Maja Mueller

2.7k total citations
9 papers, 1.7k citations indexed

About

Maja Mueller is a scholar working on Surgery, Cardiology and Cardiovascular Medicine and Molecular Biology. According to data from OpenAlex, Maja Mueller has authored 9 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Surgery, 4 papers in Cardiology and Cardiovascular Medicine and 3 papers in Molecular Biology. Recurrent topics in Maja Mueller's work include Angiogenesis and VEGF in Cancer (3 papers), Cardiovascular Health and Disease Prevention (3 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (2 papers). Maja Mueller is often cited by papers focused on Angiogenesis and VEGF in Cancer (3 papers), Cardiovascular Health and Disease Prevention (3 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (2 papers). Maja Mueller collaborates with scholars based in Germany, Switzerland and Italy. Maja Mueller's co-authors include Ulf Landmesser, Helmut Drexler, Costantina Manes, Ferdinand H. Bahlmann, Stephan Spiekermann, Sajoscha Sorrentino, Dieter Fischer, Christian Besler, Kathrin Heinrich and Arnold von Eckardstein and has published in prestigious journals such as Circulation, Hypertension and Arteriosclerosis Thrombosis and Vascular Biology.

In The Last Decade

Maja Mueller

9 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Maja Mueller Germany 8 668 567 544 430 355 9 1.7k
Costantina Manes Germany 11 464 0.7× 428 0.8× 553 1.0× 287 0.7× 236 0.7× 18 1.5k
Cristina Dı́az-Jullien Spain 17 760 1.1× 535 0.9× 339 0.6× 190 0.4× 264 0.7× 28 1.6k
Zhen-Yan Fu China 23 475 0.7× 502 0.9× 455 0.8× 505 1.2× 210 0.6× 131 1.7k
Maurizio R. Soma Italy 24 989 1.5× 724 1.3× 312 0.6× 389 0.9× 406 1.1× 49 1.9k
Shin-ichiro Miura Japan 23 493 0.7× 643 1.1× 554 1.0× 317 0.7× 186 0.5× 91 1.5k
Koji Oida Japan 19 588 0.9× 355 0.6× 462 0.8× 400 0.9× 216 0.6× 51 1.5k
Tohru Ohama Japan 24 554 0.8× 412 0.7× 337 0.6× 334 0.8× 169 0.5× 56 1.4k
Carola Doerries Germany 10 296 0.4× 575 1.0× 388 0.7× 254 0.6× 210 0.6× 11 1.3k
Roberta Baetta Italy 22 944 1.4× 621 1.1× 253 0.5× 300 0.7× 358 1.0× 39 2.1k
Peter Santer Austria 18 789 1.2× 1.0k 1.8× 477 0.9× 377 0.9× 573 1.6× 26 2.5k

Countries citing papers authored by Maja Mueller

Since Specialization
Citations

This map shows the geographic impact of Maja Mueller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maja Mueller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maja Mueller more than expected).

Fields of papers citing papers by Maja Mueller

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maja Mueller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maja Mueller. The network helps show where Maja Mueller may publish in the future.

Co-authorship network of co-authors of Maja Mueller

This figure shows the co-authorship network connecting the top 25 collaborators of Maja Mueller. A scholar is included among the top collaborators of Maja Mueller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maja Mueller. Maja Mueller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Riwanto, Meliana, Lucia Rohrer, Bernd Roschitzki, et al.. (2013). Altered Activation of Endothelial Anti- and Proapoptotic Pathways by High-Density Lipoprotein from Patients with Coronary Artery Disease. Circulation. 127(8). 891–904. 277 indexed citations
2.
Jakob, Philipp, Carola Doerries, Sylvie Briand, et al.. (2012). Loss of AngiomiR-126 and 130a in Angiogenic Early Outgrowth Cells From Patients With Chronic Heart Failure. Circulation. 126(25). 2962–2975. 103 indexed citations
3.
Sonnenschein, Kristina, Tibor Horváth, Maja Mueller, et al.. (2011). Exercise training improves in vivo endothelial repair capacity of early endothelial progenitor cells in subjects with metabolic syndrome. European Journal of Cardiovascular Prevention & Rehabilitation. 18(3). 406–414. 31 indexed citations
4.
Giannotti, Giovanna, Carola Doerries, Pavani Mocharla, et al.. (2010). Impaired Endothelial Repair Capacity of Early Endothelial Progenitor Cells in Prehypertension. Hypertension. 55(6). 1389–1397. 151 indexed citations
5.
Sorrentino, Sajoscha, Christian Besler, Lucia Rohrer, et al.. (2009). Endothelial-Vasoprotective Effects of High-Density Lipoprotein Are Impaired in Patients With Type 2 Diabetes Mellitus but Are Improved After Extended-Release Niacin Therapy. Circulation. 121(1). 110–122. 300 indexed citations
6.
Landmesser, Ulf, Stephan Spiekermann, Christoph Preuß, et al.. (2007). Angiotensin II Induces Endothelial Xanthine Oxidase Activation. Arteriosclerosis Thrombosis and Vascular Biology. 27(4). 943–948. 169 indexed citations
8.
Landmesser, Ulf, Ferdinand H. Bahlmann, Maja Mueller, et al.. (2005). Simvastatin Versus Ezetimibe. Circulation. 111(18). 2356–2363. 341 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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