Marshall V. Williams

2.8k total citations
88 papers, 2.2k citations indexed

About

Marshall V. Williams is a scholar working on Molecular Biology, Epidemiology and Oncology. According to data from OpenAlex, Marshall V. Williams has authored 88 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 22 papers in Epidemiology and 19 papers in Oncology. Recurrent topics in Marshall V. Williams's work include Cytomegalovirus and herpesvirus research (17 papers), Viral-associated cancers and disorders (16 papers) and Biochemical and Molecular Research (14 papers). Marshall V. Williams is often cited by papers focused on Cytomegalovirus and herpesvirus research (17 papers), Viral-associated cancers and disorders (16 papers) and Biochemical and Molecular Research (14 papers). Marshall V. Williams collaborates with scholars based in United States, Japan and United Kingdom. Marshall V. Williams's co-authors include Ronald Glaser, Maria E. Ariza, Gautam N. Bijur, Jonathan D. Pollack, J. Dennis Pollack, Ronald N. McElhaney, W. James Waldman, Y. C. Cheng, Jeffrey I. Cohen and Christopher J. Jones and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Immunology.

In The Last Decade

Marshall V. Williams

88 papers receiving 2.1k citations

Peers

Marshall V. Williams
Marshall V. Williams
Citations per year, relative to Marshall V. Williams Marshall V. Williams (= 1×) peers Brigitte Decallonne

Countries citing papers authored by Marshall V. Williams

Since Specialization
Citations

This map shows the geographic impact of Marshall V. Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marshall V. Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marshall V. Williams more than expected).

Fields of papers citing papers by Marshall V. Williams

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marshall V. Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marshall V. Williams. The network helps show where Marshall V. Williams may publish in the future.

Co-authorship network of co-authors of Marshall V. Williams

This figure shows the co-authorship network connecting the top 25 collaborators of Marshall V. Williams. A scholar is included among the top collaborators of Marshall V. Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marshall V. Williams. Marshall V. Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Williams, Marshall V., et al.. (2023). EBV dUTPase: A Novel Modulator of Inflammation and the Tumor Microenvironment in EBV-Associated Malignancies. Cancers. 15(3). 855–855. 5 indexed citations
2.
Williams, Marshall V., et al.. (2019). Epstein-Barr Virus dUTPase Induces Neuroinflammatory Mediators: Implications for Myalgic Encephalomyelitis/Chronic Fatigue Syndrome. Clinical Therapeutics. 41(5). 848–863. 37 indexed citations
4.
Aubrecht, Taryn G., Bachir Abi Salloum, Maria E. Ariza, et al.. (2014). Restraint Induces Sickness Responses Independent of Injection with Epstein-Barr Virus (EBV)-Encoded dUTPase. Journal of Behavioral and Brain Science. 4(11). 491–505. 4 indexed citations
5.
Ariza, Maria E., Ronald Glaser, & Marshall V. Williams. (2014). Human herpesviruses-encoded dUTPases: a family of proteins that modulate dendritic cell function and innate immunity. Frontiers in Microbiology. 5. 504–504. 37 indexed citations
6.
Binkley, Philip F., Glen E. Cooke, Min Chen, et al.. (2013). Correction: Evidence for the Role of Epstein Barr Virus Infections in the Pathogenesis of Acute Coronary Events. PLoS ONE. 8(8). 5 indexed citations
7.
Chen, Haoyan, Henri‐Alexandre Michaud, Genki Hayashi, et al.. (2012). Protective Effect of Human Endogenous Retrovirus K dUTPase Variants on Psoriasis Susceptibility. Journal of Investigative Dermatology. 132(7). 1833–1840. 20 indexed citations
8.
Lerner, A. Martin, Maria E. Ariza, Marshall V. Williams, et al.. (2012). Antibody to Epstein-Barr Virus Deoxyuridine Triphosphate Nucleotidohydrolase and Deoxyribonucleotide Polymerase in a Chronic Fatigue Syndrome Subset. PLoS ONE. 7(11). e47891–e47891. 44 indexed citations
9.
Williams, Marshall V., et al.. (2011). A Human Endogenous Retrovirus K dUTPase Triggers a TH1, TH17 Cytokine Response: Does It Have a Role in Psoriasis?. Journal of Investigative Dermatology. 131(12). 2419–2427. 50 indexed citations
10.
Glaser, Ronald, et al.. (2009). The EBV-Encoded dUTPase Activates NF-κB through the TLR2 and MyD88-Dependent Signaling Pathway. The Journal of Immunology. 182(2). 851–859. 132 indexed citations
11.
Glaser, Ronald, Monica L. Litsky, David A. Padgett, et al.. (2005). EBV-encoded dUTPase induces immune dysregulation: Implications for the pathophysiology of EBV-associated disease. Virology. 346(1). 205–218. 88 indexed citations
12.
Glaser, Ronald, David A. Padgett, Monica L. Litsky, et al.. (2004). Stress-associated changes in the steady-state expression of latent Epstein–Barr virus: Implications for chronic fatigue syndrome and cancer. Brain Behavior and Immunity. 19(2). 91–103. 99 indexed citations
13.
Long, John F., et al.. (2003). The effect of iron on the biological activities of erionite and mordenite. Environment International. 29(4). 451–458. 22 indexed citations
14.
Reash, Robin J., et al.. (2001). Assessment of Tolerant Sunfish Populations (Lepomis sp.) Inhabiting Selenium-Laden Coal Ash Effluents. Ecotoxicology and Environmental Safety. 50(3). 217–224. 39 indexed citations
15.
Studebaker, Adam, Ganesaratnam K. Balendiran, & Marshall V. Williams. (2001). The Herpesvirus Encoded dUTPase as a Potential Chemotherapeutic Target. Current Protein and Peptide Science. 2(4). 371–379. 12 indexed citations
16.
Reddy, Sanjay M., Marshall V. Williams, & Jeffrey I. Cohen. (1998). Expression of a Uracil DNA Glycosylase (UNG) Inhibitor in Mammalian Cells: Varicella-Zoster Virus Can Replicatein Vitroin the Absence of Detectable UNG Activity. Virology. 251(2). 393–401. 27 indexed citations
17.
Pollack, J. Dennis, et al.. (1993). The Metabolism of AIDS-Associated Mycoplasmas. Clinical Infectious Diseases. 17(Supplement_1). S267–S271. 6 indexed citations
18.
Tsai, Ching-Hwa, Marshall V. Williams, & Ronald Glaser. (1991). Characterization of two monoclonal antibodies to Epstein-Barr virus diffuse early antigen which react to two different epitopes and have different biological function. Journal of Virological Methods. 33(1-2). 47–52. 32 indexed citations
19.
Waldman, W. James, et al.. (1991). Preservation of natural endothelial cytopathogenicity of cytomegalovirus by propagation in endothelial cells. Archives of Virology. 117(3-4). 143–164. 105 indexed citations
20.
Holliday, Jane & Marshall V. Williams. (1991). Inhibition of herpes simplex virus types 1 and 2 replication in vitro by mercurithio analogs of deoxyuridine. Antiviral Research. 16(2). 197–203. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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