Marion Vogel

746 total citations
10 papers, 597 citations indexed

About

Marion Vogel is a scholar working on Molecular Biology, Cancer Research and Pathology and Forensic Medicine. According to data from OpenAlex, Marion Vogel has authored 10 papers receiving a total of 597 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Cancer Research and 2 papers in Pathology and Forensic Medicine. Recurrent topics in Marion Vogel's work include FOXO transcription factor regulation (4 papers), Cancer-related molecular mechanisms research (3 papers) and Lymphoma Diagnosis and Treatment (2 papers). Marion Vogel is often cited by papers focused on FOXO transcription factor regulation (4 papers), Cancer-related molecular mechanisms research (3 papers) and Lymphoma Diagnosis and Treatment (2 papers). Marion Vogel collaborates with scholars based in Germany, United States and China. Marion Vogel's co-authors include Carolin Mogler, Dorothee Terhardt, Junhao Hu, Matthias Wieland, Kshitij Srivastava, Claudia Korn, Anja Runge, Liji Cao, Markus Thomas and Eva Besemfelder and has published in prestigious journals such as Science, Blood and Biochemistry.

In The Last Decade

Marion Vogel

10 papers receiving 591 citations

Peers

Marion Vogel

MB

HEPAT

CR

SURGE

ONCOL

Tianliang Sun Germany

Guangcun Huang China

T Kano Japan

Marta I. Gallego Spain

Shuangyin Han China

Vivek Shukla United States

Doris Schneller Austria

Rafał Pawłowski United Kingdom

Monica Aasrum Norway

Seiro Satohisa Japan

Tianliang Sun Germany

Marion Vogel

223 ×0.6

MB

98 ×0.8

HEPAT

35 ×0.3

CR

85 ×0.8

SURGE

49 ×0.6

ONCOL

Citations per year, relative to Marion Vogel Marion Vogel (= 1×) peers Tianliang Sun

Countries citing papers authored by Marion Vogel

Since Specialization

Citations

This map shows the geographic impact of Marion Vogel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marion Vogel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marion Vogel more than expected).

Fields of papers citing papers by Marion Vogel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marion Vogel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marion Vogel. The network helps show where Marion Vogel may publish in the future.

Co-authorship network of co-authors of Marion Vogel

This figure shows the co-authorship network connecting the top 25 collaborators of Marion Vogel. A scholar is included among the top collaborators of Marion Vogel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marion Vogel. Marion Vogel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Xie, Linka, Hanfeng Guan, Franziska Herrmann, et al.. (2018). Fine-tuning of FOXO3A in cHL as a survival mechanism and a hallmark of abortive plasma cell differentiation. Blood. 131(14). 1556–1567. 16 indexed citations

2.

Xie, Linka, Olga Ritz, Frank Leithäuser, et al.. (2014). FOXO1 downregulation contributes to the oncogenic program of primary mediastinal B-cell lymphoma. Oncotarget. 5(14). 5392–5402. 13 indexed citations

3.

Hu, Junhao, Kshitij Srivastava, Matthias Wieland, et al.. (2014). Endothelial Cell-Derived Angiopoietin-2 Controls Liver Regeneration as a Spatiotemporal Rheostat. Science. 343(6169). 416–419. 234 indexed citations

4.

Vogel, Marion, Linka Xie, Hanfeng Guan, et al.. (2014). FOXO1 repression contributes to block of plasma cell differentiation in classical Hodgkin lymphoma. Blood. 124(20). 3118–3129. 32 indexed citations

5.

Xie, Linka, Alexey Ushmorov, Frank Leithäuser, et al.. (2012). FOXO1 is a tumor suppressor in classical Hodgkin lymphoma. Blood. 119(15). 3503–3511. 141 indexed citations

6.

Zhang, Yi, Géraldine Sicot, Xiaohui Cui, et al.. (2011). Targeting a DNA Binding Motif of the EVI1 Protein by a Pyrrole–Imidazole Polyamide. Biochemistry. 50(48). 10431–10441. 27 indexed citations

7.

Kohn, Matthias, H. Hameister, Marion Vogel, & Hildegard Kehrer‐Sawatzki. (2003). Expression pattern of the Rsk2, Rsk4 and Pdk1 genes during murine embryogenesis. Gene Expression Patterns. 3(2). 173–177. 21 indexed citations

8.

Stempfl, Thomas, Marion Vogel, Corinna Wülbeck, et al.. (2002). Identification of Circadian-Clock-Regulated Enhancers and Genes of Drosophila melanogaster by Transposon Mobilization and Luciferase Reporting of Cyclical Gene Expression. Genetics. 160(2). 571–593. 33 indexed citations

9.

Maier, Dieter, Carol L. Farr, Burkhard Poeck, et al.. (2001). Mitochondrial Single-stranded DNA-binding Protein Is Required for Mitochondrial DNA Replication and Development inDrosophila melanogaster. Molecular Biology of the Cell. 12(4). 821–830. 65 indexed citations

10.

Ricard, Cynthia S., John W. Verbsky, M. Alejandro Barbieri, et al.. (2001). Drosophila rab GDI mutants disrupt development but have normal rab membrane extraction. genesis. 31(1). 17–29. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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