Mark E. Robinson

676 total citations
17 papers, 236 citations indexed

About

Mark E. Robinson is a scholar working on Molecular Biology, Hematology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Mark E. Robinson has authored 17 papers receiving a total of 236 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 6 papers in Hematology and 4 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Mark E. Robinson's work include Acute Lymphoblastic Leukemia research (4 papers), Chronic Myeloid Leukemia Treatments (3 papers) and Acute Myeloid Leukemia Research (2 papers). Mark E. Robinson is often cited by papers focused on Acute Lymphoblastic Leukemia research (4 papers), Chronic Myeloid Leukemia Treatments (3 papers) and Acute Myeloid Leukemia Research (2 papers). Mark E. Robinson collaborates with scholars based in United States, United Kingdom and France. Mark E. Robinson's co-authors include Niklas Feldhahn, Leandro Castellano, Carles Gaston‐Massuet, Féaron C. Cassidy, Mathieu-Benoı̂t Voisin, M. Paula Longhi, Valeria Scagliotti, Federica M. Marelli‐Berg, Jakob Loschko and Marika Charalambous and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Blood.

In The Last Decade

Mark E. Robinson

15 papers receiving 234 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark E. Robinson United States 6 96 81 79 61 24 17 236
Sandra Schmidhofer Germany 8 233 2.4× 115 1.4× 110 1.4× 58 1.0× 28 1.2× 9 412
Sandra Rodríguez-López Spain 9 111 1.2× 89 1.1× 41 0.5× 72 1.2× 11 0.5× 13 267
Myrthe den Toom Netherlands 10 80 0.8× 145 1.8× 75 0.9× 41 0.7× 19 0.8× 18 284
Kaida Mu China 10 89 0.9× 86 1.1× 27 0.3× 35 0.6× 12 0.5× 22 306
Chloé Paul France 8 98 1.0× 140 1.7× 96 1.2× 39 0.6× 6 0.3× 24 474
Katarzyna Parzych United Kingdom 7 135 1.4× 45 0.6× 48 0.6× 20 0.3× 10 0.4× 7 233
Yara Barreira France 5 73 0.8× 125 1.5× 56 0.7× 18 0.3× 14 0.6× 6 259
Toshiyuki Niiya Japan 7 63 0.7× 43 0.5× 172 2.2× 115 1.9× 27 1.1× 13 297
Kristine M. Morin United States 4 58 0.6× 67 0.8× 67 0.8× 51 0.8× 77 3.2× 4 274

Countries citing papers authored by Mark E. Robinson

Since Specialization
Citations

This map shows the geographic impact of Mark E. Robinson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark E. Robinson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark E. Robinson more than expected).

Fields of papers citing papers by Mark E. Robinson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark E. Robinson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark E. Robinson. The network helps show where Mark E. Robinson may publish in the future.

