Mark Meuth

11.3k total citations · 2 hit papers
71 papers, 8.9k citations indexed

About

Mark Meuth is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Mark Meuth has authored 71 papers receiving a total of 8.9k indexed citations (citations by other indexed papers that have themselves been cited), including 59 papers in Molecular Biology, 19 papers in Oncology and 14 papers in Cancer Research. Recurrent topics in Mark Meuth's work include DNA Repair Mechanisms (26 papers), Biochemical and Molecular Research (18 papers) and CRISPR and Genetic Engineering (16 papers). Mark Meuth is often cited by papers focused on DNA Repair Mechanisms (26 papers), Biochemical and Molecular Research (18 papers) and CRISPR and Genetic Engineering (16 papers). Mark Meuth collaborates with scholars based in United Kingdom, United States and Canada. Mark Meuth's co-authors include Howard Green, Thomas Helleday, Niklas Schultz, Helen E. Bryant, Suzanne Kyle, Nicola J. Curtin, Elena López‐Knowles, Huw D. Thomas, James W.F. Catto and Freddie C. Hamdy and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Mark Meuth

68 papers receiving 8.7k citations

Hit Papers

Specific killing of BRCA2-deficien... 1974 2026 1991 2008 2005 1974 1000 2.0k 3.0k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark Meuth United Kingdom 37 6.7k 3.8k 1.5k 1.1k 875 71 8.9k
Michael G. Brattain United States 56 6.8k 1.0× 4.1k 1.1× 1.7k 1.2× 819 0.8× 753 0.9× 219 10.3k
J.M. Trent United States 16 7.0k 1.0× 5.5k 1.5× 1.6k 1.1× 980 0.9× 322 0.4× 25 9.6k
Eliot M. Rosen United States 57 4.7k 0.7× 1.8k 0.5× 1.2k 0.9× 1.5k 1.4× 630 0.7× 145 7.9k
Samuel Benchimol Canada 46 5.7k 0.8× 4.6k 1.2× 1.4k 0.9× 971 0.9× 358 0.4× 94 8.8k
Kristoffer Valerie United States 60 6.4k 0.9× 3.9k 1.0× 1.6k 1.1× 1.2k 1.1× 443 0.5× 164 10.4k
Donna L. George United States 39 8.4k 1.3× 7.0k 1.9× 2.1k 1.5× 1.0k 0.9× 472 0.5× 80 11.7k
Todd Waldman United States 39 8.0k 1.2× 5.7k 1.5× 2.0k 1.3× 696 0.6× 436 0.5× 66 10.9k
Asha S. Multani United States 40 5.0k 0.7× 3.3k 0.9× 1.9k 1.3× 691 0.6× 457 0.5× 126 8.2k
Edward V. Prochownik United States 53 6.4k 1.0× 2.1k 0.5× 1.8k 1.2× 659 0.6× 363 0.4× 170 8.6k
Khandan Keyomarsi United States 51 6.7k 1.0× 5.5k 1.5× 2.2k 1.5× 955 0.9× 664 0.8× 158 10.7k

Countries citing papers authored by Mark Meuth

Since Specialization
Citations

This map shows the geographic impact of Mark Meuth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Meuth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Meuth more than expected).

Fields of papers citing papers by Mark Meuth

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Meuth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Meuth. The network helps show where Mark Meuth may publish in the future.

