Lydia Murray

587 citations
10 papers · 461 · h-index 7

Impact in

Papers in

Lydia Murray

10 papers receiving 460 citations

Peers

Lydia Murray
Comparison fields: 5 of 80
  • Immunology and Allergy 81
  • Biological Psychiatry 10
  • Gastroenterology 19
  • Physiology 80
  • Genetics 90
Replace Mamdouh H. Kedees with:
Mamdouh H. Kedees United States
Salvatore Magrì Italy
Alí Francisco Citalán‐Madrid Mexico
Katsutoshi Kawasaki Japan
Kavitha N. Rao United States
Ruijin Wu China
Peiman Shooshtarizadeh Canada
Bianca Baker United States
Anna Jia China
Berna Kaya Switzerland
Lydia Murray relative to Mamdouh H. Kedees United States Mamdouh H. Kedees's profile →
Citations per field
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Mamdouh H. Kedees · 1×
Citations per year

Countries citing papers authored by Lydia Murray

Since Specialization
Citations

This map shows the geographic impact of Lydia Murray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lydia Murray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lydia Murray more than expected).

Fields of papers citing papers by Lydia Murray

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lydia Murray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lydia Murray. The network helps show where Lydia Murray may publish in the future.

Co-authors

The 25 scholars most cited alongside Lydia Murray, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Lydia Murray Line = papers co-authored together Lydia Murray links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 2016283
2 201352
3 201637
4 201731
5 201828
6 20179
7
No evidence for the presence of apolipoprotein epsilon4, interleukin-lalpha allele 2 and interleukin-1beta allele 2 cause an increase in programmed cell death following traumatic brain injury in humans.
20077
8 20196
9 20206
10 20112

About Lydia Murray

Lydia Murray is a scholar working on Immunology and Allergy, Genetics, Molecular Biology, Neurology and Cell Biology, having authored 10 papers that have together received 461 indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (4 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (3 papers), Traumatic Brain Injury and Neurovascular Disturbances (1 paper), Biomarkers in Disease Mechanisms (1 paper), S100 Proteins and Annexins (1 paper), Renal Diseases and Glomerulopathies (1 paper), Gut microbiota and health (1 paper) and Barrier Structure and Function Studies (1 paper). The work is most often cited by research in Immunology and Allergy (81 citations), Biological Psychiatry (10 citations), Gastroenterology (19 citations), Physiology (80 citations) and Genetics (90 citations). Lydia Murray has collaborated with scholars based in United Kingdom, Australia and Norway. Frequent co-authors include Michael A. McGuckin, Sumaira Z. Hasnain, Ran Wang, Rohan Lourie, Timothy H. Florin, Stuart E. Denman, Veronika Schreiber, Graham Magor, Kuan Yau Wong and Andrew C. Perkins. Their work appears in journals such as Human Molecular Genetics, Nutrients, Kidney International Reports, Scientific Reports and Nature.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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