Mandakh Bekhbat

1.8k total citations · 1 hit paper
34 papers, 1.2k citations indexed

About

Mandakh Bekhbat is a scholar working on Behavioral Neuroscience, Biological Psychiatry and Neurology. According to data from OpenAlex, Mandakh Bekhbat has authored 34 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Behavioral Neuroscience, 25 papers in Biological Psychiatry and 13 papers in Neurology. Recurrent topics in Mandakh Bekhbat's work include Stress Responses and Cortisol (28 papers), Tryptophan and brain disorders (25 papers) and Neuroinflammation and Neurodegeneration Mechanisms (12 papers). Mandakh Bekhbat is often cited by papers focused on Stress Responses and Cortisol (28 papers), Tryptophan and brain disorders (25 papers) and Neuroinflammation and Neurodegeneration Mechanisms (12 papers). Mandakh Bekhbat collaborates with scholars based in United States, Germany and China. Mandakh Bekhbat's co-authors include Gretchen N. Neigh, Jennifer C. Felger, David R. Goldsmith, Sydney A. Rowson, Ebrahim Haroon, Andrew H. Miller, Michael T. Treadway, Neeti D. Mehta, Sean D. Kelly and Bobbi J. Woolwine and has published in prestigious journals such as Biological Psychiatry, Pharmacological Reviews and Neuroscience.

In The Last Decade

Mandakh Bekhbat

33 papers receiving 1.2k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Inflammation-Related Functional and Structural Dysconnectivity as a Pathway to Psychopathology

2022 132 citations David R. Goldsmith, Mandakh Bekhbat et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mandakh Bekhbat United States	19	619	534	192	162	149	34	1.2k
Jan‐Sebastian Grigoleit Germany	16	432 0.7×	466 0.9×	190 1.0×	126 0.8×	208 1.4×	20	1.0k
Nils Kappelmann United Kingdom	15	796 1.3×	536 1.0×	132 0.7×	137 0.8×	101 0.7×	22	1.4k
Daniela Krause Germany	20	616 1.0×	306 0.6×	242 1.3×	197 1.2×	117 0.8×	46	1.4k
Julie Lasselin Sweden	25	527 0.9×	491 0.9×	148 0.8×	205 1.3×	303 2.0×	50	2.0k
Dennis Liu Australia	20	648 1.0×	287 0.5×	255 1.3×	238 1.5×	177 1.2×	63	1.5k
Bianka Karshikoff Sweden	18	334 0.5×	373 0.7×	131 0.7×	88 0.5×	230 1.5×	38	1.4k
Steven S. Zalcman United States	21	333 0.5×	338 0.6×	174 0.9×	169 1.0×	133 0.9×	32	1.0k
Daniele Mattei Germany	12	512 0.8×	283 0.5×	339 1.8×	357 2.2×	162 1.1×	20	1.1k
Emanuele F. Osimo United Kingdom	16	1.1k 1.8×	620 1.2×	207 1.1×	240 1.5×	176 1.2×	25	1.9k
Sinead M. O’Brien Ireland	9	752 1.2×	529 1.0×	143 0.7×	96 0.6×	97 0.7×	9	1.2k

Countries citing papers authored by Mandakh Bekhbat

Since Specialization

Citations

This map shows the geographic impact of Mandakh Bekhbat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mandakh Bekhbat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mandakh Bekhbat more than expected).

Fields of papers citing papers by Mandakh Bekhbat

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mandakh Bekhbat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mandakh Bekhbat. The network helps show where Mandakh Bekhbat may publish in the future.

Co-authorship network of co-authors of Mandakh Bekhbat

This figure shows the co-authorship network connecting the top 25 collaborators of Mandakh Bekhbat. A scholar is included among the top collaborators of Mandakh Bekhbat based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mandakh Bekhbat. Mandakh Bekhbat is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bekhbat, Mandakh, et al.. (2025). Neurotransmitter and metabolic effects of interferon-alpha in association with decreased striatal dopamine in a non-human primate model of cytokine-Induced depression. Brain Behavior and Immunity. 125. 308–318. 4 indexed citations

Bekhbat, Mandakh, David R. Goldsmith, Ngoc‐Anh Le, et al.. (2025). Lipids and C-reactive protein predict anhedonia and reward circuit functional connectivity responses to anti-cytokine and dopaminergic therapies in patients with depression. Comprehensive Psychoneuroendocrinology. 21. 100284–100284. 3 indexed citations

Bekhbat, Mandakh. (2024). Glycolytic metabolism: Food for immune cells, fuel for depression?. Brain Behavior & Immunity - Health. 40. 100843–100843. 3 indexed citations