Co-authorship network of co-authors of Mark Meuth

This figure shows the co-authorship network connecting the top 25 collaborators of Mark Meuth. A scholar is included among the top collaborators of Mark Meuth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark Meuth. Mark Meuth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Meuth, Mark. (2014). Inhibitors of cell cycle checkpoints and DNA replication cause different responses in normal versus malignant urothelial cells. Molecular & Cellular Oncology. 1(4). e968508–e968508. 2 indexed citations
2.
Hyka‐Nouspikel, Nevila, Joëlle A. Desmarais, Paul J. Gokhale, et al.. (2012). Deficient DNA Damage Response and Cell Cycle Checkpoints Lead to Accumulation of Point Mutations in Human Embryonic Stem Cells. Stem Cells. 30(9). 1901–1910. 43 indexed citations
3.
Gagou, Mary E., Anil Ganesh, Ruth Thompson, et al.. (2011). Suppression of Apoptosis by PIF1 Helicase in Human Tumor Cells. Cancer Research. 71(14). 4998–5008. 32 indexed citations
4.
Meuth, Mark. (2010). Chk1 suppressed cell death. Cell Division. 5(1). 21–21. 28 indexed citations
5.
Catto, James W.F., Saiful Miah, Helen C. Owen, et al.. (2009). Distinct MicroRNA Alterations Characterize High- and Low-Grade Bladder Cancer. Cancer Research. 69(21). 8472–8481. 272 indexed citations
6.
Myers, Katie, Mary E. Gagou, Pedro Zuazua‐Villar, René Rodrı́guez, & Mark Meuth. (2009). ATR and Chk1 Suppress a Caspase-3–Dependent Apoptotic Response Following DNA Replication Stress. PLoS Genetics. 5(1). e1000324–e1000324. 109 indexed citations
7.
Yates, David, Ishtiaq Rehman, Maysam Abbod, et al.. (2007). Promoter Hypermethylation Identifies Progression Risk in Bladder Cancer. Clinical Cancer Research. 13(7). 2046–2053. 143 indexed citations
8.
Azzouzi, Abdel‐Rahmène, James W.F. Catto, Ishtiaq Rehman, et al.. (2007). Clinically localised prostate cancer is microsatellite stable. British Journal of Urology. 99(5). 1031–1035. 12 indexed citations
9.
Oers, Johanna M.M. van, Ellen C. Zwarthoff, Ishtiaq Rehman, et al.. (2006). FGFR3 mutations have different effects on disease course in tumors from the bladder, ureter, and renal pelvis. Cancer Research. 66. 1059–1059.
10.
Bryant, Helen E., Niklas Schultz, Huw D. Thomas, et al.. (2005). Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase. Nature. 434(7035). 913–917. 3780 indexed citations breakdown →
11.
Macpherson, Gordon R., Catherine S. Healey, M. Dawn Teare, et al.. (2004). Association of a Common Variant of the CASP8 Gene With Reduced Risk of Breast Cancer. JNCI Journal of the National Cancer Institute. 96(24). 1866–1869. 126 indexed citations
13.
Catto, James W.F., Abdel-Rahmène Azzouzi, Najla Amira, et al.. (2003). Distinct patterns of microsatellite instability are seen in tumours of the urinary tract. Oncogene. 22(54). 8699–8706. 111 indexed citations
14.
Lundin, Cecilia, Klaus Erixon, Catherine Arnaudeau, et al.. (2002). Different Roles for Nonhomologous End Joining and Homologous Recombination following Replication Arrest in Mammalian Cells. Molecular and Cellular Biology. 22(16). 5869–5878. 199 indexed citations
15.
Phear, Geraldine, et al.. (1990). DNA sequence analysis of gamma radiation—induced deletions and insertions at the APRT locus of hamster cells. Molecular Carcinogenesis. 3(4). 233–242. 26 indexed citations
16.
Meuth, Mark, et al.. (1989). G-repeats: a novel hamster sine family. Nucleic Acids Research. 17(18). 7221–7228. 3 indexed citations
17.
Meuth, Mark, et al.. (1989). Hamster line and ALU-equivalent sequences are present in the small polydispersed circular DNA population of CHO cells. Nucleic Acids Research. 17(14). 5846–5846. 1 indexed citations
18.
Nalbantoglu, Joséphine, et al.. (1988). Insertion of unique and repetitive DNA fragments into the aprt locus of hamster cells. Journal of Molecular Biology. 200(3). 449–459. 28 indexed citations
19.
Nalbantoglu, Joséphine, Geraldine Phear, & Mark Meuth. (1987). DNA Sequence Analysis of Spontaneous Mutations at the aprt Locus of Hamster Cells. Molecular and Cellular Biology. 7(4). 1445–1449. 2 indexed citations
20.
Meuth, Mark, et al.. (1982). A selection system specific for the thy mutator phenotype. Somatic Cell and Molecular Genetics. 8(4). 423–432. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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