Mark E. Hemric

1.0k total citations
17 papers, 864 citations indexed

About

Mark E. Hemric is a scholar working on Cardiology and Cardiovascular Medicine, Cell Biology and Molecular Biology. According to data from OpenAlex, Mark E. Hemric has authored 17 papers receiving a total of 864 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Cardiology and Cardiovascular Medicine, 7 papers in Cell Biology and 6 papers in Molecular Biology. Recurrent topics in Mark E. Hemric's work include Cardiomyopathy and Myosin Studies (9 papers), Muscle Physiology and Disorders (6 papers) and Cellular Mechanics and Interactions (5 papers). Mark E. Hemric is often cited by papers focused on Cardiomyopathy and Myosin Studies (9 papers), Muscle Physiology and Disorders (6 papers) and Cellular Mechanics and Interactions (5 papers). Mark E. Hemric collaborates with scholars based in United States and France. Mark E. Hemric's co-authors include Joseph M. Chalovich, Madeleine W. Cunningham, Jeffrey E. Galvin, Kent E. Ward, Joe R. Haeberle, Stanley D. Kosanke, Anthony Quinn, Ya Li, David M. Warshaw and Kathleen M. Trybus and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The Journal of Immunology.

In The Last Decade

Mark E. Hemric

17 papers receiving 841 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark E. Hemric United States 14 417 392 250 194 118 17 864
Jørgen Tranum‐Jensen Denmark 15 63 0.2× 445 1.1× 216 0.9× 63 0.3× 315 2.7× 36 1.0k
Hannes Perschinka Austria 7 83 0.2× 481 1.2× 110 0.4× 41 0.2× 311 2.6× 8 839
Donn Stewart United States 8 451 1.1× 131 0.3× 159 0.6× 65 0.3× 81 0.7× 11 942
Yinghong Ma United States 20 95 0.2× 694 1.8× 45 0.2× 111 0.6× 139 1.2× 25 1.5k
Jing An China 15 96 0.2× 371 0.9× 31 0.1× 126 0.6× 65 0.6× 53 872
Nina Hofmann Germany 16 243 0.6× 434 1.1× 32 0.1× 22 0.1× 62 0.5× 40 988
Scott J. Hempson United States 11 78 0.2× 529 1.3× 43 0.2× 29 0.1× 65 0.6× 11 1.0k
Christoph Hauser Switzerland 15 79 0.2× 235 0.6× 39 0.2× 92 0.5× 36 0.3× 51 849
Masako Okada Japan 16 66 0.2× 196 0.5× 30 0.1× 55 0.3× 61 0.5× 65 747
Ling Kong United States 13 30 0.1× 381 1.0× 58 0.2× 107 0.6× 53 0.4× 28 756

Countries citing papers authored by Mark E. Hemric

Since Specialization
Citations

This map shows the geographic impact of Mark E. Hemric's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark E. Hemric with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark E. Hemric more than expected).

Fields of papers citing papers by Mark E. Hemric

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark E. Hemric. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark E. Hemric. The network helps show where Mark E. Hemric may publish in the future.

