Mario Pávez

858 total citations · 1 hit paper
18 papers, 640 citations indexed

About

Mario Pávez is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Mario Pávez has authored 18 papers receiving a total of 640 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 4 papers in Endocrinology, Diabetes and Metabolism and 3 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Mario Pávez's work include Mitochondrial Function and Pathology (6 papers), Metabolism, Diabetes, and Cancer (4 papers) and Hormonal and reproductive studies (3 papers). Mario Pávez is often cited by papers focused on Mitochondrial Function and Pathology (6 papers), Metabolism, Diabetes, and Cancer (4 papers) and Hormonal and reproductive studies (3 papers). Mario Pávez collaborates with scholars based in Chile, Netherlands and United States. Mario Pávez's co-authors include Dirk J. van Veldhuisen, B. Daan Westenbrink, Herman H.W. Silljé, Rudolf A. de Boer, Silke U. Oberdorf‐Maass, Salva R. Yurista, Jan‐Luuk Hillebrands, Harry van Goor, Sergio Lavandero and Francisco Westermeier and has published in prestigious journals such as Journal of Clinical Investigation, PLoS ONE and The FASEB Journal.

In The Last Decade

Mario Pávez

18 papers receiving 633 citations

Hit Papers

Sodium–Glucose Co-Transporter 2 Inhibition with Empaglifl... 2019 2026 2021 2023 2019 50 100 150 200

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mario Pávez Chile 12 273 256 255 141 96 18 640
Gabriela Placoná Diniz Brazil 18 408 1.5× 258 1.0× 225 0.9× 107 0.8× 145 1.5× 41 801
Asif Anwar United States 13 277 1.0× 173 0.7× 200 0.8× 76 0.5× 180 1.9× 22 721
K. Bove Denmark 15 171 0.6× 220 0.9× 71 0.3× 112 0.8× 108 1.1× 25 569
Yoko Eto Japan 10 248 0.9× 281 1.1× 101 0.4× 67 0.5× 61 0.6× 20 557
Xiaojing Wang China 14 251 0.9× 82 0.3× 110 0.4× 90 0.6× 61 0.6× 34 587
Ming Gao China 13 121 0.4× 132 0.5× 82 0.3× 70 0.5× 104 1.1× 60 498
Fernándo López-Barrera Mexico 12 204 0.7× 110 0.4× 193 0.8× 72 0.5× 70 0.7× 16 677
Yoshio Tsuruya Japan 14 281 1.0× 351 1.4× 106 0.4× 123 0.9× 83 0.9× 32 754
Hai‐qin Tang China 12 121 0.4× 128 0.5× 132 0.5× 81 0.6× 56 0.6× 26 468
Zhijian Yang China 14 244 0.9× 147 0.6× 84 0.3× 109 0.8× 36 0.4× 40 571

Countries citing papers authored by Mario Pávez

Since Specialization
Citations

This map shows the geographic impact of Mario Pávez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mario Pávez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mario Pávez more than expected).

Fields of papers citing papers by Mario Pávez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mario Pávez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mario Pávez. The network helps show where Mario Pávez may publish in the future.

