Maria Ribadeneira

1.1k citations
27 papers · 839 indexed · h-index 14

Maria Ribadeneira

26 papers receiving 810 citations

Peers

Maria Ribadeneira
Comparison fields: 5 of 85
  • Pharmacology 232
  • Biological Psychiatry 40
  • Behavioral Neuroscience 49
  • Genetics 82
  • Hematology 86
Replace Anna Barańczyk‐Kuźma with:
Anna Barańczyk‐Kuźma Poland
Phong Nguyen United States
Toshiya Ogorochi Japan
Iris Grossman Israel
Parkyong Song South Korea
Toshie Kambe Japan
Denise L. Walker United States
Elisabeth Greiner United States
Naveen Kumar Singhal United States
Sowmya Chollate United States
Maria Ribadeneira relative to Anna Barańczyk‐Kuźma Poland Anna Barańczyk‐Kuźma's profile →
Citations per field
00.5×10.8×
Anna Barańczyk‐Kuźma · 1×
Citations per year

Countries citing papers authored by Maria Ribadeneira

Since Specialization
Citations

This map shows the geographic impact of Maria Ribadeneira's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maria Ribadeneira with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maria Ribadeneira more than expected).

Fields of papers citing papers by Maria Ribadeneira

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maria Ribadeneira. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maria Ribadeneira. The network helps show where Maria Ribadeneira may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Maria Ribadeneira, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Maria Ribadeneira Line = papers co-authored together Maria Ribadeneira links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20247
2 20241
3 202228
4 202223
5 202014
6 202042
7 201922
8 20198
9
KCNQ channel openers reverse depressive symptoms via an active resilience mechanism
201635
10 2016118
11 20152
12 20131
13 20137
14 20124
15 201110
16 200713
17 200427
18 200326
19 2003276
20 199629

About Maria Ribadeneira

Maria Ribadeneira is a scholar working on Genetics, Pharmacology and Hematology, having authored 27 papers that have together received 839 indexed citations. Recurring topics across this work include Hemoglobinopathies and Related Disorders (7 papers), Pharmacogenetics and Drug Metabolism (6 papers), Drug Transport and Resistance Mechanisms (5 papers), Erythrocyte Function and Pathophysiology (5 papers), Neuroscience and Neuropharmacology Research (3 papers), Pharmacological Effects and Toxicity Studies (3 papers), Receptor Mechanisms and Signaling (3 papers) and Acute Myeloid Leukemia Research (2 papers). The work is most often cited by research in Pharmacology (232 citations), Biological Psychiatry (40 citations) and Behavioral Neuroscience (49 citations). Maria Ribadeneira has collaborated with scholars based in United States, Canada and Germany. Frequent co-authors include Yu Li, Jameson Forster, Ajay Madan, Patrick Koch, Maciej Czerwiński, Liang‐Shang Gan, Kathleen Carroll, Karl Zech, Philmore Robertson and Lida Antonian. Their work appears in journals such as Nature, Nature Communications and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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