Mark I. Rees

8.5k total citations
120 papers, 4.5k citations indexed

About

Mark I. Rees is a scholar working on Molecular Biology, Genetics and Psychiatry and Mental health. According to data from OpenAlex, Mark I. Rees has authored 120 papers receiving a total of 4.5k indexed citations (citations by other indexed papers that have themselves been cited), including 52 papers in Molecular Biology, 35 papers in Genetics and 29 papers in Psychiatry and Mental health. Recurrent topics in Mark I. Rees's work include Epilepsy research and treatment (25 papers), Ion channel regulation and function (20 papers) and Neuroscience and Neuropharmacology Research (20 papers). Mark I. Rees is often cited by papers focused on Epilepsy research and treatment (25 papers), Ion channel regulation and function (20 papers) and Neuroscience and Neuropharmacology Research (20 papers). Mark I. Rees collaborates with scholars based in United Kingdom, Australia and New Zealand. Mark I. Rees's co-authors include Richard L. M. Faull, Kristin Baer, Seo‐Kyung Chung, Victoria L. Harvey, Kirsten Harvey, Henry J. Waldvogel, Michael J. Owen, Rhys H. Thomas, Maurice A. Curtis and Maya Topf and has published in prestigious journals such as Journal of Biological Chemistry, Nature Genetics and Journal of Neuroscience.

In The Last Decade

Mark I. Rees

118 papers receiving 4.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mark I. Rees United Kingdom	41	2.1k	1.5k	853	841	505	120	4.5k
Yuwu Jiang China	30	1.6k 0.8×	905 0.6×	869 1.0×	842 1.0×	150 0.3×	227	3.2k
Jennifer M. Kwon United States	29	1.2k 0.6×	861 0.6×	634 0.7×	280 0.3×	398 0.8×	77	3.4k
Rudy Van Coster Belgium	39	3.7k 1.7×	665 0.4×	464 0.5×	308 0.4×	298 0.6×	156	5.5k
Masayuki Itoh Japan	32	1.6k 0.7×	801 0.5×	890 1.0×	311 0.4×	300 0.6×	151	3.4k
Angela M. Kaindl Germany	34	2.0k 0.9×	960 0.6×	568 0.7×	480 0.6×	480 1.0×	166	4.6k
Guoyin Feng China	40	2.6k 1.2×	933 0.6×	1.3k 1.5×	732 0.9×	251 0.5×	172	5.0k
Annapurna Poduri United States	38	2.4k 1.1×	899 0.6×	2.3k 2.7×	1.7k 2.0×	320 0.6×	151	5.4k
H. Ronald Zielke United States	30	2.2k 1.0×	1.6k 1.0×	609 0.7×	423 0.5×	182 0.4×	69	4.6k
Michael Wong United States	47	3.1k 1.5×	2.3k 1.5×	1.5k 1.7×	1.4k 1.7×	480 1.0×	151	7.6k
Yves Robitaille Canada	41	2.4k 1.2×	1.7k 1.1×	332 0.4×	640 0.8×	135 0.3×	127	5.3k

Countries citing papers authored by Mark I. Rees

Since Specialization

Citations

This map shows the geographic impact of Mark I. Rees's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark I. Rees with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark I. Rees more than expected).

Fields of papers citing papers by Mark I. Rees

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark I. Rees. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark I. Rees. The network helps show where Mark I. Rees may publish in the future.

Co-authorship network of co-authors of Mark I. Rees

This figure shows the co-authorship network connecting the top 25 collaborators of Mark I. Rees. A scholar is included among the top collaborators of Mark I. Rees based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark I. Rees. Mark I. Rees is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Watkins, Lewis M., Michelle Naughton, Marie Dittmer, et al.. (2023). Complement activation and increased anaphylatoxin receptor expression are associated with cortical grey matter lesions and the compartmentalised inflammatory response of multiple sclerosis. Frontiers in Cellular Neuroscience. 17. 1094106–1094106. 11 indexed citations

Grinton, Bronwyn E., Liam G. Fearnley, Ingrid E. Scheffer, et al.. (2022). A founder event causing a dominant childhood epilepsy survives 800 years through weak selective pressure. The American Journal of Human Genetics. 109(11). 2080–2087. 3 indexed citations

