Marek Wanior

540 total citations
8 papers, 289 citations indexed

About

Marek Wanior is a scholar working on Molecular Biology, Hematology and Organic Chemistry. According to data from OpenAlex, Marek Wanior has authored 8 papers receiving a total of 289 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Hematology and 1 paper in Organic Chemistry. Recurrent topics in Marek Wanior's work include Protein Degradation and Inhibitors (6 papers), Multiple Myeloma Research and Treatments (3 papers) and Ubiquitin and proteasome pathways (2 papers). Marek Wanior is often cited by papers focused on Protein Degradation and Inhibitors (6 papers), Multiple Myeloma Research and Treatments (3 papers) and Ubiquitin and proteasome pathways (2 papers). Marek Wanior collaborates with scholars based in Germany and Canada. Marek Wanior's co-authors include Stefan Knapp, Andreas Krämer, Andreas C. Joerger, Simone Fulda, Julius C. Enßle, Bernhard Küster, Stephanie Heinzlmeir, Julia Hofstetter, Andreas Schlösser and Christoph Sotriffer and has published in prestigious journals such as Oncogene, Journal of Medicinal Chemistry and Nature Chemical Biology.

In The Last Decade

Marek Wanior

8 papers receiving 287 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Marek Wanior Germany	7	251	86	47	39	31	8	289
Tatiana Shorstova Canada	4	393 1.6×	88 1.0×	65 1.4×	48 1.2×	27 0.9×	5	435
Matthias Reule United Kingdom	6	167 0.7×	103 1.2×	42 0.9×	22 0.6×	20 0.6×	11	273
Nandini Sakurikar United States	8	236 0.9×	136 1.6×	44 0.9×	29 0.7×	19 0.6×	10	337
Bo Kyung A. Seong United States	7	259 1.0×	89 1.0×	17 0.4×	14 0.4×	41 1.3×	7	331
Gary Simmons United States	9	144 0.6×	169 2.0×	82 1.7×	37 0.9×	63 2.0×	22	291
Troy M. Robinson United States	4	159 0.6×	112 1.3×	34 0.7×	23 0.6×	78 2.5×	6	255
Marty J. Heslin United States	5	245 1.0×	104 1.2×	37 0.8×	16 0.4×	20 0.6×	7	329
Charles Sawyers United States	5	148 0.6×	68 0.8×	93 2.0×	20 0.5×	26 0.8×	7	253
Melanie Dujka United States	7	223 0.9×	122 1.4×	48 1.0×	21 0.5×	44 1.4×	12	344
Jiro Minami Japan	9	144 0.6×	68 0.8×	103 2.2×	46 1.2×	11 0.4×	21	266

Countries citing papers authored by Marek Wanior

Since Specialization

Citations

This map shows the geographic impact of Marek Wanior's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marek Wanior with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marek Wanior more than expected).

Fields of papers citing papers by Marek Wanior

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marek Wanior. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marek Wanior. The network helps show where Marek Wanior may publish in the future.

Co-authorship network of co-authors of Marek Wanior

This figure shows the co-authorship network connecting the top 25 collaborators of Marek Wanior. A scholar is included among the top collaborators of Marek Wanior based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marek Wanior. Marek Wanior is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Schröder, Martin, Ulrich Grädler, Juliane Braun, et al.. (2022). MSC-1186, a Highly Selective Pan-SRPK Inhibitor Based on an Exceptionally Decorated Benzimidazole-Pyrimidine Core. Journal of Medicinal Chemistry. 66(1). 837–854. 6 indexed citations

2.

Meister, Michael T., et al.. (2021). Selective BH3 mimetics synergize with BET inhibition to induce mitochondrial apoptosis in rhabdomyosarcoma cells. Neoplasia. 24(2). 109–119. 5 indexed citations

3.

Wanior, Marek, Andreas Krämer, Stefan Knapp, & Andreas C. Joerger. (2021). Exploiting vulnerabilities of SWI/SNF chromatin remodelling complexes for cancer therapy. Oncogene. 40(21). 3637–3654. 89 indexed citations

4.

Hussong, Michelle, Christina Grimm, Michael T. Meister, et al.. (2020). Co-inhibition of BET proteins and PI3Kα triggers mitochondrial apoptosis in rhabdomyosarcoma cells. Oncogene. 39(19). 3837–3852. 16 indexed citations

5.

Adhikari, Bikash, Julia Hofstetter, Martin Schröder, et al.. (2020). PROTAC-mediated degradation reveals a non-catalytic function of AURORA-A kinase. Nature Chemical Biology. 16(11). 1179–1188. 111 indexed citations

6.

Hanke, Thomas, Jan Heering, Xiaomin Ni, et al.. (2020). A Selective Modulator of Peroxisome Proliferator-Activated Receptor γ with an Unprecedented Binding Mode. Journal of Medicinal Chemistry. 63(9). 4555–4561. 7 indexed citations

7.

Wanior, Marek, Xiaomin Ni, Andreas Krämer, et al.. (2020). Pan-SMARCA/PB1 Bromodomain Inhibitors and Their Role in Regulating Adipogenesis. Journal of Medicinal Chemistry. 63(23). 14680–14699. 21 indexed citations

8.

Enßle, Julius C., et al.. (2018). Co-targeting of BET proteins and HDACs as a novel approach to trigger apoptosis in rhabdomyosarcoma cells. Cancer Letters. 428. 160–172. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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