Mark Sawicki

1.5k total citations
42 papers, 1.1k citations indexed

About

Mark Sawicki is a scholar working on Epidemiology, Oncology and Molecular Biology. According to data from OpenAlex, Mark Sawicki has authored 42 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Epidemiology, 19 papers in Oncology and 13 papers in Molecular Biology. Recurrent topics in Mark Sawicki's work include Neuroendocrine Tumor Research Advances (20 papers), Pancreatic and Hepatic Oncology Research (10 papers) and Cancer-related Molecular Pathways (6 papers). Mark Sawicki is often cited by papers focused on Neuroendocrine Tumor Research Advances (20 papers), Pancreatic and Hepatic Oncology Research (10 papers) and Cancer-related Molecular Pathways (6 papers). Mark Sawicki collaborates with scholars based in United States, Japan and Poland. Mark Sawicki's co-authors include Edward Passaro, Ghassan Samara, Michael Hurwitz, Sergio Huerta, Edward H. Livingston, Richard A. Gatti, Albert Y. Wu, James R. Arteaga, Carol Kashefi and Edward H. M. Wang and has published in prestigious journals such as Oncogene, Endocrinology and Gene.

In The Last Decade

Mark Sawicki

41 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark Sawicki United States 17 501 496 330 266 181 42 1.1k
M. Manzoni Italy 19 509 1.0× 389 0.8× 164 0.5× 210 0.8× 151 0.8× 38 945
Kazuki Takakura Japan 21 414 0.8× 220 0.4× 336 1.0× 56 0.2× 255 1.4× 67 1.0k
Robert Rzepko Poland 16 200 0.4× 165 0.3× 205 0.6× 69 0.3× 134 0.7× 44 711
Natalie Prinzi Italy 19 681 1.4× 380 0.8× 182 0.6× 192 0.7× 172 1.0× 71 1.1k
Monica Ter‐Minassian United States 15 361 0.7× 234 0.5× 205 0.6× 130 0.5× 70 0.4× 29 757
Jihao Zhou United States 17 220 0.4× 411 0.8× 391 1.2× 163 0.6× 144 0.8× 61 1.7k
Colin E. Champ United States 22 329 0.7× 251 0.5× 434 1.3× 116 0.4× 201 1.1× 77 1.8k
Paul N. Staats United States 20 199 0.4× 137 0.3× 183 0.6× 45 0.2× 194 1.1× 61 1.0k
Michaël Noë United States 18 408 0.8× 184 0.4× 297 0.9× 40 0.2× 188 1.0× 40 1.0k
Hidehisa Horiguchi Japan 18 138 0.3× 195 0.4× 238 0.7× 84 0.3× 305 1.7× 41 1.0k

Countries citing papers authored by Mark Sawicki

Since Specialization
Citations

This map shows the geographic impact of Mark Sawicki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Sawicki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Sawicki more than expected).

Fields of papers citing papers by Mark Sawicki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Sawicki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Sawicki. The network helps show where Mark Sawicki may publish in the future.

