Malathi K. Kistler

894 total citations
15 papers, 437 citations indexed

About

Malathi K. Kistler is a scholar working on Molecular Biology, Genetics and Reproductive Medicine. According to data from OpenAlex, Malathi K. Kistler has authored 15 papers receiving a total of 437 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 7 papers in Genetics and 7 papers in Reproductive Medicine. Recurrent topics in Malathi K. Kistler's work include Sperm and Testicular Function (7 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (5 papers) and Reproductive Biology and Fertility (4 papers). Malathi K. Kistler is often cited by papers focused on Sperm and Testicular Function (7 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (5 papers) and Reproductive Biology and Fertility (4 papers). Malathi K. Kistler collaborates with scholars based in United States and France. Malathi K. Kistler's co-authors include W. Stephen Kistler, Gary Horvath, Paolo Sassone‐Corsi, Robert P. Lawther, Michael C. Ostrowski, Anindya Dasgupta, Wenli Ma, Robert E. Taylor, Tia Davis and Michael J. Dewey and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Biochemistry.

In The Last Decade

Malathi K. Kistler

15 papers receiving 428 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Malathi K. Kistler United States 13 292 177 174 67 41 15 437
Jovenal San Agustin United States 6 354 1.2× 311 1.8× 72 0.4× 30 0.4× 12 0.3× 6 469
Pawan Puri United States 10 206 0.7× 94 0.5× 148 0.9× 104 1.6× 29 0.7× 19 351
Liliana Daín Argentina 11 242 0.8× 155 0.9× 96 0.6× 122 1.8× 114 2.8× 28 413
Muriel Klopfenstein France 8 389 1.3× 210 1.2× 210 1.2× 149 2.2× 15 0.4× 12 550
Virginie Pierre France 6 225 0.8× 249 1.4× 341 2.0× 289 4.3× 7 0.2× 7 542
Gregor Schlüter Germany 13 246 0.8× 186 1.1× 151 0.9× 82 1.2× 3 0.1× 25 486
Robert A. Mullinax United States 2 177 0.6× 347 2.0× 236 1.4× 103 1.5× 46 1.1× 3 622
R.L. Sutherland Australia 4 130 0.4× 122 0.7× 35 0.2× 10 0.1× 39 1.0× 6 335
Jean-François Éthier Canada 11 376 1.3× 116 0.7× 203 1.2× 321 4.8× 50 1.2× 12 633
Can Dai China 11 234 0.8× 122 0.7× 209 1.2× 310 4.6× 6 0.1× 22 446

Countries citing papers authored by Malathi K. Kistler

Since Specialization
Citations

This map shows the geographic impact of Malathi K. Kistler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Malathi K. Kistler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Malathi K. Kistler more than expected).

Fields of papers citing papers by Malathi K. Kistler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Malathi K. Kistler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Malathi K. Kistler. The network helps show where Malathi K. Kistler may publish in the future.

Co-authorship network of co-authors of Malathi K. Kistler

This figure shows the co-authorship network connecting the top 25 collaborators of Malathi K. Kistler. A scholar is included among the top collaborators of Malathi K. Kistler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Malathi K. Kistler. Malathi K. Kistler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Kistler, W. Stephen, Gary Horvath, Anindya Dasgupta, & Malathi K. Kistler. (2009). Differential expression of Rfx1-4 during mouse spermatogenesis. Gene Expression Patterns. 9(7). 515–519. 21 indexed citations
2.
Horvath, Gary, Malathi K. Kistler, & W. Stephen Kistler. (2009). RFX2 is a candidate downstream amplifier of A-MYB regulation in mouse spermatogenesis. BMC Developmental Biology. 9(1). 63–63. 27 indexed citations
3.
Ma, Wenli, Gary Horvath, Malathi K. Kistler, & W. Stephen Kistler. (2008). Expression Patterns of SP1 and SP3 During Mouse Spermatogenesis: SP1 Down-Regulation Correlates with Two Successive Promoter Changes and Translationally Compromised Transcripts1. Biology of Reproduction. 79(2). 289–300. 22 indexed citations
4.
Horvath, Gary, W. Stephen Kistler, & Malathi K. Kistler. (2004). RFX2 Is a Potential Transcriptional Regulatory Factor for Histone H1t and Other Genes Expressed During the Meiotic Phase of Spermatogenesis1. Biology of Reproduction. 71(5). 1551–1559. 38 indexed citations
5.
Horvath, Gary, Anindya Dasgupta, Malathi K. Kistler, & W. Stephen Kistler. (2003). The rat histone H1d gene has intragenic activating sequences that are absent from the testis-specific variant H1t. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1625(2). 165–172. 2 indexed citations
6.
Horvath, Gary, et al.. (2001). Mice with a Targeted Disruption of the H1t Gene Are Fertile and Undergo Normal Changes in Structural Chromosomal Proteins During Spermiogenesis1. Biology of Reproduction. 64(2). 425–431. 52 indexed citations
7.
Horvath, Gary, et al.. (2001). Characterization of the H1t Promoter: Role of Conserved Histone 1 AC and TG Elements and Dominance of the Cap-Proximal Silencer1. Biology of Reproduction. 65(4). 1074–1081. 8 indexed citations
8.
Davis, Tia, et al.. (2000). Elimination of Male Germ Cells in Transgenic Mice by the Diphtheria Toxin A Chain Gene Directed by the Histone H1t Promoter1. Biology of Reproduction. 63(2). 409–416. 12 indexed citations
9.
Kistler, W. Stephen, et al.. (1997). Characterization of the Promoter Region of the Rat Testis-Specific Histone H1t Gene1. Biology of Reproduction. 56(1). 73–82. 20 indexed citations
10.
Kistler, W. Stephen, et al.. (1997). The Testis-specific Histone H1t Gene Is Strongly Repressed by a G/C-rich Region Just Downstream of the TATA Box. Journal of Biological Chemistry. 272(52). 33028–33036. 34 indexed citations
11.
Kistler, Malathi K., Paolo Sassone‐Corsi, & W. Stephen Kistler. (1994). Identification of a Functional Cyclic Adenosine 3′,5′-Monophosphate Response Element in the 5′-Flanking Region of the Gene for Transition Protein 1 (TP1), a Basic Chromosomal Protein of Mammalian Spermatids1. Biology of Reproduction. 51(6). 1322–1329. 72 indexed citations
12.
Kistler, Malathi K., et al.. (1985). Androgen regulated genes from prostate and seminal vesicle share upstream sequence homologies. Biochemical and Biophysical Research Communications. 126(2). 948–952. 25 indexed citations
13.
Kistler, Malathi K., et al.. (1983). Characterization of a genomic clone for rat seminal vesicle secretory protein IV. Nucleic Acids Research. 11(10). 3169–3186. 51 indexed citations
14.
Ostrowski, Michael C., Malathi K. Kistler, & W. Stephen Kistler. (1982). Effect of castration on the synthesis of seminal vesicle secretory protein IV in the rat. Biochemistry. 21(15). 3525–3529. 37 indexed citations
15.
Kistler, Malathi K., et al.. (1981). Isolation of recombinant plasmids containing structural gene sequences for rat seminal vesicle secretory proteins IV and V. Biochemical and Biophysical Research Communications. 99(4). 1161–1166. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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