimo Lopes has authored 73 papers receiving a total of 8.9k indexed citations (citations by other indexed papers that have themselves been cited), including 70 papers in Molecular Biology, 20 papers in Oncology and 12 papers in Cell Biology. Recurrent topics in Massimo Lopes's work include DNA Repair Mechanisms (64 papers), CRISPR and Genetic Engineering (20 papers) and Genomics and Chromatin Dynamics (15 papers). Massimo Lopes is often cited by papers focused on DNA Repair Mechanisms (64 papers), CRISPR and Genetic Engineering (20 papers) and Genomics and Chromatin Dynamics (15 papers). Massimo Lopes collaborates with scholars based in Switzerland, United States and Italy. Massimo Lopes's co-authors include Marco Foiani, José M. Sogo, Kai J. Neelsen, Matteo Berti, Arnab Ray Chaudhuri, Raquel Herrador, Ralph Zellweger, Giordano Liberi, Cecilia Cotta‐Ramusino and Yoshitami Hashimoto and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Massimo Lopes

70 papers receiving 8.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Fork Reversal and ssDNA Accumulation at Stalled Replication Forks Owing to Checkpoint Defects

2002 668 citations José M. Sogo, Massimo Lopes et al. Science profile →
The DNA replication checkpoint response stabilizes stalled replication forks

2001 612 citations Massimo Lopes, Cecilia Cotta‐Ramusino et al. profile →
Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells

2015 521 citations Ralph Zellweger, Karun Mutreja et al. The Journal of Cell Biology profile →
Replication fork reversal in eukaryotes: from dead end to dynamic response

2015 379 citations Kai J. Neelsen, Massimo Lopes Nature Reviews Molecular Cell Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Massimo Lopes Switzerland	43	8.3k	2.5k	1.5k	1.4k	889	73	8.9k
Alberto Ciccia United States	28	6.6k 0.8×	2.2k 0.9×	1.1k 0.7×	735 0.5×	844 0.9×	45	7.5k
Jean‐Yves Masson Canada	49	7.9k 0.9×	2.8k 1.1×	1.2k 0.8×	520 0.4×	1.3k 1.4×	170	9.0k
Philippe Pasero France	50	6.4k 0.8×	1.1k 0.4×	731 0.5×	1.1k 0.7×	801 0.9×	119	7.0k
Helle D. Ulrich Germany	45	7.5k 0.9×	1.6k 0.6×	1.2k 0.8×	1.8k 1.3×	754 0.8×	106	7.9k
Lorraine S. Symington United States	51	11.1k 1.3×	1.8k 0.7×	1.7k 1.1×	1.2k 0.8×	1.4k 1.5×	113	11.6k
Zuzana Štorchová Germany	35	3.5k 0.4×	876 0.4×	1.0k 0.7×	2.1k 1.5×	812 0.9×	80	5.0k
Grzegorz Ira United States	37	7.1k 0.9×	1.3k 0.5×	1.3k 0.9×	896 0.6×	1.4k 1.6×	49	7.8k
Jenő Gyuris United States	23	4.0k 0.5×	1.7k 0.7×	473 0.3×	836 0.6×	540 0.6×	56	5.5k
JoAnn Sekiguchi United States	39	5.1k 0.6×	1.7k 0.7×	786 0.5×	433 0.3×	1.0k 1.1×	95	6.3k
James R. Bischoff Spain	28	4.7k 0.6×	2.7k 1.1×	454 0.3×	1.8k 1.3×	1.5k 1.6×	49	6.4k

Countries citing papers authored by Massimo Lopes

Since Specialization

Citations

This map shows the geographic impact of Massimo Lopes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Massimo Lopes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Massimo Lopes more than expected).

Fields of papers citing papers by Massimo Lopes

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Massimo Lopes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Massimo Lopes. The network helps show where Massimo Lopes may publish in the future.

Co-authorship network of co-authors of Massimo Lopes

This figure shows the co-authorship network connecting the top 25 collaborators of Massimo Lopes. A scholar is included among the top collaborators of Massimo Lopes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Massimo Lopes. Massimo Lopes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zielińska, Karolina, Christian Koch, Francisco Caiado, et al.. (2025). The pathogenesis of therapy-related myeloid neoplasms from TP53-mutant clonal hematopoiesis. Leukemia. 40(2). 279–292.

