Manuela Frese‐Schaper
- Molecular Biology
- Immunology
- Pulmonary and Respiratory Medicine
- Pathology and Forensic Medicine
- Oncology
- Co-authors
- Steffen FreseRalph A. SchmidAnne‐Catherine AndresBéatrice ZumkehrStephan ChristenPeter Brønnum NielsenBarbara FinckhPeter Gessler
- Topics
- Systemic Lupus Erythematosus Research (4 papers)Cell death mechanisms and regulation (3 papers)Cancer therapeutics and mechanisms (2 papers)
- Partner nations
- SwitzerlandGermanyJapan
In The Last Decade
Manuela Frese‐Schaper
10 papers receiving 331 citations
Peers
Comparison fields: 5 of 76
- Molecular Biology 145
- Immunology 68
- Pulmonary and Respiratory Medicine 43
- Pathology and Forensic Medicine 40
- Oncology 36
Countries citing papers authored by Manuela Frese‐Schaper
This map shows the geographic impact of Manuela Frese‐Schaper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Manuela Frese‐Schaper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Manuela Frese‐Schaper more than expected).
Fields of papers citing papers by Manuela Frese‐Schaper
This network shows the impact of papers produced by Manuela Frese‐Schaper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Manuela Frese‐Schaper. The network helps show where Manuela Frese‐Schaper may publish in the future.
Co-authorship network of co-authors of Manuela Frese‐Schaper
This figure shows the co-authorship network connecting the top 25 collaborators of Manuela Frese‐Schaper. A scholar is included among the top collaborators of Manuela Frese‐Schaper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Manuela Frese‐Schaper. Manuela Frese‐Schaper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 15 | |
| 4 | 11 | |
| 5 | 13 | |
| 6 | 27 | |
| 7 | 30 | |
| 8 | Cytotoxic effects of camptothecin and cisplatin combined with tumor necrosis factor-related apoptosis-inducing ligand (Apo2L/TRAIL) in a model of primary culture of non-small cell lung cancer. | 5 |
| 9 | 31 | |
| 10 | 124 | |
| 11 | 87 |
About Manuela Frese‐Schaper
Manuela Frese‐Schaper is a scholar working on Rheumatology, Immunology and Nephrology, having authored 11 papers that have together received 346 indexed citations. Recurring topics across this work include Systemic Lupus Erythematosus Research (4 papers), Cell death mechanisms and regulation (3 papers) and Cancer therapeutics and mechanisms (2 papers). The work is most often cited by research in Toxicology (13 citations), Immunology (68 citations) and Developmental Neuroscience (13 citations). Manuela Frese‐Schaper has collaborated with scholars based in Switzerland, Germany and Japan. Frequent co-authors include Steffen Frese, Ralph A. Schmid, Anne‐Catherine Andres, Béatrice Zumkehr, Stephan Christen, Peter Brønnum Nielsen, Barbara Finckh, Peter Gessler, Edith R. Schmid and Bernhard Schmitt. Their work appears in journals such as The Journal of Immunology, Cancer Research and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.