Maria Reich

1.2k total citations
22 papers, 510 citations indexed

About

Maria Reich is a scholar working on Oncology, Hepatology and Surgery. According to data from OpenAlex, Maria Reich has authored 22 papers receiving a total of 510 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Oncology, 12 papers in Hepatology and 11 papers in Surgery. Recurrent topics in Maria Reich's work include Drug Transport and Resistance Mechanisms (16 papers), Pediatric Hepatobiliary Diseases and Treatments (7 papers) and Cholangiocarcinoma and Gallbladder Cancer Studies (5 papers). Maria Reich is often cited by papers focused on Drug Transport and Resistance Mechanisms (16 papers), Pediatric Hepatobiliary Diseases and Treatments (7 papers) and Cholangiocarcinoma and Gallbladder Cancer Studies (5 papers). Maria Reich collaborates with scholars based in Germany, Switzerland and United States. Maria Reich's co-authors include Verena Keitel, Dieter Häussinger, Caroline Klindt, Kathleen Deutschmann, Lina Spomer, Diran Herebıan, Ertan Mayatepek, Stefanie Kluge, Annika Sommerfeld and Carola Dröge and has published in prestigious journals such as Hepatology, Gut and Journal of the American Society of Nephrology.

In The Last Decade

Maria Reich

20 papers receiving 504 citations

Peers

Maria Reich

ONCOL

SURGE

HEPAT

EPIDE

Caroline Klindt Germany

Juliet Venter United States

Atsuhiro Morita Japan

Alessandro Gigliozzi Italy

G. Alpini United States

Jing‐Yu Gong China

Jennifer Demieville United States

Davor Slijepcevic Netherlands

A Beilfuss Germany

Yosuke Aihara Japan

Caroline Klindt Germany

Maria Reich

222 ×0.9

ONCOL

194 ×0.9

SURGE

132 ×0.7

HEPAT

140 ×0.8

EPIDE

111 ×0.9

Citations per year, relative to Maria Reich Maria Reich (= 1×) peers Caroline Klindt

Countries citing papers authored by Maria Reich

Since Specialization

Citations

This map shows the geographic impact of Maria Reich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maria Reich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maria Reich more than expected).

Fields of papers citing papers by Maria Reich

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maria Reich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maria Reich. The network helps show where Maria Reich may publish in the future.

Co-authorship network of co-authors of Maria Reich

This figure shows the co-authorship network connecting the top 25 collaborators of Maria Reich. A scholar is included among the top collaborators of Maria Reich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maria Reich. Maria Reich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Sachs, Ulrich J., et al.. (2025). Platelet autoantibodies have an impact on the platelet count in patients. Journal of Thrombosis and Haemostasis. 23(7). 2322–2326. 1 indexed citations

Reich, Maria, Tobias Franz, Haifeng Xu, et al.. (2025). Bile acid receptor Tgr5 prevents macrophage hyperinflammation during bacterial sepsis through metabolic and epigenetic silencing. iScience. 28(12). 113929–113929.

Reich, Maria, et al.. (2024). The bile acid receptor TGR5 inhibits platelet activation and thrombus formation. Platelets. 35(1). 2322733–2322733. 6 indexed citations

Stein, Stephanie, T Poch, Antonella Carambia, et al.. (2020). IL-17A/F enable cholangiocytes to restrict T cell-driven experimental cholangitis by upregulating PD-L1 expression. Journal of Hepatology. 74(4). 919–930. 26 indexed citations

Klindt, Caroline, Maria Reich, Jan Stindt, et al.. (2020). The G protein coupled bile acid receptor TGR5 (Gpbar1) modulates endothelin-1 signaling in liver. Zeitschrift für Gastroenterologie. 2 indexed citations

Klindt, Caroline, Maria Reich, Birte Hellwig, et al.. (2019). The G Protein-Coupled Bile Acid Receptor TGR5 (Gpbar1) Modulates Endothelin-1 Signaling in Liver. Cells. 8(11). 1467–1467. 42 indexed citations

Deutschmann, Kathleen, Maria Reich, Alexander Lang, et al.. (2019). Role of the bile acid receptor TGR5 (GPBAR1) in cholangiocarcinoma (CCA). Zeitschrift für Gastroenterologie.

Behnke, Kristina, Yuan Zhuang, Haifeng C. Xu, et al.. (2018). B Cell‐Mediated Maintenance of Cluster of Differentiation 169–Positive Cells Is Critical for Liver Regeneration. Hepatology. 68(6). 2348–2361. 19 indexed citations

Deutschmann, Kathleen, Maria Reich, Caroline Klindt, et al.. (2017). Bile acid receptors in the biliary tree: TGR5 in physiology and disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864(4). 1319–1325. 101 indexed citations

10.

Reich, Maria, Caroline Klindt, Kathleen Deutschmann, et al.. (2017). Role of the G Protein-Coupled Bile Acid Receptor TGR5 in Liver Damage. Digestive Diseases. 35(3). 235–240. 34 indexed citations

11.

Gowert, Nina Sarah, Maria Reich, L Donner, et al.. (2017). Defective Platelet Activation and Bleeding Complications upon Cholestasis in Mice. Cellular Physiology and Biochemistry. 41(6). 2133–2149. 16 indexed citations

12.

Klindt, Caroline, Kathleen Deutschmann, Maria Reich, Dieter Häussinger, & Verena Keitel. (2017). TGR5 plays a protective role against portal hypertension in LCA induced liver damage in mice. Journal of Hepatology. 66(1). S142–S142. 1 indexed citations

13.

Reich, Maria, Kathleen Deutschmann, Annika Sommerfeld, et al.. (2015). TGR5 is essential for bile acid-dependent cholangiocyte proliferation in vivo and in vitro. Gut. 65(3). 487–501. 156 indexed citations

14.

Sorg, Ursula R., Kristina Behnke, Daniel Degrandi, et al.. (2015). Cooperative role of lymphotoxin β receptor and tumor necrosis factor receptor p55 in murine liver regeneration. Journal of Hepatology. 64(5). 1108–1117. 9 indexed citations

15.

Klindt, Caroline, Kathleen Deutschmann, Maria Reich, et al.. (2015). TGR5 knockout mice are highly susceptible to LCA induced liver damage. Zeitschrift für Gastroenterologie. 53(12). 1 indexed citations

16.

Deutschmann, Kathleen, et al.. (2015). Role of the bile acid receptor TGR5 (Gpbar-1) in gastrointestinal tumors. Zeitschrift für Gastroenterologie. 53(12). 1 indexed citations

17.

Keitel, Verena, Maria Reich, & Dieter Häussinger. (2014). TGR5: Pathogenetic Role and/or Therapeutic Target in Fibrosing Cholangitis?. Clinical Reviews in Allergy & Immunology. 48(2-3). 218–225. 39 indexed citations

18.

Keitel, Verena, Maria Reich, Annika Sommerfeld, et al.. (2014). Role of the bile acid receptor TGR5 (Gpbar-1) in liver damage and regeneration. European journal of medical research. 19(S1). 2 indexed citations

19.

Keitel, Verena, Maria Reich, Annika Sommerfeld, et al.. (2013). 1427 THE BILE ACID RECEPTOR TGR5 PROMOTES CHOLANGIOCYTE PROLIFERATION THROUGH A cSRC–EGFR–ERK SIGNALLING PATHWAY. Journal of Hepatology. 58. S574–S575. 3 indexed citations

20.

Fischer, M, et al.. (1977). [Acute stomach lesions and changes in histamine level of various organs in immobilization stress].. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 217–20. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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