M. Koenig

493 total citations
10 papers, 319 citations indexed

About

M. Koenig is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, M. Koenig has authored 10 papers receiving a total of 319 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Cellular and Molecular Neuroscience and 2 papers in Neurology. Recurrent topics in M. Koenig's work include Mitochondrial Function and Pathology (8 papers), Genetic Neurodegenerative Diseases (8 papers) and Neurological diseases and metabolism (2 papers). M. Koenig is often cited by papers focused on Mitochondrial Function and Pathology (8 papers), Genetic Neurodegenerative Diseases (8 papers) and Neurological diseases and metabolism (2 papers). M. Koenig collaborates with scholars based in France, United States and Tunisia. M. Koenig's co-authors include Jean‐Louis Mandel, Giorgio Sirugo, Ursula Boschert, René Hen, Franck Duclos, M. Ben Hamida, Samir Belal, J. Beckmann, Christine Tranchant and Denys Chaigne and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Neurology and Human Genetics.

In The Last Decade

M. Koenig

10 papers receiving 310 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
M. Koenig France	7	249	205	60	40	39	10	319
F. Hentati Tunisia	7	288 1.2×	224 1.1×	44 0.7×	80 2.0×	50 1.3×	14	450
Ruth Darrow United States	8	283 1.1×	64 0.3×	24 0.4×	6 0.1×	23 0.6×	9	382
Tânia R. Soares Portugal	6	208 0.8×	143 0.7×	36 0.6×	59 1.5×	21 0.5×	6	319
Mário Laço Portugal	11	326 1.3×	247 1.2×	29 0.5×	77 1.9×	19 0.5×	15	437
Jongyun Myeong South Korea	14	320 1.3×	177 0.9×	55 0.9×	14 0.3×	6 0.2×	25	555
Ciara M. Walsh United Kingdom	7	302 1.2×	173 0.8×	79 1.3×	7 0.2×	11 0.3×	8	496
Lei Shang China	11	264 1.1×	63 0.3×	67 1.1×	11 0.3×	36 0.9×	14	383
Krisztina Peter United States	7	184 0.7×	103 0.5×	52 0.9×	11 0.3×	13 0.3×	7	429
Annika Pfeiffer Germany	7	216 0.9×	75 0.4×	30 0.5×	27 0.7×	33 0.8×	7	313
Paola Tanzarella Italy	8	193 0.8×	53 0.3×	36 0.6×	105 2.6×	34 0.9×	9	376

Countries citing papers authored by M. Koenig

Since Specialization

Citations

This map shows the geographic impact of M. Koenig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Koenig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Koenig more than expected).

Fields of papers citing papers by M. Koenig

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. Koenig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Koenig. The network helps show where M. Koenig may publish in the future.

Co-authorship network of co-authors of M. Koenig

This figure shows the co-authorship network connecting the top 25 collaborators of M. Koenig. A scholar is included among the top collaborators of M. Koenig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. Koenig. M. Koenig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

Anheim, Mathieu, Marie‐Céline Fleury, B. Monga, et al.. (2009). Epidemiological, clinical, paraclinical and molecular study of a cohort of 102 patients affected with autosomal recessive progressive cerebellar ataxia from Alsace, Eastern France: implications for clinical management. Neurogenetics. 11(1). 1–12. 103 indexed citations

Anheim, Mathieu, Denys Chaigne, Marie Fleury, et al.. (2008). Ataxie spastique autosomique récessive de Charlevoix-Saguenay : étude d’une famille et revue de la littérature. Revue Neurologique. 164(4). 363–368. 16 indexed citations

Hansen, John H. L., et al.. (2006). Effects of Phoneme Characteristics on TEO Feature-based Automatic Stress Detection in Speech. 1. 357–360. 7 indexed citations

Delatycki, M. B., Melanie A. Knight, M. Koenig, et al.. (1999). G130V, a common FRDA point mutation, appears to have arisen from a common founder. Human Genetics. 105(4). 343–346. 5 indexed citations

Rötig, Agnès, Pascale de Lonlay, Dominique Chrétien, et al.. (1998). Frataxin gene expansion causes aconitase and mitochondrial iron-sulphur protein deficiency in Friedreich ataxia. European Journal of Human Genetics. 6. 36–36. 2 indexed citations

Monrós, Eugènia, Joaquı́n Cañizares, María Dolores Moltó, et al.. (1996). Evidence for a Common Origin ofMost Friedreich AtaxiaChromosomes in the SpanishPopulation. European Journal of Human Genetics. 4(4). 191–198. 10 indexed citations

Hamida, M. Ben, Samir Belal, Giorgio Sirugo, et al.. (1993). Friedreich's ataxia phenotype not linked to chromosome 9 and associated with selective autosomal recessive vitamin E deficiency in two inbred Tunisian families. Neurology. 43(11). 2179–2179. 96 indexed citations

Duclos, Franck, Ursula Boschert, Giorgio Sirugo, et al.. (1993). Gene in the region of the Friedreich ataxia locus encodes a putative transmembrane protein expressed in the nervous system.. Proceedings of the National Academy of Sciences. 90(1). 109–113. 65 indexed citations

Belal, Samir, Giorgio Sirugo, P.A. Ioannou, et al.. (1992). Study of large inbred Friedreich ataxia families reveals a recombination between D9S15 and the disease locus.. PubMed. 51(6). 1372–6. 11 indexed citations

10.

Lambert, Amaury, et al.. (1991). [Intestinal absorption of insulin with a new telemetric shuttle in dogs].. PubMed. 15(3). 187–93. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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