Co-authorship network of co-authors of Mark E. Hemric

This figure shows the co-authorship network connecting the top 25 collaborators of Mark E. Hemric. A scholar is included among the top collaborators of Mark E. Hemric based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark E. Hemric. Mark E. Hemric is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Li, Ya, Janet S. Heuser, Stanley D. Kosanke, Mark E. Hemric, & Madeleine W. Cunningham. (2005). Protection against Experimental Autoimmune Myocarditis Is Mediated by Interleukin-10-Producing T Cells that Are Controlled by Dendritic Cells. American Journal Of Pathology. 167(1). 5–15. 33 indexed citations
2.
Li, Ya, Janet S. Heuser, Stanley D. Kosanke, Mark E. Hemric, & Madeleine W. Cunningham. (2004). Cryptic Epitope Identified in Rat and Human Cardiac Myosin S2 Region Induces Myocarditis in the Lewis Rat. The Journal of Immunology. 172(5). 3225–3234. 53 indexed citations
3.
Hemric, Mark E., et al.. (2004). Caldesmon Tethers Myosin V to Actin and Facilitates in vitro Motility. Journal of Muscle Research and Cell Motility. 25(2). 141–148. 2 indexed citations
4.
Galvin, Jeffrey E., Mark E. Hemric, Stanley D. Kosanke, et al.. (2002). Induction of Myocarditis and Valvulitis in Lewis Rats by Different Epitopes of Cardiac Myosin and Its Implications in Rheumatic Carditis. American Journal Of Pathology. 160(1). 297–306. 76 indexed citations
5.
Galvin, Jeffrey E., Mark E. Hemric, Kent E. Ward, & Madeleine W. Cunningham. (2000). Cytotoxic mAb from rheumatic carditis recognizes heart valves and laminin. Journal of Clinical Investigation. 106(2). 217–224. 176 indexed citations
6.
Quinn, Anthony, Kent E. Ward, Vincent A. Fischetti, Mark E. Hemric, & Madeleine W. Cunningham. (1998). Immunological Relationship between the Class I Epitope of Streptococcal M Protein and Myosin. Infection and Immunity. 66(9). 4418–4424. 1 indexed citations
7.
Quinn, Anthony, Kent E. Ward, Vincent A. Fischetti, Mark E. Hemric, & Madeleine W. Cunningham. (1998). Immunological Relationship between the Class I Epitope of Streptococcal M Protein and Myosin. Infection and Immunity. 66(9). 4418–4424. 29 indexed citations
8.
Haeberle, Joe R. & Mark E. Hemric. (1995). Are actin filaments moving under unloaded conditions in the in vitro motility assay?. PubMed. 68(4 Suppl). 306S–310S; discussion 310S. 16 indexed citations
9.
Haeberle, Joe R. & Mark E. Hemric. (1994). A model for the coregulation of smooth muscle actomyosin by caldesmon, calponin, tropomyosin, and the myosin regulatory light chain. Canadian Journal of Physiology and Pharmacology. 72(11). 1400–1409. 20 indexed citations
10.
Hemric, Mark E., Paula B. Tracy, & Joe R. Haeberle. (1994). Caldesmon enhances the binding of myosin to the cytoskeleton during platelet activation.. Journal of Biological Chemistry. 269(6). 4125–4128. 26 indexed citations
11.
Hemric, Mark E., et al.. (1993). Inhibition of Actin Stimulation of Skeletal Muscle (A1)S-1 ATPase Activity by Caldesmon. Archives of Biochemistry and Biophysics. 306(1). 39–43. 10 indexed citations
12.
Hemric, Mark E., et al.. (1993). Reversal of caldesmon binding to myosin with calcium-calmodulin or by phosphorylating caldesmon. Journal of Biological Chemistry. 268(20). 15305–15311. 25 indexed citations
13.
Haeberle, Joe R., Kathleen M. Trybus, Mark E. Hemric, & David M. Warshaw. (1992). The effects of smooth muscle caldesmon on actin filament motility. Journal of Biological Chemistry. 267(32). 23001–23006. 66 indexed citations
14.
Chalovich, Joseph M., et al.. (1990). Regulation of ATP Hydrolysis by Caldesmon. Annals of the New York Academy of Sciences. 599(1). 85–99. 22 indexed citations
15.
Hemric, Mark E. & Joseph M. Chalovich. (1990). Characterization of caldesmon binding to myosin.. Journal of Biological Chemistry. 265(32). 19672–19678. 72 indexed citations
16.
Hemric, Mark E., et al.. (1989). The binding of Caldesmon to Actin and Its Effect on the ATPase Activity of Soluble Myosin Subfragments in the Presence and Absence of Tropomyosin. Journal of Biological Chemistry. 264(16). 9602–9610. 89 indexed citations
17.
Hemric, Mark E. & Joseph M. Chalovich. (1988). Effect of caldesmon on the ATPase activity and the binding of smooth and skeletal myosin subfragments to actin.. Journal of Biological Chemistry. 263(4). 1878–1885. 148 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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