Marwa Kamel

461 total citations
21 papers, 384 citations indexed

About

Marwa Kamel is a scholar working on Genetics, Oncology and Molecular Biology. According to data from OpenAlex, Marwa Kamel has authored 21 papers receiving a total of 384 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Genetics, 6 papers in Oncology and 5 papers in Molecular Biology. Recurrent topics in Marwa Kamel's work include Estrogen and related hormone effects (7 papers), Uterine Myomas and Treatments (4 papers) and Synthesis and biological activity (3 papers). Marwa Kamel is often cited by papers focused on Estrogen and related hormone effects (7 papers), Uterine Myomas and Treatments (4 papers) and Synthesis and biological activity (3 papers). Marwa Kamel collaborates with scholars based in Egypt, United States and Saudi Arabia. Marwa Kamel's co-authors include Salama A. Salama, Concepcion Diaz‐Arrastia, Gökhan S. Kılıç, Shaleen K. Botting, Samia A. Shouman, Timothy D. Veenstra, Ayman Al‐Hendy, Sana M. Salih, Muhammad Saeed and Rajiv Kumar and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and The Journal of Clinical Endocrinology & Metabolism.

In The Last Decade

Marwa Kamel

19 papers receiving 378 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Marwa Kamel Egypt 12 137 92 91 81 65 21 384
Neli Hevir Slovenia 11 158 1.2× 135 1.5× 200 2.2× 169 2.1× 40 0.6× 12 520
Arunima Shilpi India 13 428 3.1× 172 1.9× 97 1.1× 55 0.7× 97 1.5× 17 726
Susanne Schüler‐Toprak Germany 13 136 1.0× 34 0.4× 95 1.0× 140 1.7× 96 1.5× 23 378
Yukitaka Ideyama Japan 9 87 0.6× 26 0.3× 37 0.4× 90 1.1× 46 0.7× 14 357
Yoshiya Miyahara Japan 11 157 1.1× 92 1.0× 67 0.7× 14 0.2× 26 0.4× 21 468
Felicitas Mungenast Austria 9 147 1.1× 18 0.2× 55 0.6× 66 0.8× 98 1.5× 11 407
Floriana Centritto Italy 10 268 2.0× 66 0.7× 41 0.5× 91 1.1× 76 1.2× 11 624
Yukihiro Hirata Japan 8 181 1.3× 22 0.2× 65 0.7× 15 0.2× 86 1.3× 12 336
Xueyan Wu China 10 228 1.7× 19 0.2× 44 0.5× 65 0.8× 43 0.7× 37 417
Ella Twaddle Australia 12 117 0.9× 44 0.5× 76 0.8× 313 3.9× 63 1.0× 16 455

Countries citing papers authored by Marwa Kamel

Since Specialization
Citations

This map shows the geographic impact of Marwa Kamel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marwa Kamel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marwa Kamel more than expected).

Fields of papers citing papers by Marwa Kamel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marwa Kamel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marwa Kamel. The network helps show where Marwa Kamel may publish in the future.

