Mark E. Drotar

1.2k total citations
15 papers, 753 citations indexed

About

Mark E. Drotar is a scholar working on Oncology, Immunology and Molecular Biology. According to data from OpenAlex, Mark E. Drotar has authored 15 papers receiving a total of 753 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Oncology, 5 papers in Immunology and 4 papers in Molecular Biology. Recurrent topics in Mark E. Drotar's work include Viral-associated cancers and disorders (4 papers), Lymphoma Diagnosis and Treatment (4 papers) and Immune Cell Function and Interaction (3 papers). Mark E. Drotar is often cited by papers focused on Viral-associated cancers and disorders (4 papers), Lymphoma Diagnosis and Treatment (4 papers) and Immune Cell Function and Interaction (3 papers). Mark E. Drotar collaborates with scholars based in United Kingdom, United States and Saudi Arabia. Mark E. Drotar's co-authors include Peter Burke, R. Armour Forse, Tony McBryan, Joanna B. Wilson, Peter D. Adams, John van Tuyn, Wuning Zhong, Penelope M. Tsimbouri, S.R. Chavali and Shelley L. Berger and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Mark E. Drotar

14 papers receiving 746 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mark E. Drotar United Kingdom	9	401	187	163	157	146	15	753
Joshua J. Oaks United States	9	710 1.8×	115 0.6×	78 0.5×	90 0.6×	169 1.2×	19	876
Guojin Wu United States	14	369 0.9×	184 1.0×	63 0.4×	260 1.7×	128 0.9×	33	798
Sandrine Guérin France	11	466 1.2×	185 1.0×	62 0.4×	257 1.6×	93 0.6×	16	816
Tetsuya Kurosu Japan	18	473 1.2×	186 1.0×	40 0.2×	97 0.6×	327 2.2×	35	831
Mingqiang Ren United States	18	586 1.5×	127 0.7×	40 0.2×	98 0.6×	127 0.9×	41	837
Marie‐France Gaumont‐Leclerc Canada	10	668 1.7×	247 1.3×	318 2.0×	122 0.8×	71 0.5×	11	883
Chris Bartholomew United Kingdom	17	587 1.5×	134 0.7×	45 0.3×	113 0.7×	243 1.7×	36	904
I‐Chu Tseng United States	13	256 0.6×	89 0.5×	122 0.7×	92 0.6×	100 0.7×	15	630
Federica Falà Italy	13	505 1.3×	260 1.4×	25 0.2×	231 1.5×	99 0.7×	18	831
Junko Horiguchi‐Yamada Japan	14	300 0.7×	131 0.7×	78 0.5×	69 0.4×	85 0.6×	32	433

Countries citing papers authored by Mark E. Drotar

Since Specialization

Citations

This map shows the geographic impact of Mark E. Drotar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark E. Drotar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark E. Drotar more than expected).

Fields of papers citing papers by Mark E. Drotar

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark E. Drotar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark E. Drotar. The network helps show where Mark E. Drotar may publish in the future.

Co-authorship network of co-authors of Mark E. Drotar

This figure shows the co-authorship network connecting the top 25 collaborators of Mark E. Drotar. A scholar is included among the top collaborators of Mark E. Drotar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark E. Drotar. Mark E. Drotar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Scott, Mary T., Wei Liu, Rebecca Mitchell, et al.. (2024). Activating p53 abolishes self-renewal of quiescent leukaemic stem cells in residual CML disease. Nature Communications. 15(1). 651–651. 7 indexed citations

Scott, Mary T., Wei Liu, Rebecca Mitchell, et al.. (2021). Uncoupling p53 from an Embryonic Regulome Exhausts Quiescent CML Stem Cells through Inhibition of a HIF1alpha Molecular Program. Blood. 138(Supplement 1). 1541–1541. 1 indexed citations

Al‐Sheikh, Yazeed A., Mark E. Drotar, Shelagh Boyle, et al.. (2018). Lymphomas driven by Epstein–Barr virus nuclear antigen-1 (EBNA1) are dependant upon Mdm2. Oncogene. 37(29). 3998–4012. 28 indexed citations

Abraham, Sheela A., Lisa Hopcroft, Emma Carrick, et al.. (2016). Dual targeting of p53 and c-MYC selectively eliminates leukaemic stem cells. Nature. 534(7607). 341–346. 165 indexed citations

Sinclair, Amy, Mansi Shah, Mark E. Drotar, et al.. (2016). CXCR2 and CXCL4 regulate survival and self-renewal of hematopoietic stem/progenitor cells. Blood. 128(3). 371–383. 53 indexed citations

Singh, Taranjit, David M. Nelson, Dina Dikovskaya, et al.. (2014). HIRA orchestrates a non-canonical dynamic chromatin landscape in senescence and is required for suppression of neoplasia.

Cruickshanks, Hazel A., Tony McBryan, David M. Nelson, et al.. (2013). Senescent cells harbour features of the cancer epigenome. Nature Cell Biology. 15(12). 1495–1506. 251 indexed citations

Pawlikowski, Jeff S., Tony McBryan, John van Tuyn, et al.. (2013). Wnt signaling potentiates nevogenesis. Proceedings of the National Academy of Sciences. 110(40). 16009–16014. 47 indexed citations

Tsimbouri, Penelope M., Yazeed A. Al‐Sheikh, Mark E. Drotar, William Cushley, & Joanna B. Wilson. (2008). Epstein–Barr virus nuclear antigen-1 renders lymphocytes responsive to IL-2 but not IL-15 for survival. Journal of General Virology. 89(11). 2821–2832. 7 indexed citations

10.

Drotar, Mark E., et al.. (2003). Epstein‐Barr virus nuclear antigen‐1 and Myc cooperate in lymphomagenesis. International Journal of Cancer. 106(3). 388–395. 26 indexed citations

11.

Wilson, Joanna B. & Mark E. Drotar. (2003). Considerations in Generating Transgenic Mice: DNA, RNA, and Protein Extractions from Tissues– Rapid and Effective Blotting. Humana Press eBooks. 174. 361–377. 5 indexed citations

12.

Tsimbouri, Penelope M., et al.. (2002). bcl-xL and RAG genes are induced and the response to IL-2 enhanced in EμEBNA-1 transgenic mouse lymphocytes. Oncogene. 21(33). 5182–5187. 48 indexed citations

13.

Zhong, Wuning, Peter Burke, Mark E. Drotar, S.R. Chavali, & R. Armour Forse. (1995). Effects of prostaglandin E2, cholera toxin and 8-bromo-cyclic AMP on lipopolysaccharide-induced gene expression of cytokines in human macrophages.. PubMed. 84(3). 446–52. 89 indexed citations

14.

Burke, Peter, Mark E. Drotar, Minkui Luo, Michael B. Yaffe, & R. Armour Forse. (1994). Rapid modulation of liver-specific transcription factors after injury.. PubMed. 116(2). 285–92; discussion 292. 25 indexed citations

15.

Zhong, Wuning, et al.. (1993). EFFECTS OF PROSTAGLANDIN E2 AND CHOLERA TOXIN ON LIPOPOLYSACCHARIDE INDUCED GENE EXPRESSION OF CYTOKINES IN HUMAN MACROPHAGES. Critical Care Medicine. 21(Supplement). S279–S279. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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