M E Williams

535 total citations
8 papers, 431 citations indexed

About

M E Williams is a scholar working on Pathology and Forensic Medicine, Genetics and Molecular Biology. According to data from OpenAlex, M E Williams has authored 8 papers receiving a total of 431 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Pathology and Forensic Medicine, 4 papers in Genetics and 2 papers in Molecular Biology. Recurrent topics in M E Williams's work include Lymphoma Diagnosis and Treatment (6 papers), Chronic Lymphocytic Leukemia Research (4 papers) and Cutaneous lymphoproliferative disorders research (2 papers). M E Williams is often cited by papers focused on Lymphoma Diagnosis and Treatment (6 papers), Chronic Lymphocytic Leukemia Research (4 papers) and Cutaneous lymphoproliferative disorders research (2 papers). M E Williams collaborates with scholars based in United States. M E Williams's co-authors include Andrew Arnold, Carol L. Rosenberg, S H Swerdlow, D J Innes, Steven H. Swerdlow, N L Harris, Woo‐Ick Yang, Lawrence Zukerberg, Terence T. Casey and John P. Greer and has published in prestigious journals such as Journal of Clinical Investigation, Annals of Oncology and The American Journal of Surgical Pathology.

In The Last Decade

M E Williams

8 papers receiving 423 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
M E Williams United States 7 325 244 190 102 58 8 431
S H Swerdlow United States 6 283 0.9× 226 0.9× 171 0.9× 103 1.0× 72 1.2× 8 412
DA Filippa United States 10 442 1.4× 293 1.2× 271 1.4× 81 0.8× 81 1.4× 19 533
WG Sanger United States 6 340 1.0× 201 0.8× 184 1.0× 38 0.4× 60 1.0× 8 390
RM Braziel United States 4 330 1.0× 221 0.9× 156 0.8× 74 0.7× 53 0.9× 5 434
Vera Vanhentenrijk Belgium 12 374 1.2× 241 1.0× 219 1.2× 144 1.4× 143 2.5× 13 571
M. Sol Mateo Spain 8 292 0.9× 190 0.8× 190 1.0× 107 1.0× 22 0.4× 9 401
Marisol Mateo Spain 8 512 1.6× 227 0.9× 379 2.0× 99 1.0× 78 1.3× 8 627
Iwona Wlodarska Belgium 11 250 0.8× 137 0.6× 233 1.2× 96 0.9× 113 1.9× 14 441
Ellis G. Levine United States 6 344 1.1× 193 0.8× 266 1.4× 59 0.6× 75 1.3× 7 491
Elena Ruíz-Ballesteros Spain 8 299 0.9× 164 0.7× 211 1.1× 174 1.7× 96 1.7× 8 468

Countries citing papers authored by M E Williams

Since Specialization
Citations

This map shows the geographic impact of M E Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M E Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M E Williams more than expected).

Fields of papers citing papers by M E Williams

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M E Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M E Williams. The network helps show where M E Williams may publish in the future.

Co-authorship network of co-authors of M E Williams

This figure shows the co-authorship network connecting the top 25 collaborators of M E Williams. A scholar is included among the top collaborators of M E Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M E Williams. M E Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Williams, M E, et al.. (1997). . Annals of Oncology. 8(2+). 71–73. 11 indexed citations
2.
Swerdlow, Steven H., Lawrence Zukerberg, Woo‐Ick Yang, N L Harris, & M E Williams. (1996). The Morphologic Spectrum of Non-Hodgkin's Lymphomas with BCL1/Cyclin D1 Gene Rearrangements. The American Journal of Surgical Pathology. 20(5). 627–640. 76 indexed citations
3.
Williams, M E, S H Swerdlow, Carol L. Rosenberg, & Andrew Arnold. (1993). Chromosome 11 translocation breakpoints at the PRAD1/cyclin D1 gene locus in centrocytic lymphoma.. PubMed. 7(2). 241–5. 92 indexed citations
4.
Williams, M E, S H Swerdlow, Carol L. Rosenberg, & Andrew Arnold. (1992). Characterization of chromosome 11 translocation breakpoints at the bcl-1 and PRAD1 loci in centrocytic lymphoma.. PubMed. 52(19 Suppl). 5541s–5544s. 94 indexed citations
5.
Humphries, John Eric, Holly K. Dressman, & M E Williams. (1991). Immunoglobulin gene rearrangement in multiple myeloma: Limitations of southern blot analysis. Human Pathology. 22(10). 966–971. 4 indexed citations
6.
Casey, Terence T., et al.. (1990). Monomorphic lymphomas arising in patients with Hodgkin's disease. Correlation of morphologic, immunophenotypic, and molecular genetic findings in 12 cases.. PubMed. 136(1). 81–94. 25 indexed citations
7.
Williams, M E, et al.. (1989). Ki-1-positive cutaneous large cell lymphoma of T cell type: Report of an indolent subtype. Journal of the American Academy of Dermatology. 20(2). 342–348. 29 indexed citations
8.
Innes, D J, et al.. (1989). Sequential bcl-2 and c-myc oncogene rearrangements associated with the clinical transformation of non-Hodgkin's lymphoma.. Journal of Clinical Investigation. 84(5). 1454–1459. 100 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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