Co-authorship network of co-authors of Mark E. Robinson

This figure shows the co-authorship network connecting the top 25 collaborators of Mark E. Robinson. A scholar is included among the top collaborators of Mark E. Robinson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark E. Robinson. Mark E. Robinson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Lee, Sungwoon, Mark E. Robinson, Richard J. Antaya, et al.. (2025). PIEZO1 Overexpression in Hereditary Hemorrhagic Telangiectasia Arteriovenous Malformations. Circulation. 152(9). 599–615. 2 indexed citations
2.
Léveillé, Etienne, Mark E. Robinson, Thierry Bertomeu, et al.. (2025). Abstract 2819: Metabolic determinants of ferroptosis in B-cell malignancies. Cancer Research. 85(8_Supplement_1). 2819–2819.
3.
Geraldo, Luiz Henrique, Yunling Xu, Levi L. Blazer, et al.. (2024). Monoclonal antibodies that block Roundabout 1 and 2 signaling target pathological ocular neovascularization through myeloid cells. Science Translational Medicine. 16(774). eadn8388–eadn8388. 2 indexed citations
4.
Geraldo, Luiz Henrique, Bartlomiej Bartkowiak, Mathilde Poulet, et al.. (2024). Interplay between Netrin-1 and Norrin controls arteriovenous zonation of blood–retina barrier integrity. Proceedings of the National Academy of Sciences. 121(52). e2408674121–e2408674121. 3 indexed citations
5.
Robinson, Mark E., et al.. (2024). Promoter‐centred chromatin interactions associated with EVI1 expression in EVI1 +3q− myeloid leukaemia cells. British Journal of Haematology. 204(3). 945–958.
6.
Léveillé, Etienne, Mark E. Robinson, Thierry Bertomeu, et al.. (2024). Metabolic Determinants of Ferroptosis in B-Cell Lymphoma. Blood. 144(Supplement 1). 976–976. 1 indexed citations
7.
May, Philippa C., Alistair Reid, Mark E. Robinson, et al.. (2023). FISH-negative BCR::ABL1-positive e19a2 chronic myeloid leukaemia: the most cryptic of insertions. BMC Medical Genomics. 16(1). 172–172. 4 indexed citations
8.
Kume, Kohei, Zhengshan Chen, Lai N. Chan, et al.. (2022). Divergent MYC- and BCL6-Driven Metabolic Programs Enable Dynamic Regulation of Cell Biomass in B-Cell Malignancies. Blood. 140(Supplement 1). 5900–5901. 1 indexed citations
9.
Pan, Lili, Chao Hong, Lai N. Chan, et al.. (2021). PON2 subverts metabolic gatekeeper functions in B cells to promote leukemogenesis. Proceedings of the National Academy of Sciences. 118(7). 13 indexed citations
10.
Robinson, Mark E., Valentina S. Caputo, Xiaolin Xiao, et al.. (2021). The innate sensor ZBP1-IRF3 axis regulates cell proliferation in multiple myeloma. Haematologica. 107(3). 721–732. 28 indexed citations
11.
Chan, Lai N., Christian Hurtz, Etienne Léveillé, et al.. (2021). Identification of BCL6 As Synthetic Lethality in RAS-Driven B-Cell Transformation. Blood. 138(Supplement 1). 792–792. 1 indexed citations
12.
Claudiani, Simone, Clinton C. Mason, Dragana Milojković, et al.. (2021). Carfilzomib Enhances the Suppressive Effect of Ruxolitinib in Myelofibrosis. Cancers. 13(19). 4863–4863. 1 indexed citations
13.
Caputo, Valentina S., Xiaolin Xiao, Mark E. Robinson, et al.. (2020). Brd2/4 and Myc regulate alternative cell lineage programmes during early osteoclast differentiation in vitro. iScience. 24(1). 101989–101989. 13 indexed citations
14.
Parzych, Katarzyna, Paula Saavedra-García, Gabriel N. Valbuena, et al.. (2019). The coordinated action of VCP/p97 and GCN2 regulates cancer cell metabolism and proteostasis during nutrient limitation. Oncogene. 38(17). 3216–3231. 33 indexed citations
15.
Pfeifer, Matthias, et al.. (2019). SSB1/SSB2 Proteins Safeguard B Cell Development by Protecting the Genomes of B Cell Precursors. The Journal of Immunology. 202(12). 3423–3433. 5 indexed citations
16.
Wood, Elizabeth G., Jakob Loschko, Valeria Scagliotti, et al.. (2018). Visceral Adipose Tissue Immune Homeostasis Is Regulated by the Crosstalk between Adipocytes and Dendritic Cell Subsets. Cell Metabolism. 27(3). 588–601.e4. 116 indexed citations
17.
Robinson, Mark E., Philippa C. May, Leandro Castellano, et al.. (2017). Lineage-Specific Genes Are Prominent DNA Damage Hotspots during Leukemic Transformation of B Cell Precursors. Cell Reports. 18(7). 1687–1698. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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