Martin A. Wikström

929 total citations
18 papers, 701 citations indexed

About

Martin A. Wikström is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Martin A. Wikström has authored 18 papers receiving a total of 701 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Cellular and Molecular Neuroscience, 10 papers in Molecular Biology and 5 papers in Cognitive Neuroscience. Recurrent topics in Martin A. Wikström's work include Neuroscience and Neuropharmacology Research (13 papers), Ion channel regulation and function (6 papers) and Neural dynamics and brain function (3 papers). Martin A. Wikström is often cited by papers focused on Neuroscience and Neuropharmacology Research (13 papers), Ion channel regulation and function (6 papers) and Neural dynamics and brain function (3 papers). Martin A. Wikström collaborates with scholars based in Sweden, United States and United Kingdom. Martin A. Wikström's co-authors include Sten Grillner, Russell H. Hill, Krister Kristensson, Graham L. Collingridge, Jeanette Hellgren Kotaleski, Kim T. Blackwell, Mikael Nygård, Paul M. Matthews, Maria Lindskog and Erik Svensson and has published in prestigious journals such as Nature, The Journal of Physiology and Journal of Neurophysiology.

In The Last Decade

Martin A. Wikström

18 papers receiving 688 citations

Peers

Martin A. Wikström
Ray Perrins United Kingdom
P.A.M. van Dongen Netherlands
Karine Fénelon United States
Jorge Mancillas United States
Arnaud L. Lalive Switzerland
Eric Proshansky United States
D. P. McCobb United States
Ray Perrins United Kingdom
Martin A. Wikström
Citations per year, relative to Martin A. Wikström Martin A. Wikström (= 1×) peers Ray Perrins

Countries citing papers authored by Martin A. Wikström

Since Specialization
Citations

This map shows the geographic impact of Martin A. Wikström's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin A. Wikström with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin A. Wikström more than expected).

Fields of papers citing papers by Martin A. Wikström

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin A. Wikström. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin A. Wikström. The network helps show where Martin A. Wikström may publish in the future.

Co-authorship network of co-authors of Martin A. Wikström

This figure shows the co-authorship network connecting the top 25 collaborators of Martin A. Wikström. A scholar is included among the top collaborators of Martin A. Wikström based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin A. Wikström. Martin A. Wikström is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Ericsson, Jesper, Gilad Silberberg, Brita Robertson, Martin A. Wikström, & Sten Grillner. (2011). Striatal cellular properties conserved from lampreys to mammals. The Journal of Physiology. 589(12). 2979–2992. 36 indexed citations
2.
Johansson, Jenny U., Jesper Ericsson, Juliette Janson, et al.. (2008). An Ancient Duplication of Exon 5 in the Snap25 Gene Is Required for Complex Neuronal Development/Function. PLoS Genetics. 4(11). e1000278–e1000278. 70 indexed citations
3.
Huss, Mikael, et al.. (2007). An experimentally constrained computational model of NMDA oscillations in lamprey CPG neurons. Journal of Computational Neuroscience. 25(1). 108–121. 7 indexed citations
4.
Ericsson, Jesper, Brita Robertson, & Martin A. Wikström. (2007). A lamprey striatal brain slice preparation for patch-clamp recordings. Journal of Neuroscience Methods. 165(2). 251–256. 8 indexed citations
5.
Lindskog, Maria, et al.. (2006). Transient Calcium and Dopamine Increase PKA Activity and DARPP-32 Phosphorylation. PLoS Computational Biology. 2(9). e119–e119. 83 indexed citations
6.
Nygård, Mikael, Russell H. Hill, Martin A. Wikström, & Krister Kristensson. (2005). Age-related changes in electrophysiological properties of the mouse suprachiasmatic nucleus in vitro. Brain Research Bulletin. 65(2). 149–154. 66 indexed citations
7.
Lüthi, Andreas, Martin A. Wikström, Mary J. Palmer, et al.. (2004). Bi-directional modulation of AMPA receptor unitary conductance by synaptic activity. BMC Neuroscience. 5(1). 44–44. 55 indexed citations
8.
Wikström, Martin A.. (2004). Compensating for Respiratory Artifacts in Blood Pressure Waveforms. KTH Publication Database DiVA (KTH Royal Institute of Technology). 1 indexed citations
9.
Svensson, Erik, Martin A. Wikström, Russell H. Hill, & Sten Grillner. (2003). Endogenous and exogenous dopamine presynaptically inhibits glutamatergic reticulospinal transmission via an action of D2‐receptors on N‐type Ca2+ channels. European Journal of Neuroscience. 17(3). 447–454. 28 indexed citations
10.
Wikström, Martin A., Paul M. Matthews, D. F. Roberts, Graham L. Collingridge, & Zuner A. Bortolotto. (2003). Parallel kinase cascades are involved in the induction of LTP at hippocampal CA1 synapses. Neuropharmacology. 45(6). 828–836. 45 indexed citations
11.
Sandberg, Malin, Peter Wallén, Martin A. Wikström, & Krister Kristensson. (2003). Scrapie-infected GT1-1 cells show impaired function of voltage-gated N-type calcium channels (Cav 2.2) which is ameliorated by quinacrine treatment. Neurobiology of Disease. 15(1). 143–151. 24 indexed citations
12.
Svensson, Erik, Joshua D. Woolley, Martin A. Wikström, & Sten Grillner. (2003). Endogenous dopaminergic modulation of the lamprey spinal locomotor network. Brain Research. 970(1-2). 1–8. 38 indexed citations
13.
Benke, Tim A., Andreas Lüthi, Mary J. Palmer, et al.. (2001). Mathematical modelling of non‐stationary fluctuation analysis for studying channel properties of synaptic AMPA receptors. The Journal of Physiology. 537(2). 407–420. 33 indexed citations
14.
Büschges, Ansgar, Martin A. Wikström, Sten Grillner, & Abdeljabbar El Manira. (2000). Roles of High-Voltage–Activated Calcium Channel Subtypes in a Vertebrate Spinal Locomotor Network. Journal of Neurophysiology. 84(6). 2758–2766. 31 indexed citations
15.
Wikström, Martin A., S. Grillner, & Abdeljabbar El Manira. (1999). Inhibition of N- and L-type Ca2+ currents by dopamine in lamprey spinal motoneurons. Neuroreport. 10(15). 3179–3183. 21 indexed citations
16.
Wikström, Martin A., et al.. (1998). Cholinergic modulation of extracellular ATP-induced cytoplasmic calcium concentrations in cochlear outer hair cells. Journal of Physiology-Paris. 92(5-6). 345–349. 12 indexed citations
17.
Wikström, Martin A., Russell H. Hill, Jeanette Hellgren, & Sten Grillner. (1995). The action of 5-HT on calcium-dependent potassium channels and on the spinal locomotor network in lamprey is mediated by 5-HT1A-like receptors. Brain Research. 678(1-2). 191–199. 57 indexed citations
18.
Shupliakov, Oleg, Martin A. Wikström, Lennart Brodin, et al.. (1995). Control of lamprey locomotor neurons by colocalized monoamine transmitters. Nature. 374(6519). 266–268. 86 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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