Mary Atkinson

519 total citations
8 papers, 363 citations indexed

About

Mary Atkinson is a scholar working on Genetics, Rheumatology and Epidemiology. According to data from OpenAlex, Mary Atkinson has authored 8 papers receiving a total of 363 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Genetics, 5 papers in Rheumatology and 2 papers in Epidemiology. Recurrent topics in Mary Atkinson's work include Connective tissue disorders research (8 papers), Bone and Dental Protein Studies (4 papers) and Cell Adhesion Molecules Research (2 papers). Mary Atkinson is often cited by papers focused on Connective tissue disorders research (8 papers), Bone and Dental Protein Studies (4 papers) and Cell Adhesion Molecules Research (2 papers). Mary Atkinson collaborates with scholars based in United States, United Kingdom and Canada. Mary Atkinson's co-authors include Peter H. Byers, Ulrike Schwarze, Marcia Willing, Daniel S. Greenspan, Guy G. Hoffman, Charles J. Pruchno, Barbra J. Starman, Melanie Pepin, Gillian A. Wallis and Mark D. Ludman and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical Journal and The American Journal of Human Genetics.

In The Last Decade

Mary Atkinson

8 papers receiving 356 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mary Atkinson United States 7 284 139 96 58 47 8 363
A Nicholls United Kingdom 7 214 0.8× 108 0.8× 85 0.9× 43 0.7× 42 0.9× 8 319
Katarina Lindahl Sweden 8 327 1.2× 226 1.6× 108 1.1× 23 0.4× 56 1.2× 10 415
Hideo Shikata Japan 9 130 0.5× 68 0.5× 150 1.6× 63 1.1× 58 1.2× 21 398
D. Toman United States 3 134 0.5× 168 1.2× 208 2.2× 134 2.3× 36 0.8× 8 399
Erin M. Carter United States 9 240 0.8× 88 0.6× 88 0.9× 22 0.4× 35 0.7× 20 310
J Lewthwaite United Kingdom 10 68 0.2× 174 1.3× 96 1.0× 45 0.8× 36 0.8× 11 380
Rebecca C. Pollitt United Kingdom 11 216 0.8× 110 0.8× 96 1.0× 15 0.3× 45 1.0× 20 309
B Lee United States 5 279 1.0× 59 0.4× 124 1.3× 53 0.9× 36 0.8× 5 386
Harikiran Nistala United States 8 218 0.8× 56 0.4× 184 1.9× 23 0.4× 58 1.2× 11 375
Ian D. Crabb United States 9 59 0.2× 289 2.1× 232 2.4× 50 0.9× 81 1.7× 9 531

Countries citing papers authored by Mary Atkinson

Since Specialization
Citations

This map shows the geographic impact of Mary Atkinson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary Atkinson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary Atkinson more than expected).

Fields of papers citing papers by Mary Atkinson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary Atkinson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary Atkinson. The network helps show where Mary Atkinson may publish in the future.

Co-authorship network of co-authors of Mary Atkinson

This figure shows the co-authorship network connecting the top 25 collaborators of Mary Atkinson. A scholar is included among the top collaborators of Mary Atkinson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mary Atkinson. Mary Atkinson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Pace, James M., Mary Atkinson, Marcia Willing, Gillian A. Wallis, & Peter H. Byers. (2001). Deletions and duplications of Gly-Xaa-Yaa triplet repeats in the triple helical domains of type I collagen chains disrupt helix formation and result in several types of osteogenesis imperfecta. Human Mutation. 18(4). 319–326. 34 indexed citations
2.
Schwarze, Ulrike, Mary Atkinson, Guy G. Hoffman, Daniel S. Greenspan, & Peter H. Byers. (2000). Null Alleles of the COL5A1 Gene of Type V Collagen Are a Cause of the Classical Forms of Ehlers-Danlos Syndrome (Types I and II). The American Journal of Human Genetics. 66(6). 1757–1765. 99 indexed citations
3.
Byers, Peter H., Madeleine Duvic, Mary Atkinson, et al.. (1997). Ehlers-Danlos syndrome type VIIA and VIIB result from splice-junction mutations or genomic deletions that involve exon 6 in theCOL1A1 andCOL1A2 genes of type I collagen. American Journal of Medical Genetics. 72(1). 94–105. 82 indexed citations
4.
Pepin, Melanie, Mary Atkinson, Barbra J. Starman, & Peter H. Byers. (1997). STRATEGIES AND OUTCOMES OF PRENATAL DIAGNOSIS FOR OSTEOGENESIS IMPERFECTA: A REVIEW OF BIOCHEMICAL AND MOLECULAR STUDIES COMPLETED IN 129 PREGNANCIES. Prenatal Diagnosis. 17(6). 559–570. 3 indexed citations
5.
Pepin, Melanie, Mary Atkinson, Barbra J. Starman, & Peter H. Byers. (1997). STRATEGIES AND OUTCOMES OF PRENATAL DIAGNOSIS FOR OSTEOGENESIS IMPERFECTA: A REVIEW OF BIOCHEMICAL AND MOLECULAR STUDIES COMPLETED IN 129 PREGNANCIES. Prenatal Diagnosis. 17(6). 559–570. 50 indexed citations
6.
7.
8.
Willing, Marcia, Charles J. Pruchno, Mary Atkinson, & Peter H. Byers. (1992). Osteogenesis imperfecta type I is commonly due to a COL1A1 null allele of type I collagen.. PubMed. 51(3). 508–15. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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