Treadway, Michael T., Jessica A. Cooper, Amanda R. Arulpragasam, et al.. (2024). A randomized proof-of-mechanism trial of TNF antagonism for motivational deficits and related corticostriatal circuitry in depressed patients with high inflammation. Molecular Psychiatry. 30(4). 1407–1417. 11 indexed citations

Rowson, Sydney A., Mandakh Bekhbat, Sean D. Kelly, et al.. (2024). Chronic adolescent stress alters GR-FKBP5 interactions in the hippocampus of adult female rats. Stress. 27(1). 2312467–2312467. 1 indexed citations

Bekhbat, Mandakh, et al.. (2023). Repeated social defeat stress leads to immunometabolic shifts in innate immune cells of the spleen. Brain Behavior & Immunity - Health. 34. 100690–100690. 3 indexed citations

Brydges, Christopher R., Sudeepa Bhattacharyya, Yuri Milaneschi, et al.. (2022). Metabolomic and inflammatory signatures of symptom dimensions in major depression. Brain Behavior and Immunity. 102. 42–52. 60 indexed citations

Rubin, Leah H., Mandakh Bekhbat, Susie Turkson, et al.. (2022). Glucocorticoid Receptor Function and Cognitive Performance in Women With HIV. Psychosomatic Medicine. 84(8). 893–903. 1 indexed citations

Bekhbat, Mandakh, Zhihao Li, Michael T. Treadway, et al.. (2022). Functional connectivity in reward circuitry and symptoms of anhedonia as therapeutic targets in depression with high inflammation: evidence from a dopamine challenge study. Molecular Psychiatry. 27(10). 4113–4121. 60 indexed citations

10.

Bekhbat, Mandakh, Michael T. Treadway, & Jennifer C. Felger. (2022). Inflammation as a Pathophysiologic Pathway to Anhedonia: Mechanisms and Therapeutic Implications. Current topics in behavioral neurosciences. 58. 397–419. 32 indexed citations

11.

Bekhbat, Mandakh, Mikhail G. Dozmorov, John C. Stansfield, et al.. (2021). Adolescent stress sensitizes the adult neuroimmune transcriptome and leads to sex-specific microglial and behavioral phenotypes. Neuropsychopharmacology. 46(5). 949–958. 26 indexed citations

12.

Bekhbat, Mandakh, David R. Goldsmith, Bobbi J. Woolwine, et al.. (2021). Transcriptomic signatures of psychomotor slowing in peripheral blood of depressed patients: evidence for immunometabolic reprogramming. Molecular Psychiatry. 26(12). 7384–7392. 20 indexed citations

13.

Bekhbat, Mandakh, Michael T. Treadway, David R. Goldsmith, et al.. (2020). Gene signatures in peripheral blood immune cells related to insulin resistance and low tyrosine metabolism define a sub-type of depression with high CRP and anhedonia. Brain Behavior and Immunity. 88. 161–165. 56 indexed citations

14.

Goldsmith, David R., Mandakh Bekhbat, Ngoc‐Anh Le, et al.. (2020). Protein and gene markers of metabolic dysfunction and inflammation together associate with functional connectivity in reward and motor circuits in depression. Brain Behavior and Immunity. 88. 193–202. 32 indexed citations

15.

Bekhbat, Mandakh, Karen Chu, Ngoc‐Anh Le, et al.. (2018). Glucose and lipid-related biomarkers and the antidepressant response to infliximab in patients with treatment-resistant depression. Psychoneuroendocrinology. 98. 222–229. 49 indexed citations

16.

Bekhbat, Mandakh, C. Christina Mehta, Sean D. Kelly, et al.. (2018). HIV and symptoms of depression are independently associated with impaired glucocorticoid signaling. Psychoneuroendocrinology. 96. 118–125. 14 indexed citations

17.

Bekhbat, Mandakh & Gretchen N. Neigh. (2017). Sex differences in the neuro-immune consequences of stress: Focus on depression and anxiety. Brain Behavior and Immunity. 67. 1–12. 245 indexed citations

18.

Bekhbat, Mandakh, Sydney A. Rowson, & Gretchen N. Neigh. (2017). Checks and balances: The glucocorticoid receptor and NFĸB in good times and bad. Frontiers in Neuroendocrinology. 46. 15–31. 89 indexed citations

19.

Bekhbat, Mandakh, Madelyn C. Houser, Jianjun Chang, et al.. (2016). Chronic psychological stress and high-fat high-fructose diet disrupt metabolic and inflammatory gene networks in the brain, liver, and gut and promote behavioral deficits in mice. Brain Behavior and Immunity. 59. 158–172. 75 indexed citations

20.

Nemeth, Christina L., et al.. (2014). Microglial activation occurs in the absence of anxiety-like behavior following microembolic stroke in female, but not male, rats. Journal of Neuroinflammation. 11(1). 174–174. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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