Mary J. Kennett

4.9k total citations · 2 hit papers
72 papers, 3.9k citations indexed

About

Mary J. Kennett is a scholar working on Molecular Biology, Epidemiology and Microbiology. According to data from OpenAlex, Mary J. Kennett has authored 72 papers receiving a total of 3.9k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 13 papers in Epidemiology and 12 papers in Microbiology. Recurrent topics in Mary J. Kennett's work include Bacterial Infections and Vaccines (11 papers), Peroxisome Proliferator-Activated Receptors (9 papers) and Gut microbiota and health (7 papers). Mary J. Kennett is often cited by papers focused on Bacterial Infections and Vaccines (11 papers), Peroxisome Proliferator-Activated Receptors (9 papers) and Gut microbiota and health (7 papers). Mary J. Kennett collaborates with scholars based in United States, China and India. Mary J. Kennett's co-authors include Na Xiong, Yanming Wang, Ming Li, Pingxin Li, Michael Lindberg, Joshua D. Lambert, Jihyeung Ju, Kenneth R. Reuhl, Shengmin Sang and Chung S. Yang and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Mary J. Kennett

72 papers receiving 3.8k citations

Hit Papers

PAD4 is essential for antibacterial innate immunity media... 2010 2026 2015 2020 2010 2018 250 500 750 1000

Peers

Mary J. Kennett
Mary J. Kennett
Citations per year, relative to Mary J. Kennett Mary J. Kennett (= 1×) peers Harald Carlsen

Countries citing papers authored by Mary J. Kennett

Since Specialization
Citations

This map shows the geographic impact of Mary J. Kennett's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary J. Kennett with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary J. Kennett more than expected).

Fields of papers citing papers by Mary J. Kennett

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary J. Kennett. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary J. Kennett. The network helps show where Mary J. Kennett may publish in the future.

Co-authorship network of co-authors of Mary J. Kennett

This figure shows the co-authorship network connecting the top 25 collaborators of Mary J. Kennett. A scholar is included among the top collaborators of Mary J. Kennett based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mary J. Kennett. Mary J. Kennett is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Allen, Joselyn Natasha, Adwitia Dey, Jingwei Cai, et al.. (2020). Metabolic Profiling Reveals Aggravated Non-Alcoholic Steatohepatitis in High-Fat High-Cholesterol Diet-Fed Apolipoprotein E-Deficient Mice Lacking Ron Receptor Signaling. Metabolites. 10(8). 326–326. 5 indexed citations
2.
Hu, Shaomin, Jie Yang, Yang‐Ding Lin, et al.. (2020). Coordinated co-migration of CCR10+ antibody-producing B cells with helper T cells for colonic homeostatic regulation. Mucosal Immunology. 14(2). 420–430. 13 indexed citations
3.
Snyder, Lindsay M., Juhi Arora, Mary J. Kennett, Veronika Weaver, & Margherita T. Cantorna. (2020). Retinoid Signaling in Intestinal Epithelial Cells Is Essential for Early Survival From Gastrointestinal Infection. Frontiers in Immunology. 11. 559635–559635. 8 indexed citations
4.
Singh, Vishal, Beng San Yeoh, Benoît Chassaing, et al.. (2018). Dysregulated Microbial Fermentation of Soluble Fiber Induces Cholestatic Liver Cancer. Cell. 175(3). 679–694.e22. 397 indexed citations breakdown →
5.
Yeoh, Beng San, Rodrigo Aguilera Olvera, Vishal Singh, et al.. (2016). Epigallocatechin-3-Gallate Inhibition of Myeloperoxidase and Its Counter-Regulation by Dietary Iron and Lipocalin 2 in Murine Model of Gut Inflammation. American Journal Of Pathology. 186(4). 912–926. 41 indexed citations
6.
Wijetunge, Dona Saumya S., et al.. (2016). Growth in Egg Yolk Enhances Salmonella Enteritidis Colonization and Virulence in a Mouse Model of Human Colitis. PLoS ONE. 11(3). e0150258–e0150258. 19 indexed citations
7.
Kudva, Avinash K., et al.. (2015). Chemopreventive Effects of Dietary Eicosapentaenoic Acid Supplementation in Experimental Myeloid Leukemia. Cancer Prevention Research. 8(10). 989–999. 6 indexed citations
8.
Gandhi, Ujjawal H., Naveen Kaushal, Shailaja Hegde, et al.. (2014). Selenium Suppresses Leukemia through the Action of Endogenous Eicosanoids. Cancer Research. 74(14). 3890–3901. 21 indexed citations
9.
Kudva, Avinash K., Naveen Kaushal, Sonia Mohinta, et al.. (2013). Evaluation of the Stability, Bioavailability, and Hypersensitivity of the Omega-3 Derived Anti-Leukemic Prostaglandin: Δ12-Prostaglandin J3. PLoS ONE. 8(12). e80622–e80622. 13 indexed citations
11.
Weyrich, Laura S., Jihye Park, Nicholas A. Spidale, et al.. (2012). A Type VI Secretion System Encoding Locus Is Required for Bordetella bronchiseptica Immunomodulation and Persistence In Vivo. PLoS ONE. 7(10). e45892–e45892. 31 indexed citations
12.
Werner, Jacob, et al.. (2011). Human Electromuscular Incapacitation Devices Characterization: A Comparative Study on Stress and the Physiological Effects on Swine. The Journal of Strength and Conditioning Research. 26(3). 804–810. 5 indexed citations
13.
Kennett, Mary J., et al.. (2011). IACUC Issues Related to Animal Models of Aging. ILAR Journal. 52(1). 106–109. 2 indexed citations
14.
Pérez‐Lorenzo, Rolando, et al.. (2010). Use of a TGFβ type I receptor inhibitor in mouse skin carcinogenesis reveals a dual role for TGFβ signaling in tumor promotion and progression. Carcinogenesis. 31(12). 2127–2135. 27 indexed citations
15.
Wolfe, Daniel, et al.. (2009). IL-10 Induction by Bordetella parapertussis Limits a Protective IFN-γ Response. The Journal of Immunology. 184(3). 1392–1400. 16 indexed citations
16.
Bility, Moses T., Meghann K Devlin-Durante, Nicholas Blazanin, et al.. (2008). Ligand activation of peroxisome proliferator-activated receptor β/δ (PPARβ/δ) inhibits chemically induced skin tumorigenesis. Carcinogenesis. 29(12). 2406–2414. 33 indexed citations
17.
Shan, Weiwei, Christopher J.B. Nicol, Shinji Ito, et al.. (2007). Peroxisome proliferator-activated receptor-β/δ protects against chemically induced liver toxicity in mice. Hepatology. 47(1). 225–235. 79 indexed citations
18.
Pilione, Mylisa R., Luis M. Agosto, Mary J. Kennett, & Eric T. Harvill. (2005). CD11b is required for the resolution of inflammation induced by Bordetella bronchiseptica respiratory infection. Cellular Microbiology. 8(5). 758–768. 18 indexed citations
19.
Kim, Dae Joon, Taro E. Akiyama, F. S. Harman, et al.. (2004). Peroxisome Proliferator-activated Receptor β (δ)-dependent Regulation of Ubiquitin C Expression Contributes to Attenuation of Skin Carcinogenesis. Journal of Biological Chemistry. 279(22). 23719–23727. 82 indexed citations
20.
Kennett, Mary J., et al.. (2004). Toll‐Like Receptor 4 Is Critical to Innate Host Defense in a Murine Model of Bordetellosis. The Journal of Infectious Diseases. 189(5). 833–836. 47 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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