Mark I. Rees

8.5k citations
120 papers · 4.5k indexed · h-index 41

Mark I. Rees

118 papers receiving 4.4k citations

Peers

Mark I. Rees
Comparison fields: 5 of 135
  • Cellular and Molecular Neuroscience 1.5k
  • Developmental Neuroscience 250
  • Psychiatry and Mental health 841
  • Neurology 318
  • Cell Biology 505
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Citations per year

Countries citing papers authored by Mark I. Rees

Since Specialization
Citations

This map shows the geographic impact of Mark I. Rees's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark I. Rees with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark I. Rees more than expected).

Fields of papers citing papers by Mark I. Rees

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark I. Rees. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark I. Rees. The network helps show where Mark I. Rees may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Mark I. Rees, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mark I. Rees Line = papers co-authored together Mark I. Rees links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 202311
2 20223
3 201941
4 20185
5 201561
6 201253
7 201213
8 20107
9 201025
10 201023
11 2010128
12 20092
13 2008154
14 200710
15 200316
16
A missense mutation of human Gephyrin (GPHN) is associated with Hyperekplexia and transcript isoform analysis re-defines the genomic structure of GPHN
20011
17
Reduction in voltage-dependent calcium channel function in cerebellar Purkinje cells of the mouse mutant ducky, which has a null mutation for the calcium channel accessory subunit alpha 2 delta 2
20002
18 20003
19
A transient hyperekplexia phenotype associated with compound heterozygote mutations in the human beta-subunit of the inhibitory glycine receptor (GLRB)
20002
20 199553

About Mark I. Rees

Mark I. Rees is a scholar working on Psychiatry and Mental health, Cellular and Molecular Neuroscience, Genetics, Developmental Neuroscience and Pediatrics, Perinatology and Child Health, having authored 120 papers that have together received 4.5k indexed citations. Recurring topics across this work include Epilepsy research and treatment (25 papers), Ion channel regulation and function (20 papers), Neuroscience and Neuropharmacology Research (20 papers), Genetics and Neurodevelopmental Disorders (19 papers), Genomics and Rare Diseases (16 papers), Pharmacological Effects and Toxicity Studies (13 papers), Cardiac electrophysiology and arrhythmias (10 papers) and Mitochondrial Function and Pathology (8 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.5k citations), Developmental Neuroscience (250 citations), Psychiatry and Mental health (841 citations), Neurology (318 citations) and Cell Biology (505 citations). Mark I. Rees has collaborated with scholars based in United Kingdom, Australia and New Zealand. Frequent co-authors include Richard L. M. Faull, Kristin Baer, Seo‐Kyung Chung, Victoria L. Harvey, Kirsten Harvey, Henry J. Waldvogel, Michael J. Owen, Rhys H. Thomas, Maurice A. Curtis and Maya Topf. Their work appears in journals such as Journal of Neurology Neurosurgery & Psychiatry, Human Molecular Genetics, Seizure, Heart Rhythm and Epilepsia.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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