Marcela Guzman-Ayala

945 total citations
10 papers, 722 citations indexed

About

Marcela Guzman-Ayala is a scholar working on Molecular Biology, Genetics and Surgery. According to data from OpenAlex, Marcela Guzman-Ayala has authored 10 papers receiving a total of 722 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 2 papers in Genetics and 1 paper in Surgery. Recurrent topics in Marcela Guzman-Ayala's work include Pluripotent Stem Cells Research (5 papers), Renal and related cancers (5 papers) and CRISPR and Genetic Engineering (2 papers). Marcela Guzman-Ayala is often cited by papers focused on Pluripotent Stem Cells Research (5 papers), Renal and related cancers (5 papers) and CRISPR and Genetic Engineering (2 papers). Marcela Guzman-Ayala collaborates with scholars based in United Kingdom, United States and Switzerland. Marcela Guzman-Ayala's co-authors include Daniel B. Constam, Daniel Mesnard, Nadav Ben-Haim, L. Pescatore, Félix Naef, Cindy Lu, Mirko Bischofberger, Elizabeth J. Robertson, Miguel Ramalho‐Santos and Michael Sachs and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Blood.

In The Last Decade

Marcela Guzman-Ayala

10 papers receiving 715 citations

Peers

Marcela Guzman-Ayala
Tiago Faial United States
Shawna Tan Singapore
Gloryn Chia United States
G. Cauffman Belgium
Ivan Bedzhov Germany
M.J. Abeyta United States
Guo Qing Tong Singapore
Tiago Faial United States
Marcela Guzman-Ayala
Citations per year, relative to Marcela Guzman-Ayala Marcela Guzman-Ayala (= 1×) peers Tiago Faial

Countries citing papers authored by Marcela Guzman-Ayala

Since Specialization
Citations

This map shows the geographic impact of Marcela Guzman-Ayala's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marcela Guzman-Ayala with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marcela Guzman-Ayala more than expected).

Fields of papers citing papers by Marcela Guzman-Ayala

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marcela Guzman-Ayala. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marcela Guzman-Ayala. The network helps show where Marcela Guzman-Ayala may publish in the future.

Co-authorship network of co-authors of Marcela Guzman-Ayala

This figure shows the co-authorship network connecting the top 25 collaborators of Marcela Guzman-Ayala. A scholar is included among the top collaborators of Marcela Guzman-Ayala based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marcela Guzman-Ayala. Marcela Guzman-Ayala is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Bulut-Karslıoğlu, Aydan, Hu Jin, Marcela Guzman-Ayala, et al.. (2021). Chd1 protects genome integrity at promoters to sustain hypertranscription in embryonic stem cells. Nature Communications. 12(1). 4859–4859. 11 indexed citations
2.
Garrison, Brian S., Han‐Xiang Deng, Gözde Yücel, et al.. (2021). FLT3 OR CD33 NOT EMCN Logic Gated CAR-NK Cell Therapy (SENTI-202) for Precise Targeting of AML. Blood. 138(Supplement 1). 2799–2799. 10 indexed citations
3.
Guzman-Ayala, Marcela, Michael Sachs, Courtney Onodera, et al.. (2014). Chd1 is essential for the high transcriptional output and rapid growth of the mouse epiblast. Development. 142(1). 118–127. 61 indexed citations
4.
Redshaw, Nicholas, Carme Camps, Vikas Sharma, et al.. (2013). TGF-β/Smad2/3 Signaling Directly Regulates Several miRNAs in Mouse ES Cells and Early Embryos. PLoS ONE. 8(1). e55186–e55186. 18 indexed citations
5.
Sachs, Michael, et al.. (2010). Parallel gateways to pluripotency: open chromatin in stem cells and development. Current Opinion in Genetics & Development. 20(5). 492–499. 11 indexed citations
6.
Guzman-Ayala, Marcela, Kian Leong Lee, Konstantinos J. Mavrakis, et al.. (2009). Graded Smad2/3 Activation Is Converted Directly into Levels of Target Gene Expression in Embryonic Stem Cells. PLoS ONE. 4(1). e4268–e4268. 36 indexed citations
7.
Guzman-Ayala, Marcela, Kian Leong Lee, Konstantinos J. Mavrakis, et al.. (2009). Correction: Graded Smad2/3 Activation Is Converted Directly into Levels of Target Gene Expression in Embryonic Stem Cells. PLoS ONE. 4(2). 2 indexed citations
8.
Ben-Haim, Nadav, Cindy Lu, Marcela Guzman-Ayala, et al.. (2006). The Nodal Precursor Acting via Activin Receptors Induces Mesoderm by Maintaining a Source of Its Convertases and BMP4. Developmental Cell. 11(3). 313–323. 250 indexed citations
9.
Mesnard, Daniel, Marcela Guzman-Ayala, & Daniel B. Constam. (2006). Nodal specifies embryonic visceral endoderm and sustains pluripotent cells in the epiblast before overt axial patterning. Development. 133(13). 2497–2505. 193 indexed citations
10.
Guzman-Ayala, Marcela, et al.. (2004). Nodal protein processing and fibroblast growth factor 4 synergize to maintain a trophoblast stem cell microenvironment. Proceedings of the National Academy of Sciences. 101(44). 15656–15660. 130 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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