Mandy Mayo Aust

445 total citations
7 papers, 370 citations indexed

About

Mandy Mayo Aust is a scholar working on Molecular Biology, Hematology and Infectious Diseases. According to data from OpenAlex, Mandy Mayo Aust has authored 7 papers receiving a total of 370 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Hematology and 2 papers in Infectious Diseases. Recurrent topics in Mandy Mayo Aust's work include Protein Degradation and Inhibitors (3 papers), Histone Deacetylase Inhibitors Research (3 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). Mandy Mayo Aust is often cited by papers focused on Protein Degradation and Inhibitors (3 papers), Histone Deacetylase Inhibitors Research (3 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). Mandy Mayo Aust collaborates with scholars based in United States and France. Mandy Mayo Aust's co-authors include Steven Grant, Mohamed Rahmani, Andrea Ferreira‐Gonzalez, D. Clive Williams, Elisa Attkisson, Jonathan W. Friedberg, Catherine I. Dumur, Shou Zhen Wang, Sheng Zu Zhu and Gordon D. Ginder and has published in prestigious journals such as Blood, Cancer Research and Clinical Cancer Research.

In The Last Decade

Mandy Mayo Aust

7 papers receiving 368 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mandy Mayo Aust United States 5 289 95 79 52 49 7 370
Su‐Fern Tan United States 16 348 1.2× 96 1.0× 101 1.3× 47 0.9× 46 0.9× 29 477
Irina Mantovani Italy 8 291 1.0× 74 0.8× 98 1.2× 30 0.6× 39 0.8× 10 381
Sreedhar Venkannagari United States 6 341 1.2× 115 1.2× 97 1.2× 20 0.4× 68 1.4× 8 415
Bettina Wingelhofer Austria 9 205 0.7× 74 0.8× 123 1.6× 42 0.8× 44 0.9× 12 377
Katherine Knorr United States 7 424 1.5× 83 0.9× 94 1.2× 37 0.7× 50 1.0× 9 529
Aaron M. Domina United States 6 353 1.2× 54 0.6× 143 1.8× 45 0.9× 23 0.5× 9 429
Mai Itoh Japan 11 243 0.8× 58 0.6× 80 1.0× 35 0.7× 39 0.8× 31 361
Stela Álvarez-Fernández Spain 8 216 0.7× 118 1.2× 83 1.1× 16 0.3× 49 1.0× 11 363
Nandini Sakurikar United States 8 236 0.8× 44 0.5× 136 1.7× 29 0.6× 33 0.7× 10 337
Seemana Bhattacharya United States 11 402 1.4× 72 0.8× 168 2.1× 22 0.4× 16 0.3× 23 483

Countries citing papers authored by Mandy Mayo Aust

Since Specialization
Citations

This map shows the geographic impact of Mandy Mayo Aust's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mandy Mayo Aust with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mandy Mayo Aust more than expected).

Fields of papers citing papers by Mandy Mayo Aust

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mandy Mayo Aust. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mandy Mayo Aust. The network helps show where Mandy Mayo Aust may publish in the future.

Co-authorship network of co-authors of Mandy Mayo Aust

This figure shows the co-authorship network connecting the top 25 collaborators of Mandy Mayo Aust. A scholar is included among the top collaborators of Mandy Mayo Aust based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mandy Mayo Aust. Mandy Mayo Aust is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Rahmani, Mohamed, Mandy Mayo Aust, Edith P. Hawkins, et al.. (2015). Co-administration of the mTORC1/TORC2 inhibitor INK128 and the Bcl-2/Bcl-xL antagonist ABT-737 kills human myeloid leukemia cells through Mcl-1 down-regulation and AKT inactivation. Haematologica. 100(12). 1553–1563. 25 indexed citations
2.
Rahmani, Mohamed, Jason M. Beckta, Mandy Mayo Aust, et al.. (2015). Depletion of the chromatin remodeler CHD4 sensitizes AML blasts to genotoxic agents and reduces tumor formation. Blood. 126(12). 1462–1472. 46 indexed citations
3.
Rahmani, Mohamed, Mandy Mayo Aust, Rebecca E. Parker, et al.. (2015). Co-Administration of the mTORC1/TORC2 Inhibitor INK128 and the Bcl-2/Bcl-Xl Antagonist ABT-737 Kills Human Myeloid Leukemia Cells through Mcl-1 Down-Regulation and AKT Inactivation. Blood. 126(23). 3676–3676. 3 indexed citations
4.
Rahmani, Mohamed, et al.. (2014). PI3K/mTOR Inhibition Markedly Potentiates HDAC Inhibitor Activity in NHL Cells through BIM- and MCL-1–Dependent Mechanisms In Vitro and In Vivo. Clinical Cancer Research. 20(18). 4849–4860. 76 indexed citations
5.
6.
Rahmani, Mohamed, Mandy Mayo Aust, Elisa Attkisson, et al.. (2012). Dual Inhibition of Bcl-2 and Bcl-xL Strikingly Enhances PI3K Inhibition-Induced Apoptosis in Human Myeloid Leukemia Cells through a GSK3- and Bim-Dependent Mechanism. Cancer Research. 73(4). 1340–1351. 128 indexed citations
7.
Rahmani, Mohamed, Mandy Mayo Aust, Elisa Attkisson, et al.. (2012). Inhibition of Bcl-2 antiapoptotic members by obatoclax potently enhances sorafenib-induced apoptosis in human myeloid leukemia cells through a Bim-dependent process. Blood. 119(25). 6089–6098. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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