Co-authorship network of co-authors of Mark Sawicki

This figure shows the co-authorship network connecting the top 25 collaborators of Mark Sawicki. A scholar is included among the top collaborators of Mark Sawicki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark Sawicki. Mark Sawicki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sawicki, Mark, Ankur A. Gholkar, & Jorge Z. Torres. (2019). Menin Associates With the Mitotic Spindle and Is Important for Cell Division. Endocrinology. 160(8). 1926–1936. 2 indexed citations
2.
Chen, Gao, et al.. (2008). Menin Promotes the Wnt Signaling Pathway in Pancreatic Endocrine Cells. Molecular Cancer Research. 6(12). 1894–1907. 50 indexed citations
3.
Farley, Steven, Chen Gao, Min Wang, et al.. (2006). Menin Localizes to Chromatin Through an ATR-CHK1 Mediated Pathway After UV-Induced DNA Damage. Journal of Surgical Research. 133(1). 29–37. 14 indexed citations
4.
Sawicki, Mark, et al.. (2002). Frequent deletion of chromosome 3 in malignant sporadic pancreatic endocrine tumors. Molecular and Cellular Endocrinology. 190(1-2). 109–114. 17 indexed citations
5.
Sawicki, Mark, et al.. (2001). Molecular and Genetic Mechanisms of Tumorigenesis in Multiple Endocrine Neoplasia Type-1. Molecular Endocrinology. 15(10). 1653–1664. 77 indexed citations
6.
Wu, Xinyi, et al.. (2001). Anomalous Overexpression of p27Kip1 in Sporadic Pancreatic Endocrine Tumors. Journal of Surgical Research. 96(2). 284–288. 22 indexed citations
7.
Udar, Nitin, Shunbin Xu, J.-O. Bay, et al.. (1999). Physical Map of the Region Surrounding the Ataxia-Telangiectasia Gene on Human Chromosome 11q22-23. Neuropediatrics. 30(4). 176–180. 4 indexed citations
8.
Passaro, Edward, et al.. (1998). The Origin of Sporadic Gastrinomas Within the Gastrinoma Triangle. Archives of Surgery. 133(1). 13–6; discussion 17. 33 indexed citations
9.
Chakrabarti, Rita, Eri S. Srivatsan, Thomas F. Wood, et al.. (1998). Deletion mapping of endocrine tumors localizes a second tumor suppressor gene on chromosome band 11q13. Genes Chromosomes and Cancer. 22(2). 130–137. 65 indexed citations
10.
Sawicki, Mark, et al.. (1997). Pancreatic endocrine tumors with loss of heterozygosity at the multiple endocrine neoplasia type I locus. The American Journal of Surgery. 173(6). 518–520. 7 indexed citations
11.
Wood, Thomas F., Eri S. Srivatsan, Ghassan Samara, et al.. (1996). A 1.5-Megabase Physical Map Encompassing the Multiple Endocrine Neoplasia Type-1 (MEN1) Locus on Chromosome 11q13. Genomics. 38(2). 166–173. 14 indexed citations
12.
Thomas, Howard, Mark Sawicki, Klaus J. Lewin, et al.. (1995). Pancreatic Polypeptide Immunoreactivity in Sporadic Gastrinoma. Pancreas. 11(4). 350–356. 10 indexed citations
13.
Sawicki, Mark, Ghassan Samara, Richard A. Gatti, et al.. (1994). Putative tumor-suppressor gene on chromosome 11 is important in sporadic endocrine tumor formation. The American Journal of Surgery. 167(1). 180–185. 47 indexed citations
14.
Thomas, Howard, et al.. (1993). Biologic behavior of sporadic gastrinoma located to the right and left of the superior mesenteric artery. The American Journal of Surgery. 165(1). 101–106. 13 indexed citations
15.
Samara, Ghassan, Mark Sawicki, Michael Hurwitz, & Edward Passaro. (1993). Molecular biology and therapy of disease. The American Journal of Surgery. 165(6). 720–727. 3 indexed citations
16.
Sawicki, Mark, et al.. (1993). A review of gene transfer techniques. The American Journal of Surgery. 165(3). 350–354. 9 indexed citations
17.
Passaro, Edward, Michael Hurwitz, Ghassan Samara, & Mark Sawicki. (1992). Molecular biology: An overview. The American Journal of Surgery. 164(2). 146–152. 4 indexed citations
18.
Sawicki, Mark, et al.. (1992). Loss of heterozygosity on chromosome 11 in sporadic gastrinomas. Human Genetics. 89(4). 445–449. 43 indexed citations
19.
Hurwitz, Michael E., Mark Sawicki, Ghassan Samara, & Edward Passaro. (1992). Diagnostic and prognostic molecular markersin cancer. The American Journal of Surgery. 164(3). 299–306. 15 indexed citations
20.
Sawicki, Mark. (1990). The Dichotomous Distribution of Gastrinomas. Archives of Surgery. 125(12). 1584–1584. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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