Co-authorship network of co-authors of Marwa Kamel

This figure shows the co-authorship network connecting the top 25 collaborators of Marwa Kamel. A scholar is included among the top collaborators of Marwa Kamel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marwa Kamel. Marwa Kamel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kamel, Marwa, et al.. (2025). Targeting lipid metabolism to overcome tamoxifen resistance in breast cancer: Evaluating the synergistic therapeutic potential of quercetin. Cancer Treatment and Research Communications. 44. 100953–100953.
2.
El‐Abhar, Hanan S., et al.. (2024). A study of the role of androgen receptor and androgen receptor variant 7 in TNBC patients and the effect of their targeting by Enzalutamide and EPI-001 in MDA-MB-231. The Journal of Steroid Biochemistry and Molecular Biology. 245. 106636–106636.
3.
Sobhy, Mohamed H., et al.. (2024). 2-Methoxyestradiol ameliorates doxorubicin-induced cardiotoxicity by regulating the expression of GLUT4 and CPT-1B in female rats. Naunyn-Schmiedeberg s Archives of Pharmacology. 397(9). 7129–7139. 2 indexed citations
5.
El‐Abhar, Hanan S., Dalaal M. Abdallah, Ghada Mohamed, et al.. (2023). The Enzalutamide and EPI-001 modulate cell proliferation and metastasis markers in T47D by targeting AR/ARV7. INDONESIAN JOURNAL OF PHARMACY. 2 indexed citations
6.
Allam, Heba Abdelrasheed, et al.. (2022). Design, synthesis, cytotoxic and enzyme inhibitory activities of 1,3,4-oxadiazole and 1,3,4-thiadiazine hybrids against non-small cell lung cancer. Results in Chemistry. 4. 100373–100373. 3 indexed citations
7.
Bayoumi, Asmaa M. A., Mohamed S. Abdel‐Bakky, Marwa Kamel, et al.. (2020). Ameliorative effect of 2-methoxyestradiol on radiation-induced lung injury. Life Sciences. 255. 117743–117743. 14 indexed citations
8.
Kamel, Marwa, et al.. (2020). Evaluation of androgen receptor status in urothelial carcinoma of the urinary bladder in Egyptian patients: an immunohistochemical study. SHILAP Revista de lepidopterología. 26(1). 2 indexed citations
11.
Shouman, Samia A., et al.. (2016). Leptin influences estrogen metabolism and increases DNA adduct formation in breast cancer cells. Cancer Biology and Medicine. 13(4). 505–505. 17 indexed citations
12.
Salama, Salama A., Mahmoud A. Mohammad, Concepcion Diaz‐Arrastia, et al.. (2014). Estradiol-17β Upregulates Pyruvate Kinase M2 Expression to Coactivate Estrogen Receptor-α and to Integrate Metabolic Reprogramming With the Mitogenic Response in Endometrial Cells. The Journal of Clinical Endocrinology & Metabolism. 99(10). 3790–3799. 35 indexed citations
13.
Salama, Salama A., Concepcion Diaz‐Arrastia, Gökhan S. Kılıç, & Marwa Kamel. (2012). 2-Methoxyestradiol causes functional repression of transforming growth factor β3 signaling by ameliorating Smad and non-Smad signaling pathways in immortalized uterine fibroid cells. Fertility and Sterility. 98(1). 178–184.e1. 28 indexed citations
14.
Kamel, Marwa, Samia A. Shouman, Gökhan S. Kılıç, et al.. (2012). Effect of Tumour Necrosis Factor-Alpha on Estrogen Metabolic Pathways in Breast Cancer Cells. Journal of Cancer. 3. 310–321. 39 indexed citations
15.
Salama, Salama A., Marwa Kamel, Shaleen K. Botting, et al.. (2009). Catechol-O-Methyltransferase Expression and 2-Methoxyestradiol Affect Microtubule Dynamics and Modify Steroid Receptor Signaling in Leiomyoma Cells. PLoS ONE. 4(10). e7356–e7356. 39 indexed citations
16.
Salama, Salama A., Marwa Kamel, Concepcion Diaz‐Arrastia, et al.. (2009). Effect of TNF-α on Estrogen Metabolism and Endometrial Cells: Potential Physiological and Pathological Relevance. Molecular Endocrinology. 23(1). 124–124. 3 indexed citations
17.
Salama, Salama A., et al.. (2008). Catecholestrogens induce oxidative stress and malignant transformation in human endometrial glandular cells: Protective effect of catechol‐O‐methyltransferase. International Journal of Cancer. 123(6). 1246–1254. 47 indexed citations
18.
Salama, Salama A., Marwa Kamel, Concepcion Diaz‐Arrastia, et al.. (2008). Effect of Tumor Necrosis Factor-α on Estrogen Metabolism and Endometrial Cells: Potential Physiological and Pathological Relevance. The Journal of Clinical Endocrinology & Metabolism. 94(1). 285–293. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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