Manuela Antonioli

9.6k total citations · 1 hit paper
28 papers, 2.4k citations indexed

About

Manuela Antonioli is a scholar working on Epidemiology, Cell Biology and Molecular Biology. According to data from OpenAlex, Manuela Antonioli has authored 28 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Epidemiology, 12 papers in Cell Biology and 11 papers in Molecular Biology. Recurrent topics in Manuela Antonioli's work include Autophagy in Disease and Therapy (19 papers), Endoplasmic Reticulum Stress and Disease (10 papers) and Extracellular vesicles in disease (4 papers). Manuela Antonioli is often cited by papers focused on Autophagy in Disease and Therapy (19 papers), Endoplasmic Reticulum Stress and Disease (10 papers) and Extracellular vesicles in disease (4 papers). Manuela Antonioli collaborates with scholars based in Italy, Germany and United Kingdom. Manuela Antonioli's co-authors include Gian María Fimia, Mauro Piacentini, Francesca Nazio, Francesco Cecconi, Jörn Dengjel, Martina Di Rienzo, Christine Gretzmeier, Marco Corazzari, Flavie Strappazzon and Pamela Bielli and has published in prestigious journals such as The Journal of Cell Biology, Nature Cell Biology and Oncogene.

In The Last Decade

Manuela Antonioli

26 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

mTOR inhibits autophagy by controlling ULK1 ubiquitylation, self-association and function through AMBRA1 and TRAF6

2013 622 citations Francesca Nazio, Flavie Strappazzon et al. Nature Cell Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Manuela Antonioli Italy	17	1.5k	1.3k	568	260	245	28	2.4k
Francesca Nazio Italy	25	2.0k 1.4×	1.8k 1.3×	552 1.0×	227 0.9×	396 1.6×	38	3.1k
Flavie Strappazzon Italy	22	1.6k 1.1×	1.4k 1.1×	435 0.8×	144 0.6×	237 1.0×	35	2.4k
Valentina Cianfanelli Italy	16	1.2k 0.8×	1.1k 0.8×	341 0.6×	149 0.6×	262 1.1×	23	1.9k
Pauline Isakson Sweden	14	1.1k 0.8×	986 0.7×	407 0.7×	165 0.6×	143 0.6×	21	2.0k
Andrew M. Leidal United States	17	941 0.6×	1.5k 1.1×	343 0.6×	218 0.8×	576 2.4×	25	2.4k
Edmond Y.W. Chan United Kingdom	17	2.2k 1.5×	1.3k 1.0×	932 1.6×	123 0.5×	187 0.8×	19	2.9k
Shin‐ichiro Hino Japan	9	1.2k 0.8×	1.2k 0.9×	1.2k 2.1×	177 0.7×	123 0.5×	9	2.5k
Gregory A. Wyant United States	14	704 0.5×	2.2k 1.7×	698 1.2×	321 1.2×	496 2.0×	16	3.5k
Lyndsay M. Murrow United States	10	794 0.5×	2.1k 1.6×	285 0.5×	803 3.1×	536 2.2×	12	3.4k
Guang‐Chao Chen Taiwan	21	606 0.4×	1.0k 0.8×	420 0.7×	184 0.7×	126 0.5×	50	1.9k

Countries citing papers authored by Manuela Antonioli

Since Specialization

Citations

This map shows the geographic impact of Manuela Antonioli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Manuela Antonioli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Manuela Antonioli more than expected).

Fields of papers citing papers by Manuela Antonioli

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Manuela Antonioli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Manuela Antonioli. The network helps show where Manuela Antonioli may publish in the future.

Co-authorship network of co-authors of Manuela Antonioli

This figure shows the co-authorship network connecting the top 25 collaborators of Manuela Antonioli. A scholar is included among the top collaborators of Manuela Antonioli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Manuela Antonioli. Manuela Antonioli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Vescovo, Tiziana, et al.. (2025). Gut microbiota interplay with autophagy-EMT dynamics in colorectal cancer. Frontiers in Cell and Developmental Biology. 13. 1608248–1608248.

Melaiu, Ombretta, Stefan Ebert, Nicola Tumino, et al.. (2025). Inhibition of autophagy enhances the antitumor efficacy of T/CAR T cell against neuroblastoma. Journal of Experimental & Clinical Cancer Research. 44(1). 185–185.

