Mark Hazel

9 papers receiving 929 citations

Mark Hazel's Hit Papers

Retinopathy induced in mice by targeted disruption of the rhodopsin gene 1997 · 453 citations
4530+9+19Years since publication100200300400

Peers

Mark Hazel
Comparison fields: 5 of 74
  • Cellular and Molecular Neuroscience 216
  • Ophthalmology 92
  • Molecular Biology 729
  • Rehabilitation 51
  • Cell Biology 129
Replace Ilaria Palmisano with:
Ilaria Palmisano United Kingdom
Andrea Flaccus Germany
Selma A. Serra Spain
Véronique Planchamp Germany
Irfan Y. Tamboli Germany
Kenneth P. Mitton United States
Gerald Wallace United States
Praveena Gupta United States
Kaori Kawai Japan
Chandana Kondapalli United Kingdom
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Citations per field
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Citations per year

Countries citing papers authored by Mark Hazel

Since Specialization
Citations

This map shows the geographic impact of Mark Hazel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Hazel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Hazel more than expected).

Fields of papers citing papers by Mark Hazel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Hazel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Hazel. The network helps show where Mark Hazel may publish in the future.

Co-authors

The 25 scholars most cited alongside Mark Hazel, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mark Hazel Line = papers co-authored together Mark Hazel links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1
Retinopathy induced in mice by targeted disruption of the rhodopsin gene
Hit paper breakdown →
1997453
2 2002272
3 201199
4 200357
5 200436
6 200515
7 19556
8 19595
9 19963

About Mark Hazel

Mark Hazel is a scholar working on Molecular Biology, Surgery, Cell Biology, Genetics and Cancer Research, having authored 9 papers that have together received 946 indexed citations. Recurring topics across this work include Pancreatic function and diabetes (3 papers), Cancer, Hypoxia, and Metabolism (2 papers), Estrogen and related hormone effects (1 paper), Glycosylation and Glycoproteins Research (1 paper), Muscle metabolism and nutrition (1 paper), Diabetes and associated disorders (1 paper), Muscle Physiology and Disorders (1 paper) and Aerodynamics and Acoustics in Jet Flows (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (216 citations), Ophthalmology (92 citations), Molecular Biology (729 citations), Rehabilitation (51 citations) and Cell Biology (129 citations). Mark Hazel has collaborated with scholars based in United States, Ireland and Hungary. Frequent co-authors include Donald A. McClain, Robert C. Cooksey, Glendon J. Parker, Dona C. Love, John A. Hanover, Peter Humphries, Ronald A. Bush, Derrick E. Rancourt, Paul A. Sieving and Alexandra Erven. Their work appears in journals such as American Journal of Physiology-Endocrinology and Metabolism, Endocrinology, Experimental Biology and Medicine, Journal of Experimental Biology and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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