Mart Saarma

650 total citations
8 papers, 522 citations indexed

About

Mart Saarma is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Mart Saarma has authored 8 papers receiving a total of 522 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 3 papers in Cellular and Molecular Neuroscience and 3 papers in Cell Biology. Recurrent topics in Mart Saarma's work include Nerve injury and regeneration (3 papers), Signaling Pathways in Disease (2 papers) and Cellular transport and secretion (2 papers). Mart Saarma is often cited by papers focused on Nerve injury and regeneration (3 papers), Signaling Pathways in Disease (2 papers) and Cellular transport and secretion (2 papers). Mart Saarma collaborates with scholars based in Finland, United Kingdom and France. Mart Saarma's co-authors include Maria Lindahl, Dirk G. de Rooij, Xiaoxian Meng, Hannu Sariola, Urmas Arumäe, Dmitry Poteryaev, Judith Thomas-Crusells, Marc Billaud, Alexey TITIEVSKY and Tõnis Timmusk and has published in prestigious journals such as FEBS Letters, European Journal of Biochemistry and Cell and Tissue Research.

In The Last Decade

Mart Saarma

7 papers receiving 498 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mart Saarma Finland	7	250	232	137	75	73	8	522
Alexey TITIEVSKY Finland	7	298 1.2×	351 1.5×	179 1.3×	54 0.7×	46 0.6×	8	629
J.F.R. Cavanagh United Kingdom	7	245 1.0×	235 1.0×	252 1.8×	28 0.4×	39 0.5×	9	530
G. Mercier France	10	189 0.8×	237 1.0×	88 0.6×	70 0.9×	48 0.7×	15	570
H. Hyatt Sachs United States	9	247 1.0×	191 0.8×	192 1.4×	23 0.3×	55 0.8×	10	467
Kathryn L. Farr United States	6	333 1.3×	220 0.9×	146 1.1×	41 0.5×	17 0.2×	6	783
Carlos G. Martínez-Moreno Mexico	14	72 0.3×	170 0.7×	72 0.5×	60 0.8×	70 1.0×	39	519
Simone Musco United States	9	190 0.8×	386 1.7×	23 0.2×	43 0.6×	34 0.5×	15	677
Fani Memi United Kingdom	14	149 0.6×	264 1.1×	143 1.0×	76 1.0×	21 0.3×	17	480
Ylva Skoglösa Sweden	9	304 1.2×	313 1.3×	47 0.3×	35 0.5×	58 0.8×	11	507
Simon Shields United Kingdom	8	169 0.7×	156 0.7×	372 2.7×	48 0.6×	24 0.3×	9	599

Countries citing papers authored by Mart Saarma

Since Specialization

Citations

This map shows the geographic impact of Mart Saarma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mart Saarma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mart Saarma more than expected).

Fields of papers citing papers by Mart Saarma

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mart Saarma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mart Saarma. The network helps show where Mart Saarma may publish in the future.

Co-authorship network of co-authors of Mart Saarma

This figure shows the co-authorship network connecting the top 25 collaborators of Mart Saarma. A scholar is included among the top collaborators of Mart Saarma based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mart Saarma. Mart Saarma is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

8 of 8 papers shown

1.

Mahato, Arun Kumar & Mart Saarma. (2025). Biology of RET receptor and its ligands: focus on the nervous system. Endocrine Related Cancer. 32(6).

2.

Meng, Xiaoxian, et al.. (2001). Promotion of seminomatous tumors by targeted overexpression of glial cell line-derived neurotrophic factor in mouse testis.. PubMed. 61(8). 3267–71. 94 indexed citations

3.

Saarma, Mart. (2000). GDNF – a stranger in the TGF‐β superfamily?. European Journal of Biochemistry. 267(24). 6968–6971. 113 indexed citations

4.

Laurikainen, Antti, Jukka O. Hiltunen, Sampsa Vanhatalo, Erik Klinge, & Mart Saarma. (2000). Glial cell line-derived neurotrophic factor is expressed in penis of adult rat and retrogradely transported in penile parasympathetic and sensory nerves. Cell and Tissue Research. 302(3). 321–329. 37 indexed citations

5.

Lindahl, Maria, Tõnis Timmusk, Jari Rossi, Mart Saarma, & Matti S. Airaksinen. (2000). Expression and Alternative Splicing of Mouse Gfra4 Suggest Roles in Endocrine Cell Development. Molecular and Cellular Neuroscience. 15(6). 522–533. 79 indexed citations

6.

Heinonen, Outi, et al.. (1999). Toward Understanding the Neuronal Pathogenesis of Aspartylglucosaminuria: Expression of Aspartylglucosaminidase in Brain during Development. Molecular Genetics and Metabolism. 67(4). 294–307. 10 indexed citations

7.

Poteryaev, Dmitry, Alexey TITIEVSKY, Judith Thomas-Crusells, et al.. (1999). GDNF triggers a novel Ret‐independent Src kinase family‐coupled signaling via a GPI‐linked GDNF receptor α1. FEBS Letters. 463(1-2). 63–66. 142 indexed citations

8.

Isosomppi, Juha, Outi Heinonen, Jukka O. Hiltunen, et al.. (1999). Developmental expression of palmitoyl protein thioesterase in normal mice. Developmental Brain Research. 118(1-2). 1–11. 47 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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