Ulferts, Svenja, Massimo Lopes, Kei Miyamoto, & Robert Grosse. (2024). Nuclear actin dynamics and functions at a glance. Journal of Cell Science. 137(6). 10 indexed citations

González‐Acosta, Daniel & Massimo Lopes. (2023). DNA replication and replication stress response in the context of nuclear architecture. Chromosoma. 133(1). 57–75. 3 indexed citations

Rjh, Smith, et al.. (2023). Large-scale expansions of Friedreich's ataxia GAA•TTC repeats in an experimental human system: role of DNA replication and prevention by LNA-DNA oligonucleotides and PNA oligomers. Nucleic Acids Research. 51(16). 8532–8549. 11 indexed citations

Andrš, Martin, Henriette Stoy, Nagaraja Chappidi, et al.. (2023). Excessive reactive oxygen species induce transcription-dependent replication stress. Nature Communications. 14(1). 1791–1791. 44 indexed citations

Stoy, Henriette, Katharina Zwicky, Kevin S. Lang, et al.. (2023). Direct visualization of transcription-replication conflicts reveals post-replicative DNA:RNA hybrids. Nature Structural & Molecular Biology. 30(3). 348–359. 40 indexed citations

Palumbieri, Maria Dilia, Chiara Merigliano, Daniel González‐Acosta, et al.. (2023). Nuclear actin polymerization rapidly mediates replication fork remodeling upon stress by limiting PrimPol activity. Nature Communications. 14(1). 7819–7819. 19 indexed citations

Clerbaux, Laure‐Alix, Pierre Cordier, Kristian Unger, et al.. (2023). Mcl-1 deficiency in murine livers leads to nuclear polyploidisation and mitotic errors: Implications for hepatocellular carcinoma. JHEP Reports. 5(10). 100838–100838. 2 indexed citations

Straub, Tobias, Jana Krietsch, Andrea Sanchi, et al.. (2023). Single molecule MATAC-seq reveals key determinants of DNA replication origin efficiency. Nucleic Acids Research. 51(22). 12303–12324. 1 indexed citations

10.

Dibitetto, Diego, Shannon Marshall, Andrea Sanchi, et al.. (2022). DNA-PKcs promotes fork reversal and chemoresistance. Molecular Cell. 82(20). 3932–3942.e6. 28 indexed citations

11.

Rainey, Michael D., et al.. (2020). CDC7 kinase promotes MRE11 fork processing, modulating fork speed and chromosomal breakage. EMBO Reports. 21(8). e48920–e48920. 23 indexed citations

12.

Berti, Matteo, David Cortez, & Massimo Lopes. (2020). The plasticity of DNA replication forks in response to clinically relevant genotoxic stress. Nature Reviews Molecular Cell Biology. 21(10). 633–651. 233 indexed citations

13.

Chappidi, Nagaraja, Zuzana Naščáková, Ralph Zellweger, et al.. (2019). Fork Cleavage-Religation Cycle and Active Transcription Mediate Replication Restart after Fork Stalling at Co-transcriptional R-Loops. Molecular Cell. 77(3). 528–541.e8. 114 indexed citations

14.

Mutreja, Karun, Lepakshi Ranjha, Ralph Zellweger, et al.. (2016). The MMS22L–TONSL heterodimer directly promotes RAD51‐dependent recombination upon replication stress. The EMBO Journal. 35(23). 2584–2601. 57 indexed citations

15.

Neelsen, Kai J., et al.. (2013). Oncogenes induce genotoxic stress by mitotic processing of unusual replication intermediates. The Journal of Cell Biology. 200(6). 699–708. 151 indexed citations

16.

Neelsen, Kai J., Raquel Herrador, Ralph Zellweger, et al.. (2013). Deregulated origin licensing leads to chromosomal breaks by rereplication of a gapped DNA template. Genes & Development. 27(23). 2537–2542. 68 indexed citations

17.

Toller, Isabella M., Kai J. Neelsen, Martin Steger, et al.. (2011). Carcinogenic bacterial pathogen Helicobacter pylori triggers DNA double-strand breaks and a DNA damage response in its host cells. Proceedings of the National Academy of Sciences. 108(36). 14944–14949. 254 indexed citations

18.

Hashimoto, Yoshitami, Arnab Ray Chaudhuri, Massimo Lopes, & Vincenzo Costanzo. (2010). Rad51 protects nascent DNA from Mre11-dependent degradation and promotes continuous DNA synthesis. Nature Structural & Molecular Biology. 17(11). 1305–1311. 433 indexed citations

19.

Cotta‐Ramusino, Cecilia, Daniele Fachinetti, Chiara Lucca, et al.. (2005). Exo1 Processes Stalled Replication Forks and Counteracts Fork Reversal in Checkpoint-Defective Cells. Molecular Cell. 17(1). 153–159. 224 indexed citations

20.

Sogo, José M., Massimo Lopes, & Marco Foiani. (2002). Fork Reversal and ssDNA Accumulation at Stalled Replication Forks Owing to Checkpoint Defects. Science. 297(5581). 599–602. 668 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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