Mary E. Fling

1.7k total citations · 1 hit paper
16 papers, 1.4k citations indexed

About

Mary E. Fling is a scholar working on Infectious Diseases, Molecular Biology and Epidemiology. According to data from OpenAlex, Mary E. Fling has authored 16 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Infectious Diseases, 6 papers in Molecular Biology and 5 papers in Epidemiology. Recurrent topics in Mary E. Fling's work include HIV/AIDS drug development and treatment (5 papers), Antibiotic Resistance in Bacteria (4 papers) and Pneumocystis jirovecii pneumonia detection and treatment (4 papers). Mary E. Fling is often cited by papers focused on HIV/AIDS drug development and treatment (5 papers), Antibiotic Resistance in Bacteria (4 papers) and Pneumocystis jirovecii pneumonia detection and treatment (4 papers). Mary E. Fling collaborates with scholars based in United States, United Kingdom and Denmark. Mary E. Fling's co-authors include C A Richards, Lynn P. Elwell, Linda M. Thurmond, Katherine L. Baker-Neblett, Allen D. Roses, Denise Shortino, Michael Mosteller, Eric Lai, David J. Dow and Seth Hetherington and has published in prestigious journals such as The Lancet, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Mary E. Fling

16 papers receiving 1.3k citations

Hit Papers

Genetic variations in HLA-B region and hypersensitivity r... 2002 2026 2010 2018 2002 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mary E. Fling United States 16 377 368 330 293 247 16 1.4k
F J Silverblatt United States 21 240 0.6× 256 0.7× 505 1.5× 344 1.2× 142 0.6× 36 1.5k
Pierrette Gourde Canada 19 198 0.5× 217 0.6× 215 0.7× 246 0.8× 105 0.4× 40 1.0k
M. T. Labro France 21 205 0.5× 322 0.9× 200 0.6× 505 1.7× 215 0.9× 59 1.3k
Hirotoshi Suzuki Japan 20 140 0.4× 197 0.5× 1.2k 3.5× 125 0.4× 421 1.7× 53 2.2k
N. L. Shipkowitz United States 19 239 0.6× 239 0.6× 244 0.7× 484 1.7× 173 0.7× 29 1.0k
Edwin M. Oden United States 17 96 0.3× 492 1.3× 382 1.2× 138 0.5× 215 0.9× 32 1.1k
K. Seeger Germany 22 72 0.2× 274 0.7× 507 1.5× 133 0.5× 208 0.8× 75 1.7k
W. Opferkuch Germany 26 200 0.5× 250 0.7× 396 1.2× 263 0.9× 442 1.8× 119 2.1k
Kunihiko Fukuchi Japan 21 183 0.5× 63 0.2× 567 1.7× 362 1.2× 72 0.3× 88 1.4k
Robert Ferone United States 26 468 1.2× 60 0.2× 1.1k 3.3× 396 1.4× 84 0.3× 62 2.0k

Countries citing papers authored by Mary E. Fling

Since Specialization
Citations

This map shows the geographic impact of Mary E. Fling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary E. Fling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary E. Fling more than expected).

Fields of papers citing papers by Mary E. Fling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary E. Fling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary E. Fling. The network helps show where Mary E. Fling may publish in the future.

Co-authorship network of co-authors of Mary E. Fling

This figure shows the co-authorship network connecting the top 25 collaborators of Mary E. Fling. A scholar is included among the top collaborators of Mary E. Fling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mary E. Fling. Mary E. Fling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Pillai, Sreekumar, Diane J. Cousens, Ashley Barnes, et al.. (2004). A coding polymorphism in the CYSLT2 receptor with reduced affinity to LTD4 is associated with asthma. Pharmacogenetics. 14(9). 627–633. 48 indexed citations
2.
Fling, Mary E., Zhengyu Xue, Susan Armour, et al.. (2003). In vitro characterization of a human calcitonin receptor gene polymorphism. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 522(1-2). 93–105. 21 indexed citations
3.
Hetherington, Seth, Arlene R. Hughes, Michael Mosteller, et al.. (2002). Genetic variations in HLA-B region and hypersensitivity reactions to abacavir. The Lancet. 359(9312). 1121–1122. 583 indexed citations breakdown →
4.
Stimmel, Julie B., B M Merrill, Lee F. Kuyper, et al.. (2000). Site-specific Conjugation on Serine → Cysteine Variant Monoclonal Antibodies. Journal of Biological Chemistry. 275(39). 30445–30450. 55 indexed citations
5.
Whitlow, Marc, Andrew Howard, David B. Stewart, et al.. (1997). X-ray Crystallographic Studies of Candida albicans Dihydrofolate Reductase. Journal of Biological Chemistry. 272(48). 30289–30298. 60 indexed citations
6.
Fling, Mary E., et al.. (1991). Analysis of a Candida albicans gene that encodes a novel mechanism for resistance to benomyl and methotrexate. Molecular and General Genetics MGG. 227(2). 318–329. 171 indexed citations
7.
Heikkilä, Elli, A. Siitonen, M Jahkola, et al.. (1990). Increase of Trimethoprim Resistance among Shigella Species, 1975-1988: Analysis of Resistance Mechanisms. The Journal of Infectious Diseases. 161(6). 1242–1248. 43 indexed citations
8.
Baccanari, David P., Robert L. Tansik, S S Joyner, et al.. (1989). Characterization of Candida albicans Dihydrofolate Reductase. Journal of Biological Chemistry. 264(2). 1100–1107. 37 indexed citations
9.
Fling, Mary E., et al.. (1988). Nucleotide sequence of the dihydrofolate reductase gene of Saccharomyces cerevisiae. Gene. 63(2). 165–174. 24 indexed citations
10.
Fling, Mary E., et al.. (1988). Characterization of plasmid pAZ1 and the type III dihydrofolate reductase gene. Plasmid. 19(1). 30–38. 29 indexed citations
11.
Mayer, Kenneth H., Mary E. Fling, John Hopkins, & TJ O'Brien. (1985). Trimethoprim Resistance In Multiple Genera of Enterobacteriaceae at a U.S. Hospital: Spread of the Type II Dihydrofolate Reductase Gene by a Single Plasmid. The Journal of Infectious Diseases. 151(5). 783–789. 24 indexed citations
12.
Joyner, S S, Mary E. Fling, David A. Stone, & David P. Baccanari. (1984). Characterization of an R-plasmid dihydrofolate reductase with a monomeric structure.. Journal of Biological Chemistry. 259(9). 5851–5856. 22 indexed citations
13.
Fling, Mary E. & C A Richards. (1983). The nucleotide sequence of the trimethoprim-resistant dihydrofolate reductase gene harbored by Tn7. Nucleic Acids Research. 11(15). 5147–5158. 100 indexed citations
14.
Burchall, James J., Lynn P. Elwell, & Mary E. Fling. (1982). Molecular Mechanisms of Resistance to Trimethoprim. Clinical Infectious Diseases. 4(2). 246–254. 36 indexed citations
15.
Fling, Mary E., et al.. (1982). Monitoring of plasmid-encoded, trimethoprim-resistant dihydrofolate reductase genes: detection of a new resistant enzyme. Antimicrobial Agents and Chemotherapy. 22(5). 882–888. 57 indexed citations
16.
Fling, Mary E. & Lynn P. Elwell. (1980). Protein expression in Escherichia coli minicells containing recombinant plasmids specifying trimethoprim-resistant dihydrofolate reductases. Journal of Bacteriology. 141(2). 779–785. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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