Malika M. Morar

1.3k total citations
12 papers, 1.0k citations indexed

About

Malika M. Morar is a scholar working on Immunology, Infectious Diseases and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Malika M. Morar has authored 12 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 4 papers in Infectious Diseases and 3 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Malika M. Morar's work include Immune Cell Function and Interaction (6 papers), T-cell and B-cell Immunology (4 papers) and Immunotherapy and Immune Responses (3 papers). Malika M. Morar is often cited by papers focused on Immune Cell Function and Interaction (6 papers), T-cell and B-cell Immunology (4 papers) and Immunotherapy and Immune Responses (3 papers). Malika M. Morar collaborates with scholars based in United States, Australia and Denmark. Malika M. Morar's co-authors include Jeffrey A. Bluestone, Lukas T. Jeker, Dimitri de Kouchkovsky, Sujan Shresta, Tyler R. Prestwood, Lauren E. Yauch, Wendy Rosenthal, Raphaël M. Zellweger, Michel DuPage and Alexander Marson and has published in prestigious journals such as Blood, Immunity and Nature Immunology.

In The Last Decade

Malika M. Morar

12 papers receiving 1.0k citations

Peers

Malika M. Morar
Sarah C. Vick United States
Malika M. Morar
Citations per year, relative to Malika M. Morar Malika M. Morar (= 1×) peers Sarah C. Vick

Countries citing papers authored by Malika M. Morar

Since Specialization
Citations

This map shows the geographic impact of Malika M. Morar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Malika M. Morar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Malika M. Morar more than expected).

Fields of papers citing papers by Malika M. Morar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Malika M. Morar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Malika M. Morar. The network helps show where Malika M. Morar may publish in the future.

Co-authorship network of co-authors of Malika M. Morar

This figure shows the co-authorship network connecting the top 25 collaborators of Malika M. Morar. A scholar is included among the top collaborators of Malika M. Morar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Malika M. Morar. Malika M. Morar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Morar, Malika M., et al.. (2022). Lower Extremity Salvage in a Diabetic Patient With Cutaneous Mucormycosis and COVID-19 After Open Patella Fracture. Ochsner Journal. 22(2). 163–168. 3 indexed citations
2.
Daffis, Stéphane, Malika M. Morar, Divya Pattabiraman, et al.. (2018). Cytokine-dependent activation of MAIT cells by the TLR8 agonist GS-9688 but not the TLR7 agonist GS-9620. Journal of Hepatology. 68. S800–S800. 6 indexed citations
4.
DuPage, Michel, Gaurav Chopra, Wendy Rosenthal, et al.. (2015). The Chromatin-Modifying Enzyme Ezh2 Is Critical for the Maintenance of Regulatory T Cell Identity after Activation. Immunity. 42(2). 227–238. 236 indexed citations
5.
Gool, Frédéric Van, Ari B. Molofsky, Malika M. Morar, et al.. (2014). Interleukin-5–producing group 2 innate lymphoid cells control eosinophilia induced by interleukin-2 therapy. Blood. 124(24). 3572–3576. 83 indexed citations
6.
Baumjohann, Dirk, Robin Kageyama, Jonathan M. Clingan, et al.. (2013). The microRNA cluster miR-17∼92 promotes TFH cell differentiation and represses subset-inappropriate gene expression. Nature Immunology. 14(8). 840–848. 165 indexed citations
7.
Baumjohann, Dirk, Robin Kageyama, Jonathan M. Clingan, et al.. (2013). 16. Cytokine. 63(3). 246–247. 1 indexed citations
8.
Kouchkovsky, Dimitri de, Jonathan H. Esensten, Wendy Rosenthal, et al.. (2013). microRNA-17–92 Regulates IL-10 Production by Regulatory T Cells and Control of Experimental Autoimmune Encephalomyelitis. The Journal of Immunology. 191(4). 1594–1605. 101 indexed citations
9.
Prestwood, Tyler R., et al.. (2012). Trafficking and Replication Patterns Reveal Splenic Macrophages as Major Targets of Dengue Virus in Mice. Journal of Virology. 86(22). 12138–12147. 53 indexed citations
10.
Jeker, Lukas T., Xuyu Zhou, Dimitri de Kouchkovsky, et al.. (2012). MicroRNA 10a Marks Regulatory T Cells. PLoS ONE. 7(5). e36684–e36684. 97 indexed citations
11.
Prestwood, Tyler R., Malika M. Morar, Raphaël M. Zellweger, et al.. (2012). Gamma Interferon (IFN-γ) Receptor Restricts Systemic Dengue Virus Replication and Prevents Paralysis in IFN-α/β Receptor-Deficient Mice. Journal of Virology. 86(23). 12561–12570. 97 indexed citations
12.
Yauch, Lauren E., Tyler R. Prestwood, Malika M. Morar, et al.. (2010). CD4+ T Cells Are Not Required for the Induction of Dengue Virus-Specific CD8+ T Cell or Antibody Responses but Contribute to Protection after Vaccination. The Journal of Immunology. 185(9). 5405–5416. 160 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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