Martha R. Stampfer
- Molecular Biology top 0.5%
- Oncology top 0.5%
- Cancer Research top 0.5%
- Genetics top 1%
- Physiology top 1%
- Co-authors
- Paul YaswenJames C. GarbeJ. C. BartleyJoyce Taylor‐PapadimitriouR G HamA. J. HackettKeith R. YamamotoR. C. Hallowes
- Topics
- Cancer Cells and Metastasis (37 papers)Telomeres, Telomerase, and Senescence (27 papers)Cancer-related Molecular Pathways (23 papers)
- Cited by
- OncologyCancer ResearchAging
- Partner nations
- United StatesUnited KingdomNorway
In The Last Decade
Martha R. Stampfer
130 papers receiving 9.1k citations
Peers
Comparison fields: 5 of 142
- Molecular Biology 6.0k
- Oncology 3.7k
- Cancer Research 1.8k
- Genetics 1.5k
- Physiology 1.3k
Countries citing papers authored by Martha R. Stampfer
This map shows the geographic impact of Martha R. Stampfer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martha R. Stampfer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martha R. Stampfer more than expected).
Fields of papers citing papers by Martha R. Stampfer
This network shows the impact of papers produced by Martha R. Stampfer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martha R. Stampfer. The network helps show where Martha R. Stampfer may publish in the future.
Co-authorship network of co-authors of Martha R. Stampfer
This figure shows the co-authorship network connecting the top 25 collaborators of Martha R. Stampfer. A scholar is included among the top collaborators of Martha R. Stampfer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martha R. Stampfer. Martha R. Stampfer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 22 | |
| 2 | 14 | |
| 3 | 59 | |
| 4 | 32 | |
| 5 | 24 | |
| 6 | 7 | |
| 7 | 80 | |
| 8 | 25 | |
| 9 | 43 | |
| 10 | 127 | |
| 11 | 94 | |
| 12 | 60 | |
| 13 | 14 | |
| 14 | Errata: p53 Mutations in human immortalized epithelial cell lines (Carcinogenesis (1993) 14 (833-839)) | 40 |
| 15 | 74 | |
| 16 | 375 | |
| 17 | 119 | |
| 18 | 31 | |
| 19 | 66 | |
| 20 | 66 |
About Martha R. Stampfer
Martha R. Stampfer is a scholar working on Aging, Oncology and Cancer Research, having authored 132 papers that have together received 9.4k indexed citations. Recurring topics across this work include Cancer Cells and Metastasis (37 papers), Telomeres, Telomerase, and Senescence (27 papers) and Cancer-related Molecular Pathways (23 papers). The work is most often cited by research in Oncology (3.7k citations), Cancer Research (1.8k citations) and Aging (200 citations). Martha R. Stampfer has collaborated with scholars based in United States, United Kingdom and Norway. Frequent co-authors include Paul Yaswen, James C. Garbe, J. C. Bartley, Joyce Taylor‐Papadimitriou, R G Ham, A. J. Hackett, Keith R. Yamamoto, R. C. Hallowes, Mark A. LaBarge and Bernard W. Futscher. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.