Marianne Mollenauer
- Immunology top 5%
- Molecular Biology
- Oncology top 10%
- Cancer Research top 10%
- Cell Biology
- Co-authors
- Arthur WeissJeroen P. RooseVirginia Smith ShapiroLarry KaneVikas GuptaJames C. StoneD QianTomohiro Kurosaki
- Topics
- T-cell and B-cell Immunology (9 papers)Immune Cell Function and Interaction (7 papers)NF-κB Signaling Pathways (5 papers)
- Cited by
- ImmunologyOncologyCancer Research
- Partner nations
- United StatesCanadaGermany
In The Last Decade
Marianne Mollenauer
19 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 84
- Immunology 790
- Molecular Biology 607
- Oncology 389
- Cancer Research 214
- Cell Biology 100
Countries citing papers authored by Marianne Mollenauer
This map shows the geographic impact of Marianne Mollenauer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marianne Mollenauer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marianne Mollenauer more than expected).
Fields of papers citing papers by Marianne Mollenauer
This network shows the impact of papers produced by Marianne Mollenauer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marianne Mollenauer. The network helps show where Marianne Mollenauer may publish in the future.
Co-authorship network of co-authors of Marianne Mollenauer
This figure shows the co-authorship network connecting the top 25 collaborators of Marianne Mollenauer. A scholar is included among the top collaborators of Marianne Mollenauer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marianne Mollenauer. Marianne Mollenauer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 99 | |
| 2 | 88 | |
| 3 | 45 | |
| 4 | 72 | |
| 5 | 58 | |
| 6 | 46 | |
| 7 | 0 | |
| 8 | 38 | |
| 9 | 1 | |
| 10 | 135 | |
| 11 | 169 | |
| 12 | 26 | |
| 13 | 73 | |
| 14 | 92 | |
| 15 | 132 | |
| 16 | 46 | |
| 17 | 30 | |
| 18 | Nuclear factor of activated T cells and AP-1 are insufficient for IL-2 promoter activation: requirement for CD28 up-regulation of RE/AP. | 37 |
| 19 | 94 | |
| 20 | 74 |
About Marianne Mollenauer
Marianne Mollenauer is a scholar working on Immunology, Cancer Research and Immunology and Allergy, having authored 20 papers that have together received 1.4k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (9 papers), Immune Cell Function and Interaction (7 papers) and NF-κB Signaling Pathways (5 papers). The work is most often cited by research in Immunology (790 citations), Oncology (389 citations) and Cancer Research (214 citations). Marianne Mollenauer has collaborated with scholars based in United States, Canada and Germany. Frequent co-authors include Arthur Weiss, Jeroen P. Roose, Virginia Smith Shapiro, Larry Kane, Vikas Gupta, James C. Stone, D Qian, Tomohiro Kurosaki, Mary Ho and Xu Zheng. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.