Fonferko‐Shadrach, Beata, Arron Lacey, Angus Roberts, et al.. (2019). Using natural language processing to extract structured epilepsy data from unstructured clinic letters: development and validation of the ExECT (extraction of epilepsy clinical text) system. BMJ Open. 9(4). e023232–e023232. 41 indexed citations

Sen, Arjune, Patricia Dugan, Piero Perucca, et al.. (2018). The phenotype of bilateral hippocampal sclerosis and its management in “real life” clinical settings. Epilepsia. 59(7). 1410–1420. 5 indexed citations

Oegema, Renske, Thomas D. Cushion, Ian G. Phelps, et al.. (2015). Recognizable cerebellar dysplasia associated with mutations in multiple tubulin genes. Human Molecular Genetics. 24(18). 5313–5325. 61 indexed citations

Pickrell, William Owen, Arron Lacey, Rhys H. Thomas, Phil E M Smith, & Mark I. Rees. (2012). Weight change associated with antiepileptic drugs. Journal of Neurology Neurosurgery & Psychiatry. 84(7). 796–799. 53 indexed citations

Pickrell, William Owen, Mark I. Rees, & Seo‐Kyung Chung. (2012). Next Generation Sequencing Methodologies - An Overview. Advances in protein chemistry and structural biology. 89. 1–26. 13 indexed citations

Thomas, Rhys H., et al.. (2010). Implications for families of advances in understanding the genetic basis of epilepsy. Seizure. 19(10). 675–679. 7 indexed citations

Rees, Mark I.. (2010). The genetics of epilepsy—The past, the present and future. Seizure. 19(10). 680–683. 25 indexed citations

10.

Lane, M. R., Jackie Crawford, Seo‐Kyung Chung, et al.. (2010). Elevated serum gastrin levels in Jervell and Lange-Nielsen syndrome: A marker of severe KCNQ1 dysfunction?. Heart Rhythm. 8(4). 551–554. 23 indexed citations

11.

Jacobsen, Jessie C., C. Simon Bawden, Skye R. Rudiger, et al.. (2010). An ovine transgenic Huntington's disease model. Human Molecular Genetics. 19(10). 1873–1882. 128 indexed citations

12.

Johnston, Janet, Mark I. Rees, & Pamela E. Smith. (2009). Epilepsy genetics: clinical beginnings and social consequences. QJM. 102(7). 497–499. 2 indexed citations

13.

Harvey, Victoria L., Maya Topf, Kirsten Harvey, & Mark I. Rees. (2008). The genetics of hyperekplexia: more than startle!. Trends in Genetics. 24(9). 439–447. 154 indexed citations

14.

Skinner, Jonathan R., et al.. (2007). Coinheritance of long QT syndrome and Kearns-Sayre syndrome. Heart Rhythm. 4(12). 1568–1572. 10 indexed citations

15.

Waldvogel, Henry J., et al.. (2003). GABAA receptor subunit and gephyrin protein changes differ in the globus pallidus in Huntington's diseased brain. Brain Research. 994(2). 265–270. 16 indexed citations

16.

Rees, Mark I., Kristin Baer, Hamish E. Ward, et al.. (2001). A missense mutation of human Gephyrin (GPHN) is associated with Hyperekplexia and transcript isoform analysis re-defines the genomic structure of GPHN. ORCA Online Research @Cardiff (Cardiff University). 1 indexed citations

17.

Barclay, J. Elaine, et al.. (2000). Reduction in voltage-dependent calcium channel function in cerebellar Purkinje cells of the mouse mutant ducky, which has a null mutation for the calcium channel accessory subunit alpha 2 delta 2. UCL Discovery (University College London). 2 indexed citations

18.

Watts, Patrick, Mark I. Rees, Angus Clarke, et al.. (2000). Linkage analysis in an autosomal dominant ‘zonular nuclear pulverulent’ congenital cataract, mapped to chromosome 13q11-13. Eye. 14(2). 172–175. 3 indexed citations

19.

Rees, Mark I., T. Lewis, Geert Mortier, et al.. (2000). A transient hyperekplexia phenotype associated with compound heterozygote mutations in the human beta-subunit of the inhibitory glycine receptor (GLRB). ORCA Online Research @Cardiff (Cardiff University). 2 indexed citations

20.

Craddock, Nick, et al.. (1995). No evidence for allelic association between bipolar disorder and monoamine oxidase a gene polymorphisms. American Journal of Medical Genetics. 60(4). 322–324. 53 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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