Mary C. Halloran

2.2k citations
43 papers · 1.7k indexed · h-index 26

Mary C. Halloran

42 papers receiving 1.7k citations

Peers

Mary C. Halloran
Comparison fields: 5 of 103
  • Developmental Neuroscience 313
  • Cell Biology 790
  • Cellular and Molecular Neuroscience 713
  • Molecular Biology 1.0k
  • Aging 24
Replace Kit Wong with:
Kit Wong United States
Mineko Kengaku Japan
Daijiro Konno Japan
Thomas S. Vihtelic United States
Thomas Pratt United Kingdom
Robert Hindges United Kingdom
Young‐Ki Bae South Korea
Asha Dwivedy United Kingdom
Catherine Krull United States
Torsten Trowe United States
Mary C. Halloran relative to Kit Wong United States Kit Wong's profile →
Citations per field
00.5×2.7×
Kit Wong · 1×
Citations per year

Countries citing papers authored by Mary C. Halloran

Since Specialization
Citations

This map shows the geographic impact of Mary C. Halloran's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary C. Halloran with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary C. Halloran more than expected).

Fields of papers citing papers by Mary C. Halloran

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary C. Halloran. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary C. Halloran. The network helps show where Mary C. Halloran may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Mary C. Halloran, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mary C. Halloran Line = papers co-authored together Mary C. Halloran links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20260
2 20223
3 20213
4 201927
5 201810
6 201641
7 201440
8 201244
9 201130
10 200916
11 200874
12 200740
13 200632
14 200646
15 200570
16 200543
17 200453
18 200177
19 199824
20 199611

About Mary C. Halloran

Mary C. Halloran is a scholar working on Developmental Neuroscience, Cell Biology and Cellular and Molecular Neuroscience, having authored 43 papers that have together received 1.7k indexed citations. Recurring topics across this work include Axon Guidance and Neuronal Signaling (25 papers), Zebrafish Biomedical Research Applications (16 papers), Neurogenesis and neuroplasticity mechanisms (10 papers), Angiogenesis and VEGF in Cancer (9 papers), Developmental Biology and Gene Regulation (9 papers), Nerve injury and regeneration (5 papers), Hippo pathway signaling and YAP/TAZ (5 papers) and Cellular Mechanics and Interactions (4 papers). The work is most often cited by research in Developmental Neuroscience (313 citations), Cell Biology (790 citations) and Cellular and Molecular Neuroscience (713 citations). Mary C. Halloran has collaborated with scholars based in United States, Japan and United Kingdom. Frequent co-authors include Matthew R. Clay, John Y. Kuwada, Wataru Shoji, Marc A. Wolman, Jason D. Berndt, Fengyun Su, James T. Warren, Mika Sato‐Maeda, Zsolt Lele and Patrick H. Krone. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and Development.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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