Co-authorship network of co-authors of Mario Pávez

This figure shows the co-authorship network connecting the top 25 collaborators of Mario Pávez. A scholar is included among the top collaborators of Mario Pávez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mario Pávez. Mario Pávez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Dobner, Jochen, Mario Pávez, Lukas Cyganek, et al.. (2024). mtDNA analysis using Mitopore. Molecular Therapy — Methods & Clinical Development. 32(2). 101231–101231. 2 indexed citations
2.
Pávez, Mario & Lukas Cyganek. (2022). Recent Advances in Modeling Mitochondrial Cardiomyopathy Using Human Induced Pluripotent Stem Cells. Frontiers in Cell and Developmental Biology. 9. 800529–800529. 7 indexed citations
3.
Bömer, Nils, Mario Pávez, Niels Grote Beverborg, et al.. (2022). Micronutrient deficiencies in heart failure: Mitochondrial dysfunction as a common pathophysiological mechanism?. Journal of Internal Medicine. 291(6). 713–731. 40 indexed citations
4.
Vermeer, M., Maria C. Bolling, Jacqueline M. Bliley, et al.. (2021). Gain-of-function mutation in ubiquitin ligase KLHL24 causes desmin degradation and dilatation in hiPSC-derived engineered heart tissues. Journal of Clinical Investigation. 131(17). 21 indexed citations
5.
Pávez, Mario, Pablo Sánchez-Aguilera, Nils Bömer, et al.. (2021). ATPase Inhibitory Factor-1 Disrupts Mitochondrial Ca2+ Handling and Promotes Pathological Cardiac Hypertrophy through CaMKIIδ. International Journal of Molecular Sciences. 22(9). 4427–4427. 10 indexed citations
6.
Bömer, Nils, Mario Pávez, Annet N. Linders, et al.. (2021). Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes. International Journal of Molecular Sciences. 22(21). 11906–11906. 18 indexed citations
7.
Meems, Laura M.G., Willem P.T. Ruifrok, Alexander H. Maass, et al.. (2021). The erythropoietin receptor expressed in skeletal muscle is essential for mitochondrial biogenesis and physiological exercise. Pflügers Archiv - European Journal of Physiology. 473(8). 1301–1313. 11 indexed citations
8.
Troncoso, Mayarling F., Mario Pávez, Javier Nebreda Durán, et al.. (2021). Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy. Biological Research. 54(1). 3–3. 21 indexed citations
9.
Bömer, Nils, Mario Pávez, Annet N. Linders, et al.. (2021). <i>DIO2</i> Is a Regulator of Mitochondrial Function, Morphology and UPR <sup>mt</sup> in Human Cardiomyocytes. SSRN Electronic Journal. 1 indexed citations
10.
Yurista, Salva R., Herman H.W. Silljé, Silke U. Oberdorf‐Maass, et al.. (2019). Sodium–Glucose Co-Transporter 2 Inhibition with Empagliflozin Improves Cardiac Function in Non-Diabetic Rats with Left Ventricular Dysfunction After Myocardial Infarction. European Journal of Heart Failure. 21(7). 862–873. 248 indexed citations breakdown →
11.
Durán, Javier Nebreda, Mario Pávez, Mayarling F. Troncoso, et al.. (2017). Ca2+/Calmodulin-Dependent Protein Kinase II and Androgen Signaling Pathways Modulate MEF2 Activity in Testosterone-Induced Cardiac Myocyte Hypertrophy. Frontiers in Pharmacology. 8. 604–604. 25 indexed citations
12.
Westermeier, Francisco, Jaime A. Riquelme, Mario Pávez, et al.. (2016). New Molecular Insights of Insulin in Diabetic Cardiomyopathy. Frontiers in Physiology. 7. 125–125. 91 indexed citations
13.
Durán, Javier Nebreda, César Oyarce, Mario Pávez, et al.. (2016). GSK-3β/NFAT Signaling Is Involved in Testosterone-Induced Cardiac Myocyte Hypertrophy. PLoS ONE. 11(12). e0168255–e0168255. 36 indexed citations
14.
Pávez, Mario, et al.. (2016). Ancho de distribución eritrocitaria como potencial biomarcador clínico en enfermedades cardiovasculares. Revista médica de Chile. 144(5). 634–642. 16 indexed citations
15.
Westermeier, Francisco, Mario Bustamante, Mario Pávez, et al.. (2015). Novel players in cardioprotection: Insulin like growth factor-1, angiotensin-(1–7) and angiotensin-(1–9). Pharmacological Research. 101. 41–55. 22 indexed citations
16.
Wilson, Carlos, Ariel Contreras‐Ferrat, Cesár Osorio‐Fuentealba, et al.. (2013). Testosterone increases GLUT4‐dependent glucose uptake in cardiomyocytes. Journal of Cellular Physiology. 228(12). 2399–2407. 45 indexed citations
17.
Valladares-Ide, Denisse, Ariel Contreras‐Ferrat, Mario Pávez, et al.. (2013). Electrical Stimuli Are Anti-Apoptotic in Skeletal Muscle via Extracellular ATP. Alteration of This Signal in Mdx Mice Is a Likely Cause of Dystrophy. PLoS ONE. 8(11). e75340–e75340. 24 indexed citations
18.
Valladares-Ide, Denisse, et al.. (2012). ATP release is altered in a mouse model for Duchenne muscular dystrophy and signals for proteins that promote cell death. The FASEB Journal. 26(S1). 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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