Sankar, Devanarayanan Siva, Stéphanie Pébernard, Christine Vionnet, et al.. (2024). The ULK1 effector BAG2 regulates autophagy initiation by modulating AMBRA1 localization. Cell Reports. 43(9). 114689–114689. 8 indexed citations

Ureshino, Rodrigo Portes, Cláudia Bincoletto, Manuela Antonioli, et al.. (2023). NAADP-Evoked Ca2+ Signaling Leads to Mutant Huntingtin Aggregation and Autophagy Impairment in Murine Astrocytes. International Journal of Molecular Sciences. 24(6). 5593–5593. 9 indexed citations

Ciccosanti, Fabiola, Manuela Antonioli, Alessandra Sacchi, et al.. (2022). Proteomic analysis identifies a signature of disease severity in the plasma of COVID-19 pneumonia patients associated to neutrophil, platelet and complement activation. Clinical Proteomics. 19(1). 38–38. 14 indexed citations

Vescovo, Tiziana, et al.. (2020). Regulation of Autophagy in Cells Infected With Oncogenic Human Viruses and Its Impact on Cancer Development. Frontiers in Cell and Developmental Biology. 8. 47–47. 30 indexed citations

Mitchell, Robert D., Jonathan Sheard, Manuela Antonioli, et al.. (2019). Secretome of adipose-derived mesenchymal stem cells promotes skeletal muscle regeneration through synergistic action of extracellular vesicle cargo and soluble proteins. Stem Cell Research & Therapy. 10(1). 116–116. 154 indexed citations

D’Eletto, Manuela, Federica Rossin, Maria Grazia Farrace, et al.. (2018). Transglutaminase Type 2 Regulates ER-Mitochondria Contact Sites by Interacting with GRP75. Cell Reports. 25(13). 3573–3581.e4. 135 indexed citations

Mitchell, Robert D., Manuela Antonioli, Oliver Kretz, et al.. (2017). Protein and Molecular Characterization of a Clinically Compliant Amniotic Fluid Stem Cell-Derived Extracellular Vesicle Fraction Capable of Accelerating Muscle Regeneration Through Enhancement of Angiogenesis. Stem Cells and Development. 26(18). 1316–1333. 42 indexed citations

10.

Nazio, Francesca, Marianna Carinci, Cristina Valacca, et al.. (2016). Fine-tuning of ULK1 mRNA and protein levels is required for autophagy oscillation. The Journal of Cell Biology. 215(6). 841–856. 112 indexed citations

11.

Rossin, Federica, Manuela D’Eletto, Maria Grazia Farrace, et al.. (2016). Transglutaminase type 2-dependent selective recruitment of proteins into exosomes under stressful cellular conditions. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1863(8). 2084–2092. 43 indexed citations

12.

Antonioli, Manuela, Martina Di Rienzo, Mauro Piacentini, & Gian María Fimia. (2016). Emerging Mechanisms in Initiating and Terminating Autophagy. Trends in Biochemical Sciences. 42(1). 28–41. 215 indexed citations

13.

Antonioli, Manuela, et al.. (2016). ACQUA: Automated Cyanobacterial Quantification Algorithm for toxic filamentous genera using spline curves, pattern recognition and machine learning. Journal of Microbiological Methods. 124. 48–56. 8 indexed citations

14.

Nardacci, Roberta, Giuseppe Filomeni, Federica Rossin, et al.. (2015). Reticulon protein-1C is a key component of MAMs. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1853(3). 733–745. 15 indexed citations

15.

Antonioli, Manuela, et al.. (2015). AMBRA1-regulated autophagy in vertebrate development. The International Journal of Developmental Biology. 59(1-2-3). 109–117. 13 indexed citations

16.

Antonioli, Manuela, Francesca Nazio, Tiziana Vescovo, et al.. (2014). AMBRA1 Interplay with Cullin E3 Ubiquitin Ligases Regulates Autophagy Dynamics. Developmental Cell. 31(6). 734–746. 113 indexed citations

17.

Corazzari, Marco, Francesca Rapino, Fabiola Ciccosanti, et al.. (2014). Oncogenic BRAF induces chronic ER stress condition resulting in increased basal autophagy and apoptotic resistance of cutaneous melanoma. Cell Death and Differentiation. 22(6). 946–958. 131 indexed citations

18.

Nazio, Francesca, Flavie Strappazzon, Manuela Antonioli, et al.. (2013). mTOR inhibits autophagy by controlling ULK1 ubiquitylation, self-association and function through AMBRA1 and TRAF6. Nature Cell Biology. 15(4). 406–416. 622 indexed citations breakdown →

19.

Fimia, Gian María, Marco Corazzari, Manuela Antonioli, & Mauro Piacentini. (2012). Ambra1 at the crossroad between autophagy and cell death. Oncogene. 32(28). 3311–3318. 84 indexed citations

20.

Bartolomeo, Sabrina Di, Marco Corazzari, Francesca Nazio, et al.. (2010). The dynamic interaction of AMBRA1 with the dynein motor complex regulates mammalian autophagy. The Journal of Cell Biology. 191(1). 155–168. 375 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact